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The protein encoded by TNFRSF19 is a member of the TNF-receptor superfamily. Zusätzlich bieten wir Ihnen Tumor Necrosis Factor Receptor Superfamily, Member 19 Proteine (18) und Tumor Necrosis Factor Receptor Superfamily, Member 19 Kits (14) und viele weitere Produktgruppen zu diesem Protein an.
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TNFRSF19 may contribute to the development of colorectal tumors with deregulated beta-catenin (zeige CTNNB1 Antikörper) activity.
Results show aberrant expression and/or signaling by TROY as a contributor to the dispersion of glioblastoma cells and therapeutic resistance.
the association of RhoGDIalpha with TROY contributed to TROY-dependent RhoA (zeige RHOA Antikörper) activation and neurite outgrowth inhibition after Nogo (zeige RTN4 Antikörper)-66 stimulation.
TROY is up-regulated in human colorectal cancer cell lines and in intestinal tumors in mice. It functions as a negative modulator of the Wnt (zeige WNT2 Antikörper) pathway in LGR5 (zeige LGR5 Antikörper)-positive stem cells.
study demonstrated that TNFR2 (zeige TNFRSF1B Antikörper) and TROY mRNA levels are enhanced in cutaneous squamous cell carcinoma cells compared to healthy ones in a Tunisian population
The FAM134B (zeige FAM134B Antikörper) and TNFRSF19 showed a dramatically strong synergistic epistasis in explaining the genetic dissection of the susceptibility to complex vascular dementia.
TNFRSF19 overexpression in glioma cells activates Rac1 signaling in a Pyk2 (zeige PTK2B Antikörper)-dependent manner to drive glioma cell invasion and migration
Findings provide new insights into the pathogenesis of NPC (zeige NPC1 Antikörper) by highlighting the involvement of pathways related to TNFRSF19 and MDS1-EVI1 (zeige MECOM Antikörper) in addition to HLA molecules.
TROY regulates melanoma growth.
the ternary complex of NgR (zeige RTN4R Antikörper)/TROY/LINGO-1 (zeige LINGO1 Antikörper) expressed on astrocytes, macrophages/microglia and neurones, by interacting with Nogo-A (zeige RTN4 Antikörper) on oligodendrocytes, might modulate glial-neuronal interactions in demyelinating lesions of multiple sclerosis
High TROY expression is associated with glioblastoma cell invasion.
TROY negatively modulates remyelination in the central nervous system.
TROY is expressed in microglia, involved in their migration and targeted by propentofylline.
TROY plays some important roles in neurogenesis of embryonic stages.
This gene was a target of WNT (zeige WNT2 Antikörper) signaling in the dorsal somite.
Analysis of expression patterns of eda (zeige EDA Antikörper), edar (zeige EDAR Antikörper) and tnfrsf19 in mouse embryogenesis.
Both mRNA and protein of TROY are detected in the olfactory bulb during embryogenesis; TROY expression is most intense in the inner layer. In the postnatal olfactory bulb, TROY-expressing cells are also detected in the glomerular layer.
Our results implicate TROY in mediating myelin inhibition, offering new insights into the molecular mechanisms of regeneration failure in the adult nervous system.
The protein encoded by this gene is a member of the TNF-receptor superfamily. This receptor is highly expressed during embryonic development. It has been shown to interact with TRAF family members, and to activate JNK signaling pathway when overexpressed in cells. This receptor is capable of inducing apoptosis by a caspase-independent mechanism, and it is thought to play an essential role in embryonic development. Alternatively spliced transcript variants encoding distinct isoforms have been described.
tumor necrosis factor receptor superfamily member 19
, tumor necrosis factor receptor superfamily, member 19
, tumor necrosis factor receptor superfamily member 19-like
, toxicity and JNK inducer
, Toxicity and JNK inducer