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PHLDA1 encodes an evolutionarily conserved proline-histidine rich nuclear protein. Zusätzlich bieten wir Ihnen PHLDA1 Antikörper (48) und PHLDA1 Proteine (5) und viele weitere Produktgruppen zu diesem Protein an.
Our study negatively correlates expression of PHLDA1 and Aurora A (zeige AURKA ELISA Kits) in IMR-32 cells and sheds new light on functions of PHLDA1 in the neuroblastoma (zeige ARHGEF16 ELISA Kits) tumor cells, suggesting its role as a pro-apoptotic protein
Suggest decreased expression of PHLDA1 may play an important role in tumor progression, and may become a new adjunct biomarker in the prognosis in gastric adenocarcinoma.
Data suggest that high PHLDA1 expression is controlled through an ER-NFkappaB (zeige NFKB1 ELISA Kits)-miR (zeige MLXIP ELISA Kits)-181 regulatory axis and may contribute to a poor clinical outcome in patients with ER+ breast tumors by enhancing stem-like properties in these tumors.
PHLDA1 expression is a useful addition in differentiating trichoblastoma and basal cell carcinoma.
A role for PHLDA1 as an apoptosis suppressor in oral cancer cells.
Data show that downregulation of aurora A kinase (zeige AURKA ELISA Kits) by the therapeutic antibody is associated with decreased levels of MYCN (zeige MYCN ELISA Kits) protein in cytoplasm, and induced expression of PHLDA1 and P53 (zeige TP53 ELISA Kits) proteins.
The follicular stem cell marker PHLDA1 (TDAG51) indicates that most basaloid tumors in nevus sebaceus are basal cell carcinomas and not trichoblastomas.
PHLDA1 differentiates between desmoplastic trichoepithelioma and morpheaform basal cell carcinoma but shows variable staining in microcystic adnexal carcinoma.
the release of Ca(2 (zeige CA2 ELISA Kits)+) from endoplasmic reticulum stores mediates epithelial-to-mesenchymal transition in human proximal tubular epithelium via the induction of TDAG51
crucial negative regulator and effector of Aurora A kinase (zeige AURKA ELISA Kits) in breast cancer
PHLDA1 plays a critical role in the development of progressive lung contusion and subsequent inflammation.
These findings suggested that TDAG51 up-regulation is a dependent event during LPS (zeige TLR4 ELISA Kits)-mediated proliferation and cell cycle progression, and which increase our understanding of the interaction mechanism between LPS (zeige TLR4 ELISA Kits) and macrophages.
The JAK2 (zeige JAK2 ELISA Kits)-ERK1/2 (zeige MAPK1/3 ELISA Kits)-STAT3 (zeige STAT3 ELISA Kits) pathway is an important signaling pathway for regulation of PHLDA1 expression.
TDAG51 has a protective role in oxidative stress-induced (zeige SQSTM1 ELISA Kits) cell death in mouse embryonic fibroblasts.
Our results suggest that the balance between the cell survival and death signals mediated by HSP70 (zeige HSP70 ELISA Kits) and TDAG51, respectively, may be disturbed by the altered expression of HSF1 (zeige HSF1 ELISA Kits) during the progression of disease in this amyotropic lateral sclerosis model.
TDAG51 is involved in energy homeostasis at least in part by regulating lipogenesis in liver and white adipose tissue.
PHLDA1 expression marks the putative epithelial stem cells, downregulates ITGA2 (zeige ITGA2 ELISA Kits) and ITGA6 (zeige ITGA6 ELISA Kits), and contributes to intestinal tumorigenesis
TDAG51 is induced by homocysteine, promotes detachment-mediated PCD (zeige PCBD1 ELISA Kits), and contributes to the development of atherosclerosis observed in hyperhomocysteinemia
This gene encodes an evolutionarily conserved proline-histidine rich nuclear protein. The encoded protein may play an important role in the anti-apoptotic effects of insulin-like growth factor-1.
, PQR protein
, T-cell death-associated gene 51 protein
, apoptosis-associated nuclear protein
, pleckstrin homology-like domain family A member 1
, proline- and glutamine-rich protein
, proline- and histidine-rich protein
, proline-histidine rich protein
, T-cell death associated