Tumor Necrosis Factor Receptor Superfamily, Member 19 (TNFRSF19) ELISA Kits

Human Tumor Necrosis Factor Receptor Superfamily, Member 19 (TNFRSF19) Interaktionspartner

1. TNFRSF19 may contribute to the development of colorectal tumors with deregulated beta-catenin (zeige CTNNB1 ELISA Kits) activity.

2. Results show aberrant expression and/or signaling by TROY as a contributor to the dispersion of glioblastoma cells and therapeutic resistance.

3. the association of RhoGDIalpha with TROY contributed to TROY-dependent RhoA (zeige RHOA ELISA Kits) activation and neurite outgrowth inhibition after Nogo (zeige RTN4 ELISA Kits)-66 stimulation.

4. TROY is up-regulated in human colorectal cancer cell lines and in intestinal tumors in mice. It functions as a negative modulator of the Wnt (zeige WNT2 ELISA Kits) pathway in LGR5 (zeige LGR5 ELISA Kits)-positive stem cells.

5. study demonstrated that TNFR2 (zeige TNFRSF1B ELISA Kits) and TROY mRNA levels are enhanced in cutaneous squamous cell carcinoma cells compared to healthy ones in a Tunisian population

6. The FAM134B and TNFRSF19 showed a dramatically strong synergistic epistasis in explaining the genetic dissection of the susceptibility to complex vascular dementia.

7. TNFRSF19 overexpression in glioma cells activates Rac1 signaling in a Pyk2 (zeige PTK2B ELISA Kits)-dependent manner to drive glioma cell invasion and migration

8. Findings provide new insights into the pathogenesis of NPC (zeige NPC1 ELISA Kits) by highlighting the involvement of pathways related to TNFRSF19 and MDS1-EVI1 (zeige MECOM ELISA Kits) in addition to HLA molecules.

9. TROY regulates melanoma growth.

10. the ternary complex of NgR (zeige RTN4R ELISA Kits)/TROY/LINGO-1 expressed on astrocytes, macrophages/microglia and neurones, by interacting with Nogo-A (zeige RTN4 ELISA Kits) on oligodendrocytes, might modulate glial-neuronal interactions in demyelinating lesions of multiple sclerosis

Mouse (Murine) Tumor Necrosis Factor Receptor Superfamily, Member 19 (TNFRSF19) Interaktionspartner

1. High TROY expression is associated with glioblastoma cell invasion.

2. TROY negatively modulates remyelination in the central nervous system.

3. the association of RhoGDIalpha with TROY contributed to TROY-dependent RhoA (zeige RHOA ELISA Kits) activation and neurite outgrowth inhibition after Nogo (zeige RTN4 ELISA Kits)-66 stimulation.

4. TROY is up-regulated in human colorectal cancer cell lines and in intestinal tumors in mice. It functions as a negative modulator of the Wnt (zeige WNT2 ELISA Kits) pathway in LGR5 (zeige LGR5 ELISA Kits)-positive stem cells.

5. TROY is expressed in microglia, involved in their migration and targeted by propentofylline.

6. TROY plays some important roles in neurogenesis of embryonic stages.

7. This gene was a target of WNT (zeige WNT2 ELISA Kits) signaling in the dorsal somite.

8. Analysis of expression patterns of eda (zeige EDA ELISA Kits), edar (zeige EDAR ELISA Kits) and tnfrsf19 in mouse embryogenesis.

9. Both mRNA and protein of TROY are detected in the olfactory bulb during embryogenesis; TROY expression is most intense in the inner layer. In the postnatal olfactory bulb, TROY-expressing cells are also detected in the glomerular layer.

10. Our results implicate TROY in mediating myelin inhibition, offering new insights into the molecular mechanisms of regeneration failure in the adult nervous system.