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The protein encoded by BCL2L14 belongs to the BCL2 protein family. Zusätzlich bieten wir Ihnen BCL2-Like 14 (Apoptosis Facilitator) Antikörper (40) und BCL2-Like 14 (Apoptosis Facilitator) Proteine (7) und viele weitere Produktgruppen zu diesem Protein an.
Data indicate that LRP6 (zeige LRP6 ELISA Kits), BCL2L14, DUSP16 (zeige DUSP16 ELISA Kits), CREBL2 (zeige CREBL2 ELISA Kits), and CDKN1B (zeige CDKN1B ELISA Kits) were involed in centromeric (12p11.21-12p13.2) deletion in ETV6 (zeige ETV6 ELISA Kits)-RUNX1 (zeige RUNX1 ELISA Kits) B-cell precursor acute lymphoblastic leukemia (BCP (zeige OPN1SW ELISA Kits)-ALL).
Single nucleotide polymorphism in BCL2L14 is associated with lung cancer.
prior knockdown of Bcl-G expression ablates the stimulation of basal apoptosis by FAU (zeige FAU ELISA Kits), consistent with an essential downstream role for Bcl-G, itself a candidate tumour suppressor, in mediating the apoptosis regulatory role of FAU (zeige FAU ELISA Kits).
siRNA downregulation of Bcl-G inhibited breast cancer cell apoptosis. Adding an siRNA against Fau (zeige FAU ELISA Kits) revealed control of Bcl-G by Fau (zeige FAU ELISA Kits). The most important factors controlling Bcl-G are post-translational modification by Fau (zeige FAU ELISA Kits) & MELK (zeige MELK ELISA Kits), not transcription rate.
There was no somatic mutation of BH3 domains of Bad, Bmf (zeige BMF ELISA Kits) and Bcl-G genes in transitional cell carcinoma samples. The data presented here indicate that BH3 domain mutation of these genes is rare in TCCs and may not contribute to the pathogenesis of TCCs.
the kinase activity of MELK (zeige MELK ELISA Kits) is likely to affect mammary carcinogenesis through inhibition of the pro-apoptotic function of Bcl-GL
data presented here indicate that BH3 domain mutation of the proapoptotic genes Bad, Bmf (zeige BMF ELISA Kits) and Bcl-G is rare in laryngeal squamous cell carcinoma and may not contribute to the apoptosis-resistance mechanisms of laryngeal squamous cell carcinoma
JAB1 (zeige COPS5 ELISA Kits) is involved in the regulation of mitochondrial apoptotic pathway through specific interaction with BclGs.
Increased BclG(L) expression may contribute to the aberrant CD4 (zeige CD4 ELISA Kits)+ T cell apoptosis which causes an inappropriate immune response and impaired homeostasis in systemic lupus erythematosus.
CD3 (zeige CD3E ELISA Kits)/CD28 (zeige CD28 ELISA Kits)-inducible MNSFbeta (zeige FAU ELISA Kits)-Bcl-G complex may be involved in the regulation of T cell function and survival.
Bcl-G is expressed highly in mature spermatids in the testis, CD8 (zeige CD8A ELISA Kits)(+) conventional dendritic cells (DCs) in hematopoietic tissues and diverse epithelial cell types, including those lining the gastrointestinal and respiratory tracts.
The protein encoded by this gene belongs to the BCL2 protein family. BCL2 family members form hetero- or homodimers and act as anti- or pro-apoptotic regulators that are involved in a wide variety of cellular activities. Overexpression of this gene has been shown to induce apoptosis in cells. Three alternatively spliced transcript variants encoding two distinct isoforms have been reported for this gene.
apoptosis facilitator Bcl-2-like protein 14
, apoptosis regulator BCL-G