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Human HMOX1 ELISA Kit für Sandwich ELISA - ABIN366536
Varol, Uzuno?lu, Erba?, Süt, Say?n: VEGFR-1, Bcl-2, and HO-1 Ratios in Pregnant Women With Hypertension. in Clinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis 2015
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Mouse (Murine) HMOX1 ELISA Kit für Sandwich ELISA - ABIN424042
Wang, Wang: Senescence may mediate conversion of tau phosphorylation-induced apoptotic escape to neurodegeneration. in Experimental gerontology 2015
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Rat (Rattus) HMOX1 ELISA Kit für Sandwich ELISA - ABIN368210
Bijjem, Padi, lal Sharma: Pharmacological activation of heme oxygenase (HO)-1/carbon monoxide pathway prevents the development of peripheral neuropathic pain in Wistar rats. in Naunyn-Schmiedeberg's archives of pharmacology 2013
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study evaluated the time-course of HO-1 and catalase (zeige CAT ELISA Kits) gene expressions in nodules and roots of soybean plants subjected to cadmium treatment
Data suggest expression of hsp32 is up-regulated in kidney epithelial cells upon in vitro exposure to heavy metal water pollutants (Cd, As) or proteasomal inhibitors (MG132, withaferin A, celastrol); heat shock may be synergistic factor.
this study provides a better understanding of the role of the HO-1/Carbon monoxide system in controlling heart function in lower vertebrates.
The induced expression of HO-1 by fenofibrate appeared to be essential for mediating the protective effects of fenofibrate, as the inhibition of HO-1 activity significantly diminished the protective effects of fenofibrate against the GM-mediated death of sensory hair cells in cochlea
Suggest that Gata-1 (zeige GATA1 ELISA Kits) and Nrf2a (zeige NFE2L2 ELISA Kits) play differential roles in regulating the heme degradation enzymes hmox1a/bvra (zeige BLVRA ELISA Kits)/bvrb (zeige BLVRB ELISA Kits) during an early developmental period of heightened cellular stress.
Bach1 (zeige BACH1 ELISA Kits) regulates the liver specificity and transience of the Nrf2a (zeige NFE2L2 ELISA Kits)-dependent induction of hmox1a and that heme mediates this regulation through Bach1 (zeige BACH1 ELISA Kits) inhibition based on its level in each tissue.
Data indicate that Heme oxygenase 1 (HY1) functioned negatively and acted upstream of ABSCISIC ACID-INSENSITIVE4 (ABI4) in drought signaling.
HY1 confers cadmium tolerance by decreasing nitric oxide production and improving iron homeostasis.
Data indicate that AtHO1 (HY1; heme oxygenase-1)-overexpressing plants generated more NO, whereas knock-down of AtHO1 expression reduced the level of nitric oxide (NO) in plants.
HY1 mutant exhibited progressive salt hypersensitivity.
Disrupting the binding of the AtHBP5 to haem oxygenase 1 (HY1) leads to oxidative stress.
Mutation of HY1 causes UV-C hypersensitivity by impairing carotenoid and flavonoid biosynthesis and the down-regulation of antioxidant defence.
HY1 and HY5 additively regulate the expression of light regulated genes and accumulation of chlorophyll and anthocyanin during early seedling development.
HY1 (Heme oxygenase 1) plays an important role in salt acclimation.
all members of the HO1 subfamily (HY1, HO3 and HO4) are active monomeric HOs and can convert haem to BV IXalpha using spinach Fd (ferredoxin) as an electron donor
HO2 (zeige HMOX2 ELISA Kits) has an activity high enough to substitute for HO1 under aerobic conditions.
The induction of HO-1 expression by proteasome inhibition was reduced in PINK1 G309D mutant cells.
there was little difference in HMOX1 genotypes distribution in patients with chronic obstructive pulmonary disease with different severity.
investigation of the possible role of HMOX1 in adipose tissue, adipogenesis and iron metabolism; adipose tissue HMOX1 was increased in obese participants and positively associated with obesity-related metabolic disturbances, and markers of iron accumulation, inflammation and oxidative stress
the HO-1 interactome in prostate cancer, is identified.
induction of HO1 by M. tuberculosis infection may be a mycobacterial virulence mechanism to enhance inflammation and bacterial growth
Data (including data from studies using cells from knockout mice) confirm that the NRF2 (zeige GABPA ELISA Kits)/HMOX1 axis is a critical pathway in the hyperglycemic-mediated dysregulation and inflammatory features of chondrocytes in type 2 diabetes-associated osteoarthritis. (NRF2 (zeige GABPA ELISA Kits) = nuclear factor [erythroid-derived 2]-like 2 protein; HMOX1 = heme oxygenase [decycling] 1)
To promote intracellular iron flux, an iron chaperone appears to be essential for receiving iron from heme catabolism. Data suggest that PCBP2 (zeige PCBP2 ELISA Kits) competes with CPR (zeige POR ELISA Kits) for binding HO1; PCBP2 (zeige PCBP2 ELISA Kits) K homology 3 domain is important for HO1/PCBP2 (zeige PCBP2 ELISA Kits) interaction; heme prompts HO1/CPR (zeige POR ELISA Kits) multimer and decreases HO1/PCBP2 (zeige PCBP2 ELISA Kits) multimer. [PCBP2 (zeige PCBP2 ELISA Kits) = poly(rC) binding protein 2 (zeige PCBP2 ELISA Kits); CPR (zeige POR ELISA Kits) = cytochrome P450 reductase (zeige POR ELISA Kits); HO1 = heme oxidase 1]
Mechanistically, HO-1 induction by all CRLPs requires NADPH oxidase 4 (zeige NOX4 ELISA Kits), with PUFA-containing particles additionally dependent upon mitochondrial reactive oxygen species.These studies define new molecular pathways coupling endothelial cell activation by model CMRs with adaptive regulation of Nrf2 (zeige GABPA ELISA Kits)-dependent HO-1 expression
We identified miR (zeige MLXIP ELISA Kits)-1254 as a negative regulator inhibiting HO-1 translation by directly targeting HO-1 3'UTR (zeige UTS2R ELISA Kits) via its seed region, and suppressing HO-1 transcription via non-seed region-dependent inhibition of transcriptional factor AP-2 alpha (TFAP2A (zeige TFAP2A ELISA Kits)), a transcriptional activator of HO-1.
