Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
GFAP encodes one of the major intermediate filament proteins of mature astrocytes. Zusätzlich bieten wir Ihnen GFAP Kits (84) und GFAP Proteine (38) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 952 products:
Cat (Feline) Polyclonal GFAP Primary Antibody für IEM, ICC - ABIN789007
Segal, Takahashi, McKay: Changes in neurotrophin responsiveness during the development of cerebellar granule neurons. in Neuron 1993
Show all 199 Pubmed References
Human Polyclonal GFAP Primary Antibody für IHC (p), WB - ABIN3044350
Zhou, Wang, Li, Wang, Wu, Zhang: Paeoniflorin attenuates the neuroinflammatory response in a rat model of chronic constriction injury. in Molecular medicine reports 2017
Show all 50 Pubmed References
Human Monoclonal GFAP Primary Antibody für IHC (fro), IHC (p) - ABIN3043647
Lu, Yuan, Ou, Cai, Wang, Sun, Zhang: Autophagy and apoptosis during adult adipose-derived stromal cells differentiation into neuron-like cells in vitro. in Neural regeneration research 2015
Show all 49 Pubmed References
Human Polyclonal GFAP Primary Antibody für IHC (p), WB - ABIN3043832
Zhao, Guo, Li, Zang, Qu, Zhou, Li, Sun: Amelioration of dementia induced by A? 22-35 through rectal delivery of undecapeptide-hEGF to mouse brain. in International journal of pharmaceutics 2011
Show all 49 Pubmed References
Cat (Feline) Polyclonal GFAP Primary Antibody für ICC, IF - ABIN152487
Henion, Qu, Smith: Expression of dystroglycan, fukutin and POMGnT1 during mouse cerebellar development. in Brain research. Molecular brain research 2003
Show all 32 Pubmed References
Mammalian Monoclonal GFAP Primary Antibody für ISt, IHC - ABIN1304664
Savic, Stojiljkovic, Lavrnja, Parabucki, Bjelobaba, Nedeljkovic, Herdegen, Pekovic: Ribavirin shows immunomodulatory effects on activated microglia. in Immunopharmacology and immunotoxicology 2014
Show all 19 Pubmed References
Human Polyclonal GFAP Primary Antibody für FACS, IF (cc) - ABIN726200
Zhao, Zhao, Wang, Xu, Miao, Feng, Chen, Kovács, Fan, Zhang: Mice deficient in Epg5 exhibit selective neuronal vulnerability to degeneration. in The Journal of cell biology 2013
Show all 16 Pubmed References
Chicken Polyclonal GFAP Primary Antibody für ICC, IP - ABIN1742363
Christensen, Schneider-Axmann, Lucassen, Bayer, Wirths: Accumulation of intraneuronal Abeta correlates with ApoE4 genotype. in Acta neuropathologica 2010
Show all 13 Pubmed References
Bird (Avian) Monoclonal GFAP Primary Antibody für ICC, IF - ABIN446355
Brai, Marathe, Zentilin, Giacca, Nimpf, Kretz, Scotti, Alberi: Notch1 activity in the olfactory bulb is odour-dependent and contributes to olfactory behaviour. in The European journal of neuroscience 2014
Show all 7 Pubmed References
Human Monoclonal GFAP Primary Antibody für ICC, IHC (p) - ABIN457420
Bignami, Eng, Dahl, Uyeda: Localization of the glial fibrillary acidic protein in astrocytes by immunofluorescence. in Brain research 1972
Show all 6 Pubmed References
There was significantly more GFAP immunoreactivity in the prefrontal cortex and hippocampus of aged animals compared to adult or middle-aged animals.
Desmin (zeige DES Antikörper), Glial Fibrillary Acidic Protein, Vimentin (zeige VIM Antikörper), and Peripherin (zeige PRPH Antikörper) are type III intermediate filaments that have roles in health and disease [review]
Plasma concentration of GFAP demonstrated associations with stroke occurrence in a West African cohort but was not associated with stroke severity or mortality.
This study demonstrated that Concentrations of microparticles expressing GFAP and AQP4 (zeige AQP4 Antikörper) were significantly higher in the traumatic brain injury group compared with healthy controls.
