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SELPLG encodes a glycoprotein that functions as a high affinity counter-receptor for the cell adhesion molecules P-, E- and L- selectin expressed on myeloid cells and stimulated T lymphocytes. Zusätzlich bieten wir Ihnen SELPLG Antikörper (264) und SELPLG Proteine (13) und viele weitere Produktgruppen zu diesem Protein an.
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Human SELPLG ELISA Kit für Sandwich ELISA - ABIN417559
Wang, Chen, Lee, Chang: Endoplasmic reticulum stress contributes to ferritin molecules-mediated macrophage migration via P-selectin glycoprotein ligand-1. in Molecular nutrition & food research 2016
Studies indicate that P-selectin glycoprotein ligand-1 (PSGL-1) PSGL-1 can negatively regulate T cell function.
Results suggest that PSGL-1 may play an oncogenic role in the development of intestinal tumors, and this is likely mediated through activation of NFkB signaling by MIP (zeige TNPO1 ELISA Kits)-1g.
c-Myc (zeige MYC ELISA Kits) regulates P-selectin glycoprotein ligand-1 expression in monocytes during HIV-1 infection.
The percentage of CXCR3 (zeige CXCR3 ELISA Kits)(+) CD4 (zeige CD4 ELISA Kits)(+) TEM (zeige CYLD ELISA Kits) cells negatively correlated with the severity of the cutaneous disease in psoriasis patients. Importantly CLA(+) CD4 (zeige CD4 ELISA Kits)(+) TCM cells expressing CCR6 (zeige CCR6 ELISA Kits)(+) or CCR4 (zeige CCR4 ELISA Kits)(+)CXCR3 (zeige CXCR3 ELISA Kits)(+) negatively correlated with psoriasis severity suggesting recruitment to the skin compartment.
Platelet-leukocyte aggregations increased in acute ischemic stroke patients rapidly within 3h. The I allele of PSGL-1 M62I was associated with risk of developing acute ischemic stroke, especially large artery atherosclerosis stroke and small artery occlusion stroke. Small artery occlusion stroke patients with the II genotype of PSGL-1 M62I have the higher level of platelet-neutrophil aggregates.
The significant presence of CLA+ T cells and E-selectin (zeige SELE ELISA Kits) expressions in the OLPG suggests their involvement in the etiopathogenesis of OLP; however, only a weak correlation between CLA+ T cells and E-selectin (zeige SELE ELISA Kits) was observed.
PSGL-1 is a novel receptor for S. pneumoniae that contributes to protection against invasive pneumococcal disease.
This study provides a better understanding of the biology of P-selectin (zeige SELP ELISA Kits) and PSGL-1 and their roles in dissemination and resensitization of Multiple myeloma treatment.
Report influence of SELPLG variation on leukocyte-platelet interactions in cardiovascular disease.
E-selectin (zeige SELE ELISA Kits) interactions with glycoprotein ligands (CD44 (zeige CD44 ELISA Kits)/hematopoietic cell E-/L-selectin (zeige SELL ELISA Kits) ligand and PSGL-1) mediate the initial capturing of cells out of flow.
results indicate that P-selectin (zeige SELP ELISA Kits) deletion significantly decreases tumor stiffness in Rip1 (zeige RALBP1 ELISA Kits)-Tag2 mice by inhibiting LOX (zeige LOX ELISA Kits) expression.
this study shows that PSGL-1 plays a fundamental role in balancing viral control and immunopathology and also functions to regulate T cell responses in the tumor microenvironment
Psgl-1 deficiency accelerates bleomycin-induced lung fibrosis and inflammation in mice through activating the PI3K/AKT (zeige AKT1 ELISA Kits) axis.
Circulating soluble P-selectin (zeige SELP ELISA Kits) must dimerize to promote inflammation and thrombosis in mice.
The results from the present study suggest that activated platelets secrete Pselectin to promote cardiac inflammation and fibrosis in Ang (zeige ANG ELISA Kits) IIinduced hypertension.
These findings indicate a novel pathway in tumor metastasis, i.e., tumor cell mediated activation of P-selectin (zeige SELP ELISA Kits) in platelets, followed by activation and secretion of Asm (zeige SMPD1 ELISA Kits) and in turn release of ceramide and tumor metastasis. The data suggest that p38 MAPK (zeige MAPK14 ELISA Kits) acts downstream from P-selectin (zeige SELP ELISA Kits) and is necessary for the secretion of Asm (zeige SMPD1 ELISA Kits).
Psgl-1 deficiency is protective against the prothrombotic effects of IL-1beta (zeige IL1B ELISA Kits) .
PSGL-1, CD43, and CD44 moved normally to the uropods of chemokine-stimulated C1galt1(-/-) neutrophils
these results demonstrate that P-selectin (zeige SELP ELISA Kits) expression on ECs is regulated in part by glycosylation mechanisms and that glycosylation inhibitors efficiently reduce the adhesion of sRBCs and leukocytes to ECs.
endothelial colony-forming cells interact with activated neutrophils via PSGL-1 and L-selectin (zeige SELL ELISA Kits)
This gene encodes a glycoprotein that functions as a high affinity counter-receptor for the cell adhesion molecules P-, E- and L- selectin expressed on myeloid cells and stimulated T lymphocytes. As such, this protein plays a critical role in leukocyte trafficking during inflammation by tethering of leukocytes to activated platelets or endothelia expressing selectins. This protein requires two post-translational modifications, tyrosine sulfation and the addition of the sialyl Lewis x tetrasaccharide (sLex) to its O-linked glycans, for its high-affinity binding activity. Aberrant expression of this gene and polymorphisms in this gene are associated with defects in the innate and adaptive immune response. Alternate splicing results in multiple transcript variants.
P-selectin glycoprotein ligand 1
, cutaneous lymphocyte-associated associated antigen
, selectin P ligand
, P-selectin glycoprotein ligand 1 propeptide
, P-selectin glycoprotein ligand-1
, leukocyte cell surface adhesion molecule
, selectin, platelet (p-selectin) ligand