Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
ECM1 encodes a soluble protein that is involved in endochondral bone formation, angiogenesis, and tumor biology. Zusätzlich bieten wir Ihnen ECM1 Kits (57) und ECM1 Proteine (12) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 102 products:
Human Monoclonal ECM1 Primary Antibody für ELISA, EM - ABIN4306907
Tang, Tao, Yun-Liu, Sun, Geng, Jiang: Immunocytochemical localization of secretory component in Paneth cell secretory granules-rat Paneth cells participate in acquired immunity. in Journal of molecular histology 2005
Show all 7 Pubmed References
the present study demonstrated the association between ECM1 gene mutation and patients with LP. Patients with LP exhibited one homozygous point mutation (C220G) as previously reported, one novel homozygous mutation (c.508insCTG) and two heterozygous mutations (C220G/P.R481X and c.507delT/c.l473delT).
The results showed a significant upregulation of ECM1 and ITGB3 (zeige ITGB3 Antikörper), and a significant downregulation for FBLN5 (zeige FBLN5 Antikörper) in pelvic organ prolapse patients.
Three patients with homozygous mutations in sixth and seventh exons of the ECM1 gene had a drug-resistant course at the end of long-term follow-up
This proteome analysis indicate that ECM1 is a potential novel plasma protein biomarker for the detection of primary ESCC and evaluation of neoplasms progression
The TT genotype of ECM1 gene rs3737240 SNP significantly increased susceptibility for Ulcerative Colitis and azathioprine use in Ulcerative Colitis patients in a Turkish population.
our work has identified a novel function of ECM1 in inhibiting Th17 cell differentiation in the experimental autoimmune encephalomyelitis model
ECM1, which displayed a high expression in hepatocellular carcinoma (HCC (zeige FAM126A Antikörper)) specimens, was closely associated with clinicopathologic data and may promote migration and invasion of HCC (zeige FAM126A Antikörper) cells by inducing epithelia-mesenchymal transition.
Cell invasion (matrigel) was reduced only in the Hs578T cells (p < 0.01). Silencing decreased the expression of the prometastatic molecules S100A4 (zeige S100A4 Antikörper) and TGFbetaR2 in both cell lines and CD44 (zeige CD44 Antikörper) in Hs578T cells. We conclude that ECM1 is a key player in the metastatic process and regulates the actin cytoskeletal architecture of aggressive breast cancer cells at least in part via alterations in S100A4 (zeige S100A4 Antikörper) and Rho A (zeige RHOA Antikörper).
Overexpression of miR (zeige MLXIP Antikörper)-486-3p inhibited cell growth and metastasis by targeting ECM1.
For 1q21 loci, we confirmed gene ECM1 as the most plausible gene from this region to be involved in pathogenesis of inflammatory bowel disease
data identify ECM1 as a soluble protein to promote TFH cell differentiation and antibody production.
ECM1 seems to be critical for PTHrP action in chondrogenesis, as blockage of ECM1 nearly abolishes PTHrP regulation of chondrocyte hypertrophy, and overexpression of ECM1 rescues disorganized growth plates of PTHrP-null mice; ECM1 and progranulin (zeige GRN Antikörper) chondrogenic growth factor constitute an interaction network.
HA accumulation primes the vasculature for atherosclerosis by crosslinking and reorganizing the extracellular matrix (ECM (zeige MMRN1 Antikörper)) and by pushing VSMC differentiation towards a less mature phenotype.
Retinoid signaling in the stroma activates expression of Ecm1, which in turn down-regulates Ret (zeige RET Antikörper) expression in the ureteric bud cleft, where bifurcation normally occurs and normal branching progresses.
This gene encodes a soluble protein that is involved in endochondral bone formation, angiogenesis, and tumor biology. It also interacts with a variety of extracellular and structural proteins, contributing to the maintenance of skin integrity and homeostasis. Mutations in this gene are associated with lipoid proteinosis disorder (also known as hyalinosis cutis et mucosae or Urbach-Wiethe disease) that is characterized by generalized thickening of skin, mucosae and certain viscera. Alternatively spliced transcript variants encoding distinct isoforms have been described for this gene.
secretory component p85
, extracellular matrix protein 1