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anti-Human S100A9 Antikörper:
anti-Rat (Rattus) S100A9 Antikörper:
anti-Mouse (Murine) S100A9 Antikörper:
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Human Polyclonal S100A9 Primary Antibody für ICC, IF - ABIN409938
Sumi, Shimizu, Himeno: Involvement of Nrf2 activation in the upregulation of S100A9 by exposure to inorganic arsenite. in International journal of molecular medicine 2012
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Human Polyclonal S100A9 Primary Antibody für WB - ABIN519967
Bianchi, Niemiec, Siler, Urban, Reichenbach: Restoration of anti-Aspergillus defense by neutrophil extracellular traps in human chronic granulomatous disease after gene therapy is calprotectin-dependent. in The Journal of allergy and clinical immunology 2011
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Human Monoclonal S100A9 Primary Antibody für ELISA, WB - ABIN948434
Li, Riau, Setiawan, Mehta, Ti, Tong, Tan, Beuerman: S100A expression in normal corneal-limbal epithelial cells and ocular surface squamous cell carcinoma tissue. in Molecular vision 2011
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Human Monoclonal S100A9 Primary Antibody für EIA, FACS - ABIN111888
Brandtzaeg, Jones, Flavell, Fagerhol: Mac 387 antibody and detection of formalin resistant myelomonocytic L1 antigen. in Journal of clinical pathology 1989
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Human Monoclonal S100A9 Primary Antibody für EIA, FACS - ABIN111889
Delabie, de Wolf-Peeters, van den Oord, Desmet: Differential expression of the calcium-binding proteins MRP8 and MRP14 in granulomatous conditions: an immunohistochemical study. in Clinical and experimental immunology 1990
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Human Monoclonal S100A9 Primary Antibody für EIA, FACS - ABIN238498
Mortezai, Harder, Schnabel, Moors, Gauly, Schlüter, Wagener, Buck: Tandem affinity depletion: a combination of affinity fractionation and immunoaffinity depletion allows the detection of low-abundance components in the complex proteomes of body fluids. in Journal of proteome research 2010
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Human Polyclonal S100A9 Primary Antibody für IHC (p), IHC - ABIN449656
McCormick, Rahimi, Bobryshev, Gaus, Zreiqat, Cai, Lord, Geczy: S100A8 and S100A9 in human arterial wall. Implications for atherogenesis. in The Journal of biological chemistry 2005
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Human Monoclonal S100A9 Primary Antibody für IA, FACS - ABIN2192037
Robinson, Tessier, Poulsom, Hogg: The S100 family heterodimer, MRP-8/14, binds with high affinity to heparin and heparan sulfate glycosaminoglycans on endothelial cells. in The Journal of biological chemistry 2002
Human Monoclonal S100A9 Primary Antibody für ELISA, WB - ABIN519968
Feng, Lee, Chang, Chan, Kuo, Lin, Chung, Kuo, Yu, Lin, Wang, Chou, Huang, Kuo: CD14(+)S100A9(+) monocytic myeloid-derived suppressor cells and their clinical relevance in non-small cell lung cancer. in American journal of respiratory and critical care medicine 2012
Report role of fecal calprotectin (zeige S100A8 Antikörper) assay in the diagnosis and management of inflammatory bowel disease.
S100A8 (zeige S100A8 Antikörper) and S100A9 aggravate Coxsackievirus B3-induced myocarditis and might serve as therapeutic targets in inflammatory cardiomyopathies.
Calprotectin (zeige S100A8 Antikörper) had the highest association with ultrasound synovitis and predicted treatment response. It may thus be considered as a marker for evaluating inflammation and responsiveness in patients with rheumatoid arthritis on biologic disease-modifying anti-rheumatic drug treatment.
Data support a role of S100A9 to initiate and amplify the neutrophilic inflammation in asthma, possibly via inducing IL-1beta (zeige IL1B Antikörper), IL-17 (zeige IL17A Antikörper) and IFN-gamma (zeige IFNG Antikörper).
The median faecal calprotectin (zeige S100A8 Antikörper) level was similar in cases with abdominal pain-related functional gastrointestinal disorders (7.8 mug/g, 95% CI: 7.8-8.4) including those with gastritis, and controls (9.1 mug/g, 95% CI: 7.8-11.3). Faecal calprotectin (zeige S100A8 Antikörper) is not a predictor of gastritis and is similar in children with functional abdominal pain symptoms and in controls.
