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Proteins of the matrix metalloproteinase (MMP) family are involved in the breakdown of extracellular matrix in normal physiological processes, such as embryonic development, reproduction, and tissue remodeling, as well as in disease processes, such as arthritis and metastasis. Zusätzlich bieten wir Ihnen MMP16 Antikörper (141) und MMP16 Kits (21) und viele weitere Produktgruppen zu diesem Protein an.
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miR-155 contributed to the up-regulation of MMP-16, and MMP-16 further degraded aggrecan and collagen type II, leading to the dehydration and degeneration of intervertebral discs
Neuronal knockdown of MT3-MMP resulted in the accumulation of NgR1 (zeige NEUROG1 Proteine) at the cell surface and reduced the accumulation of the NgR1 (zeige NEUROG1 Proteine) cleavage fragment in medium conditioned by cortical neurons
Study demonstrates that MT3-MMP, is expressed in mesenchymal tissues of the skeleton and in peri (zeige POSTN Proteine)-skeletal soft connective tissue; and MT3-MMP-deficient mice display growth inhibition tied to a decreased viability of mesenchymal cells in skeletal tissues.
Overexpression of MMP2 (zeige MMP2 Proteine) and MMP16 in endometrial cancerous tissues corresponded to down-regulation of miR (zeige MLXIP Proteine)-377, miR (zeige MLXIP Proteine)-382 and miR (zeige MLXIP Proteine)-410, while decreased expression of TIMP2 (zeige TIMP2 Proteine) was associated with miR (zeige MLXIP Proteine)-200b up-regulation.
MMP16 was highly expressed and correlated with poor prognosis in gastric cancer patients by promoting proliferation and invasion of gastric cancer cells
Findings suggest that these MMP16 rs10090371, ADAMTS3 rs788935, TLL2 (zeige TLL2 Proteine) rs10882807 and MMP9 (zeige MMP9 Proteine) rs3918251 may be promising prognostic biomarkers for cutaneous melanoma specific survival (CMSS).
the present findings indicate that MT3-MMP is down-regulated in ESCC, which correlates to lymph node metastasis and poor survival of patients with this disease.
more severe intervertebral disc degeneration is correlated with higher matrix metallopeptidase 16
results suggest that miR (zeige MLXIP Proteine)-145 functions as a tumor metastasis suppressor gene by down-regulating MMP16 and may be a potential target in osteosarcoma treatment
the results support a role for MMP16 in promoting invasive properties of the meningioma tumours
These results demonstrate that the MT3-MMP promoter is frequently hypermethylated in colorectal cancer and that downregulation of MT3-MMP may be important for cell migration in colorectal cancer.
MMP16 is a putative indicator of adverse melanoma prognosis.
Expression of MMP-16 in HTB9 and T24 cells increases following transforming growth factorbeta1 treatment.
Proteins of the matrix metalloproteinase (MMP) family are involved in the breakdown of extracellular matrix in normal physiological processes, such as embryonic development, reproduction, and tissue remodeling, as well as in disease processes, such as arthritis and metastasis. Most MMP's are secreted as inactive proproteins which are activated when cleaved by extracellular proteinases. The encoded protein activates MMP2 by cleavage. This gene was once referred to as MT-MMP2, but was renamed as MT-MMP3 or MMP16.
, MT-MMP 3
, Membrane type 3-MMP
, matrix metalloproteinase 16
, matrix metalloproteinase-16
, membrane-type matrix metalloproteinase 3
, membrane-type-3 matrix metalloproteinase
, Putative transmembrane protein C8orf57
, matrix metalloproteinase 16 (membrane-inserted)
, membrane type-matrix metalloproteinase