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FOXN3 is a member of the forkhead/winged helix transcription factor family. Zusätzlich bieten wir Ihnen FOXN3 Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
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Dog (Canine) Polyclonal FOXN3 Primary Antibody für WB - ABIN2777625
Yu, Zhang, Zhou, Wu, Qu, Wei, Xing, Dong, Zhai, Wan, Ouyang, Li, Zhang, Zhou, Zhang, Wu, He: Gene expression profiling in human fetal liver and identification of tissue- and developmental-stage-specific genes through compiled expression profiles and efficient cloning of full-length cDNAs. in Genome research 2001
Show all 2 Pubmed References
the rs8004664 risk allele drives excessive expression of FOXN3 during fasting and that FOXN3 regulates fasting blood glucose.
FoxN3 regulates craniofacial and eye development by recruiting histone deacetylase (zeige HDAC1 Antikörper) complexes .
FOXN3 bind to beta-catenin (zeige CTNNB1 Antikörper) and inhibited beta-catenin (zeige CTNNB1 Antikörper)/TCF (zeige HNF4A Antikörper) signaling by blocking the interaction between beta-catenin (zeige CTNNB1 Antikörper) and TCF4 (zeige TCF4 Antikörper). Loss of FOXN3 in colon cancer activates beta-catenin (zeige CTNNB1 Antikörper)/TCF (zeige HNF4A Antikörper) signaling and promotes the growth and migration of cancer cells.
FOXN3 functions as a tumor suppressor in hepatocellular carcinoma by downregulating the expression of E2F5.
the FOXN3-NEAT1-SIN3A (zeige SIN3A Antikörper) complex promotes epithelial-to-mesenchymal transition and invasion of breast cancer cells in vitro as well as dissemination and metastasis of breast cancer in vivo
Foxn3 is a direct transcriptional suppressor of N-cadherin (zeige CDH2 Antikörper) in colorectal metastasis.
MEN1 patients with MEN1 mutations leading to CHES1-loss of interaction have a higher risk of malignant pancreatic neuroendocrine tumors with an aggressive course of disease and disease-related death.
CHES1 decreases protein synthesis and cell proliferation in tumor cell lines but not in normal fibroblasts.
the Foxn3 mutation leads to partial embryonic and postnatal lethality, growth retardation, eye formation defects, dental anomalies and craniofacial defects.
Data suggest that CHES1 recruits Ski-interacting protein (zeige SNW1 Antikörper) (SKIP) to repress genes important for tumorigenesis and the response to cancer treatments.
Meninis involved in the activation of S-phase arrest in multple endocrein neoplasia by interacting with CHES1.
This gene is a member of the forkhead/winged helix transcription factor family. Checkpoints are eukaryotic DNA damage-inducible cell cycle arrests at G1 and G2. Checkpoint suppressor 1 suppresses multiple yeast checkpoint mutations including mec1, rad9, rad53 and dun1 by activating a MEC1-independent checkpoint pathway. Alternative splicing is observed at the locus, resulting in distinct isoforms.
forkhead box N3
, checkpoint suppressor 1
, forkhead box protein N3
, forkhead box protein FoxN3b
, checkpoint suppressor 1-like
, forkhead box protein N3-like
, forkhead box protein FoxN3a