The authors observed higher serum levels of GFAP and UCH-L1 (zeige UCHL1 Antikörper) in brain-injured children compared with controls and also demonstrated a step-wise increase of biomarker concentrations over the continuum of severity from mild to severe traumatic brain injury. Serum UCH-L1 (zeige UCHL1 Antikörper) and GFAP concentrations also strongly predicted poor outcome.
Study examined if QKI6B expression can predict the outcome of GFAP, and several oligodendrocyte-related genes, in the prefrontal cortex of brain samples of schizophrenic individuals. QKI6B significantly predicts the expression of GFAP, but does not predict oligodendrocyte-related gene outcome, as previously seen with other QKI (zeige QKI Antikörper) isoforms.
GFAP, along with tau and AmyloidBeta42, were increased in plasma up to 90 days after traumatic brain injury compared with controls.
Results show that the positive rates and expression levels of nestin (zeige NES Antikörper), tyrosine hydroxylase (TH (zeige TH Antikörper)), GFAP and IL-17 (zeige IL17A Antikörper) were significantly decreased while Foxp3 (zeige FOXP3 Antikörper) and the ratio of Foxp3 (zeige FOXP3 Antikörper)/IL-17 (zeige IL17A Antikörper) were statistically elevated in BM of AML (zeige RUNX1 Antikörper) patients.
GFAP levels >0.29 ng/ml were seen only in intracerebral hemorrhage, thus confirming the diagnosis of ICH (zeige COL4a2 Antikörper) during prehospital care.
These results indicate that autoantibodies against GFAP could serve as a predictive marker for the development of overt autoimmune diabetes.
Higher median plasma GFAP values were documented in intracerebral hemorrhage compared with acute ischemic stroke, stroke mimics, and controls.
Isolation of an evolutionary conserved novel GFAP isoform, GFAPkappa, produced by alternative splicing and polyadenylation of the 3'-region of the human GFAP pre-mRNA is described.
This report the successful prediction and validation of Gfap as an miR (zeige MLXIP Antikörper)-3099 target gene using a combination of bioinformatics resources with enrichment of annotations based on functional ontologies and a spatio-temporal expression dataset.
GFAP is specifically expressed in the auricular chondrocytes, and assumes a pivotal role in resistance against mechanical stress.
compared open-skull and thinned-skull imaging methods for two-photon laser microscopy of live astrocytes in neocortex of GFAP-GFP transgenic mice
work reveals that an Alexander disease-causing mutation alters GFAP turnover kinetics in vivo and provides an essential foundation for future studies aimed at preventing or reducing the accumulation of GFAP.
Tat (zeige TAT Antikörper) expression or GFAP expression led to formation of GFAP aggregates and induction of unfolded protein response (UPR) and endoplasmic reticulum (ER) stress in astrocytes.
Study provides evidence that transcription of one of the astrocyte-specific genes, Gfap, is cooperatively regulated by co-expressed genes and their regulatory factors.
This study demonstrated the GFAP-ApoE4 mice exhibited motor impairments when compared to GFAP-ApoE3 and wild-type mice.
PINK1 (zeige PINK1 Antikörper) deficiency causes defects in GFAP-positive astrogliogenesis during brain development.
Gnasxl (zeige GNAS Antikörper) deficiency does not directly affect glial development in the hypothalamus, since it is expressed in neurons, and Gfap-positive astrocytes and tanycytes appear normal during early postnatal stages.
Aggregation-prone GFAP mutation in Alexander disease was validated using a zebrafish model; The p.Asp128Asn GFAP mutation is likely to be a disease-causing mutation
The distribution of GFAP immunoreactivity implies that enteric glia are widespread in the fish gastrointestinal tract.
Generation of transgenic zebrafish that express green fluorescent protein (GFP) in glial cells driven by the zebrafish glial fibrillary acidic protein (GFAP) regulatory elements.
Cells expressing the two reporters display radial glial morphology, colocalize with the NSC marker Sox2 (zeige SOX2 Antikörper), undergo proliferation, and are capable of self-renewal within the matrix of distinct thickness in the telencephalon.
This gene encodes one of the major intermediate filament proteins of mature astrocytes. It is used as a marker to distinguish astrocytes from other glial cells during development. Mutations in this gene cause Alexander disease, a rare disorder of astrocytes in the central nervous system. Alternative splicing results in multiple transcript variants encoding distinct isoforms.
glial fibrillary acidic protein
, glial fibrillary acidic protein alpha
, intermediate filament
, intermediate filament protein
, zrf-1 antigen