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These analyses suggest that in zebrafish, as in humans, wnt9b plays distinct roles in directing morphogenetic movements of developing branchial arch elements.
Data indicate that mutations in wingless-type MMTV integration site family, member 9B protein (WNT9B) were frequently associated with Mayer-Rokitansky-Kuster-Hauser syndrome (MRKHS).
Epigenetic mechanism causes Wnt9b deficiency and nonsyndromic cleft lip and palate in the A/WySn mouse strain.
Expression of Wnt9b in Six2 (zeige SIX2 ELISA Kits)-positive cells disrupts cell fate decisions in the kidney and the gastrointestinal tract.
Epithelial Wnt7b (zeige WNT7B ELISA Kits) and Wnt9b as possible ligands of Fzd1 (zeige Fzd1 ELISA Kits)-mediated beta-catenin (Ctnnb1 (zeige CTNNB1 ELISA Kits))-dependent (canonical) Wnt (zeige WNT2 ELISA Kits) signaling in the undifferentiated ureteric mesenchyme
Study has identified a previously unknown regulatory link between WNT9B and FGF signaling during lip and upper jaw development.
Notch (zeige NOTCH1 ELISA Kits) pathway activation can replace the requirement for Wnt4 (zeige WNT4 ELISA Kits) and Wnt9b in mesenchymal-to-epithelial transition of nephron stem cells
wnt9b signaling regulates stem cell proliferation and differention in the developing kidney depending on the activity of the transcription factor Six2 (zeige SIX2 ELISA Kits) in the responding cell.
transgenic overexpression of Wnt9b in the ureteric bud causes reduced branching in multiple founder lines.
expression in most but not all tissues of the 16.5-day embryo; no significant elevation of Wnt9b expression was detected in 29 mouse mammary tumor virus-induced tumors
Wnt9b is a common organizing signal regulating diverse components of the mammalian urogenital system.
Multi-layered mutation in hedgehog (zeige SHH ELISA Kits)-related genes in Gorlin syndrome may affect the phenotype
This association study successfully identified two susceptibility Single Nucleotide Polymorphisms, (WNT9B and PBX1 (zeige PBX1 ELISA Kits)) associated with Mayer-Rokitansky-Kuster-Hauser syndrome risk, both separately and interactively.
Genome association study shows a highly conserved 32 kb intergenic region containing regulatory elements between WNT3 (zeige WNT3 ELISA Kits) and WNT9B in patients with classic bladder exstrophy.
results indicate that mutations in the coding sequence of WNT9B are not responsible for Mullerian duct abnormalities in the Chinese population
Two novel mutations (a missense mutation in exon 1, and one in the 3-UTR (zeige UTS2R ELISA Kits)) may be pathogenic variants in Mayer-Rokitansky-Kuster-Hauser syndrome patients and warrant further functional study.
secretion of WNT2B (zeige WNT2B ELISA Kits) and WNT9B and stabilization of beta-catenin (CTNNB1 (zeige CTNNB1 ELISA Kits)) upon virus infection negatively regulate expression of representative inducible genes IFNB1 (zeige IFNB1 ELISA Kits), IFIT1 (zeige IFIT1 ELISA Kits) and TNF (zeige TNF ELISA Kits) in a CTNNB1 (zeige CTNNB1 ELISA Kits)-dependent effector mechanism
The signals from the stromal fibroblasts cooperate with Wnt9b to promote differentiation of the progenitor cells.
Mutations in the coding sequence of WNT4 (zeige WNT4 ELISA Kits), WNT5A (zeige WNT5A ELISA Kits), WNT7A (zeige WNT7A ELISA Kits), and WNT9B are not responsible for the Mayer-Rokitansky-Kuster-Hauser syndrome.
Ligand for members of the frizzled family of seven transmembrane receptors. Probable developmental protein. May be a signaling molecule which affects the development of discrete regions of tissues. Is likely to signal over only few cell diameters (By similarity).
, Golgi SNAP receptor complex member 2
, wingless-type MMTV integration site family, member 9B
, protein Wnt-9b
, protein Wnt-14b
, wingless-type MMTV integration site 15
, wingless-type MMTV integration site family, member 15
, wingless related MMTV integration site 9B
, wingless-type MMTV integration site 9B