Data suggest that fecal calprotectin (zeige S100A8 Antikörper) may be a potential biomarker to identify patients with ankylosing spondylitis (AS) at risk of developing inflammatory bowel disease (IBD).
S100A9 induces anti-apoptotic effect on normal and allergic neutrophils by increasing cytokine secretion of monocytes
S100A9, a protein previously described to be involved in modulation of inflammatory response, was found to interact with schistosoma MEG (zeige PTPN4 Antikörper)-14.
Elevated S100A8 (zeige S100A8 Antikörper) and S100A9 gene expression in SP-infected HMEECs and in the middle ear mucosa of OM, minor co-localized with neutrophil markers suggests that middle ear epithelial cell secretion of S100A8 (zeige S100A8 Antikörper) and S100A9 may play a role in the pathogenesis of recurrent and chronic OM
The findings show that reduced PTP1B (zeige PTPN1 Antikörper) responses contribute to disease symptoms in part by enhancing S100A9 expression during viral-associated chronic obstructive pulmonary disease exacerbations.
Data suggest that up-regulation of S100A8 (zeige S100A8 Antikörper) and S100A9 is a key component of early endometrial response to uterine involution in the post-partum period and to prevent chronic endometritis/uterine inflammation; up-regulation can be influenced by diet.
Study verified porcine calprotectin (S100A8 (zeige S100A8 Antikörper)/A9) expression at the protein level in multiple Haemophilus parasuis infected tissues and explored their molecular characterization.
this study shows that S100A9 inhibits B lymphopoiesis by acting on myeloid cells and promoting the release of inflammatory molecules, including IL-1beta (zeige IL1B Antikörper)
An excessive expansion of monocytic-myeloid-derived suppressor cells in S100A9-/- neonates compared to wild-type neonates. This strong baseline expansion was associated with hyperinflammatory responses during endotoxemia and fatal septic courses
Neutrophil-derived S100A8 (zeige S100A8 Antikörper)/A9 promotes thrombocytosis in diabetic mice
S100A9 knockdown almost completely abrogated the effects of IRF7 (zeige IRF7 Antikörper) deletion on granulocytic myeloid-derived suppressor cells development and tumor metastasis.
Perinatal alarmins S100A8 (zeige S100A8 Antikörper) and S100A9 specifically altered MyD88 (zeige MYD88 Antikörper)-dependent proinflammatory gene programs. S100 programming prevented hyperinflammatory responses without impairing pathogen defense. TRIF (zeige RNF138 Antikörper)-adaptor-dependent regulatory genes remained unaffected by perinatal S100 programming and responded strongly to lipopolysaccharide, but were barely expressed.
vitro studies on the mechanisms/pathways involved in leukemic cell differentiation revealed that binding of S100A9 to Toll-like receptor 4 (TLR4 (zeige TLR4 Antikörper)) promotes activation of p38 mitogen-activated protein kinase (zeige MAPK14 Antikörper), extracellular signal-regulated kinases 1 and 2, and Jun N-terminal kinase (zeige MAPK8 Antikörper) signaling pathways, leading to myelomonocytic and monocytic AML (zeige RUNX1 Antikörper) cell differentiation.
these results indicate that CD14 (zeige CD14 Antikörper) is a co-receptor of TLR4 (zeige TLR4 Antikörper) in the S100A9-induced cytokine response.
Mechanistic studies showed that in MDSCs, ATF3 (zeige ATF3 Antikörper) was transrepressed by the GC receptor GR through direct binding to the negative GR-response element. S100A9 is the major transcriptional target of ATF3 (zeige ATF3 Antikörper) in Granulocytic Myeloid-derived Suppressor Cells.
Calprotectin (zeige S100A8 Antikörper) expression in laminar epidermis occurs after extravasation of leukocytes indicating that leukocyte emigration may be an initiating factor in laminar epithelial stress and inflammation in black walnut extract-induced laminitis.
The protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21. This protein may function in the inhibition of casein kinase and altered expression of this protein is associated with the disease cystic fibrosis.
, S100 calcium-binding protein A9 (calgranulin B)
, calgranulin B
, calprotectin L1H subunit
, leukocyte L1 complex heavy chain
, migration inhibitory factor-related protein 14
, protein S100-A9
, S100 calcium binding protein A9 (calgranulin B)
, intracellular calcium-binding protein (MRP14)
, myeloid-related protein 14
, protein MRP-126
, S100 calcium binding protein A9
, S100 calcium-binding protein A9
, neutrophil cytosolic 23 kDa protein
, RNA-binding region containing protein 2-like