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Controls phosphatidylcholine synthesis. Zusätzlich bieten wir Ihnen Phosphate Cytidylyltransferase 1, Choline, alpha Antikörper (74) und Phosphate Cytidylyltransferase 1, Choline, alpha Proteine (7) und viele weitere Produktgruppen zu diesem Protein an.
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PCYT1A mutations were identified in patients with isolated retinal dystrophy (zeige MERTK ELISA Kits) without any skeletal involvement from two Italian families.
CCT contributes to phospholipid compositional homeostasis. [Review]
PCYT1A-generated phosphatidylcholine (zeige SGMS2 ELISA Kits) has a role in the normal function of white adipose tissue and insulin (zeige INS ELISA Kits) action
We report loss-of-function mutations in PCYT1A as the cause of spondylometaphyseal dysplasia with cone-rod dystrophy.
Mutations in PCYT1A cause spondylometaphyseal dysplasia with cone-rod dystrophy
N-Methylaspartate induced nitric oxide synthase (zeige NOS ELISA Kits) activation and nuclear factor-kB subunit p65 nuclear translocation in A549 cells were responsible for decreased CTP:phosphocholine cytidylyltransferase A expression
Analyses showed genotype effects of PCYT1A genes on spina bifida risk, but did not show evidence of gene-nutrient. interactions.
The findings reported herein indicate that palmitate-induced cisternal endoplasmic reticulum expansion is dependent on the activation of XBP-1/CCTalpha-mediated phospholipid accumulation in RAW 264.7 cells.
Data suggest that CTalpha is an important factor in hepatocyte proliferation in vitro; however, liver regeneration, DNA synthesis, and phosphatidylcholine (zeige SGMS2 ELISA Kits) biosynthesis after partial hepatectomy are not impaired in CTalpha(-/-) knockout mice.
CaMKI (zeige CAMK1 ELISA Kits) vies with CRM1/exportin 1 (zeige XPO1 ELISA Kits) for access to a nuclear export signal, and assembly of a CaMKI (zeige CAMK1 ELISA Kits)-14-3-3 zeta (zeige YWHAZ ELISA Kits)-CCTalpha complex is a key effector mechanism that drives nuclear CCTalpha translocation.
This study illustrates, for the first time, the connection between the acute accumulation of farnesylated prelamin A and involvement of CCT-alpha in generating an nucleoplasmic reticulum.
Enforced expression of either CK(alpha) or CCTalpha increased the rate of synthesis and the amount of PtdCho, and these cells initiated neuronal differentiation.
14-3-3zeta controls CCTalpha nuclear import in response to calcium signals, thereby regulating mammalian phospholipid synthesis.
activation during the S phase of the cell cycle is mediated by the transcription factor Sp1 (zeige SP1 ELISA Kits)
cyclin-dependent kinase 2 (CDK2 (zeige CDK2 ELISA Kits)) phosphorylation of Sp1 (zeige SP1 ELISA Kits) activates CTalpha transcription during S phase.
hepatic CTalpha has a role in regulating both hepatic and systemic lipid and lipoprotein metabolism
ssion of CCTalpha increases surfactant PtdCho synthesis without affecting surfactant protein levels but disrupts glycogen (zeige GYS1 ELISA Kits) metabolism in differentiating type II cells via its regulatory domain
Controls phosphatidylcholine synthesis.
phosphate cytidylyltransferase 1, choline, alpha
, choline-phosphate cytidylyltransferase A-like
, CCT A
, CT A
, CTP:phosphocholine cytidylyltransferase A
, choline-phosphate cytidylyltransferase A
, phosphorylcholine transferase A
, CTP:phosphocholine cytidylyl transferase
, CTP:phosphocholine cytidylyltransferase alpha
, choline phosphate cytidylyltransferase 1 alpha
, phosphate cytidylyltransferase 1, choline, alpha isoform
, CTP phosphocholine cytidylyl transferase
, Phosphorylcholine transferase A