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MINA is a c-Myc (MYC\; MIM 190080) target gene that may play a role in cell proliferation or regulation of cell growth. Zusätzlich bieten wir Ihnen MYC Induced Nuclear Antigen Antikörper (88) und MYC Induced Nuclear Antigen Proteine (8) und viele weitere Produktgruppen zu diesem Protein an.
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Results indicate that mdig can inhibit the phosphorylation of GSK-3beta and promote the phosphorylation and destabilization of beta-catenin (zeige CTNNB1 ELISA Kits), in order to suppress the expression of slug, snail (zeige SNAI1 ELISA Kits), and ZEB1 (zeige ZEB1 ELISA Kits) and the occurrence of EMT (zeige ITK ELISA Kits), and thereby inhibit the invasion and metastasis of non-small cell lung cancer (NSCLC).
mdig directly interacts with c-myc (zeige MYC ELISA Kits) and JAK1 (zeige JAK1 ELISA Kits) in multiple myeloma (MM) cell lines, which contributes to hyperactivation of the IL-6 (zeige IL6 ELISA Kits)-JAK (zeige JAK3 ELISA Kits)-STAT3 (zeige STAT3 ELISA Kits) signaling important for the pathogenesis of MM.
MDIG overexpression substantially promoted hepatocellular carcinoma cell proliferation, cell migration and spreading, whereas knockdown of MDIG would reverse above-mentioned effect.
Findings reveal the role of MINA in glioblastoma initiation and progression by controlling the cell-cycle progression through transcriptionally regulating cyclin (zeige PCNA ELISA Kits) and CDK (zeige CDK4 ELISA Kits) expression via the demethylation of H3K9me3.
Data suggest that mineral dust-induced gene protein (mdig) accomplishes its functions on chromatin, DNA repair and cell growth through interacting with the partner proteins.
Histone modifier genes (JMJD1C, RREB1, MINA, KDM7A) alter conotruncal heart phenotypes in 22q11.2 deletion syndrome.
in lung cancer tissues, upregulation of mdig expression accompanies with the downregulation of p27(KIP1 (zeige CDKN1B ELISA Kits)) expression and in bronchial stump
Mina53 can be used as a marker for pancreatic cancer
Mina: a Th2 response regulator meets TGFbeta (zeige TGFB1 ELISA Kits).
Increased expression of mdig in cancer tissues.
Sp1 (zeige SP1 ELISA Kits)/3 transcription factors trigger Mina expression through additive activity targeted to a cluster of four Sp1 (zeige SP1 ELISA Kits)/3 binding sites forming the P1 promoter.
Mina53 plays a role in the allergic response to inhaled allergens, possibly through controlling IL-4 (zeige IL4 ELISA Kits) production.
Mina53 is prominently expressed in proliferating, undifferentiated spermatogonia, and plays a role in cell proliferation from the spermatogonial stage to the meiotic prophase in spermatogenesis, but not in meiotic divisions per se.
Mina specifically bound to and repressed the Il4 (zeige IL4 ELISA Kits) promoter. Mina overexpression in transgenic mice impaired Il4 (zeige IL4 ELISA Kits) expression, whereas its knockdown in primary CD4 (zeige CD4 ELISA Kits)(+) T cells led to Il4 (zeige IL4 ELISA Kits) derepression.
MINA is a c-Myc (MYC\; MIM 190080) target gene that may play a role in cell proliferation or regulation of cell growth. (Tsuneoka et al., 2002
60S ribosomal protein L27a histidine hydroxylase
, bifunctional lysine-specific demethylase and histidyl-hydroxylase MINA
, histone lysine demethylase MINA
, mineral dust induced gene protein
, mineral dust-induced gene protein
, myc-induced nuclear antigen, 53 kDa
, nucleolar protein 52
, ribosomal oxygenase MINA
, myc-induced nuclear antigen
, MYC induced nuclear antigen
, MYC-induced nuclear antigen
, MYC-induced nuclear antigen-like