cells with elevated HO-1 produced less reactive oxygen species, resulting in nuclear localization of HDAC4 (zeige HDAC4 ELISA Kits) and miR (zeige MLXIP ELISA Kits)-206 repression.
findings collectively suggest that miR (zeige MYLIP ELISA Kits)-506 acts as a tumor suppressor via regulation of ROCK1 (zeige ROCK1 ELISA Kits) expression and may thus be a promising therapeutic target for HCC (zeige FAM126A ELISA Kits)
Exogenous administration of CO exacerbated allergic symptoms, resulting in higher levels of both CO and heme oxygenase-1 expression, and a further reduction in H2S levels and CSE expression.
Down-regulation of HO-1 is associated with pulmonary arterial hypertension and right ventricular failure.
The study revealed the involvement of HO-1 in classical swine fever virus proliferation.
The protective properties of flavonoids, such as EGCG, against endothelial inflammation may be regulated in part though induction of HO-1 and subsequent activator protein-1 signaling.
Glutamate (zeige GRIN2C ELISA Kits) regulates Ca2 (zeige CA2 ELISA Kits)+ signals in smooth muscle cells of newborn piglet brain slice arterioles through astrocyte- and heme oxygenase-dependent mechanisms.
obalt protoporphyrin prevents postictal cerebral vascular dysfunction by upregulating HO-1.
Data demonstrate that lipopolysaccharides evoke a heat shock response, with an increase heat shock proteins 70 and Hsp32) and of VEGF (zeige VEGFA ELISA Kits), a specific endothelial cell growth factor (zeige FGF1 ELISA Kits).
Interaction of soluble factors in plasma possibly generated during PICSO are not responsible for upregulation of HO-1 and VEGF mRNA.
the data were consistent with HO-1 acting as an anti-viral factor and these findings suggested that induction of HO-1 may be a useful prevention and treatment strategy against BVDV infection.
These findings suggest that bronchiolar epithelial cells and macrophages up-regulate Nrf2 (zeige NFE2L2 ELISA Kits) expression early in the course of infection, which results in increased expression of HO-1 within these cells.
investigation of molecular mechanisms of microvascular complications in diabetes/hyperglycemia: regulation of HO-1 gene expression in aortic endothelial cells by advanced glycation end products
Sickle blood increases endothelial heme oxygenase activity.
data provide evidence for the involvement of the thioredoxin/thioredoxin (zeige TXN ELISA Kits) receptor system, in the regulation of haem oxygenase-1 expression in aortic endothelial cells during pro-oxidant challenge
Heme oxygenase-1 induction modulates hypoxic pulmonary vasoconstriction through upregulation of ecSOD/SOD3 (zeige SOD3 ELISA Kits).
Results establish an essential role for the receptor CX3CR1 (zeige CX3CR1 ELISA Kits) in gut (zeige GUSB ELISA Kits) macrophages in resolving inflammation in the intestine, where it helps protects against colitis-associated cancer by regulating HMOX-1 expression.
study demonstrates that human respiratory syncytial virus (hRSV) infection can be modulated by the expression of HO-1 both in vitro and in vivo; thus, HO-1 activity may play a critical role during hRSV infection
This is the first study to dissect the mechanism by which the antiadipogenic and anti-inflammatory lipid, Epoxyeicosatrienoic acid, induces the PGC-1alpha (zeige PPARGC1A ELISA Kits) signaling cascade and reprograms the adipocyte phenotype by regulating mitochondrial function and HO-1 expression.
The findings suggest that HO-1 by modulating TLR9 (zeige TLR9 ELISA Kits) expression in PMCs promotes pleural innate immunity in MRSA (zeige MSR1 ELISA Kits) empyema.
HO-1 regulates not only cytokine/chemokine (zeige CCL1 ELISA Kits) production in pregnant uteri but also myeloid cell receptor numbers.
Administration of EET alters Wnt1 (zeige WNT1 ELISA Kits), NOV (zeige NOV ELISA Kits), and HO-1 signaling to prevent obesity-induced cardiomyopathy in obese mice.
Studied the immunosuppressive role of HO-1 in sepsis with a focus on its effects on helper T-cell (Th) differentiation and regulatory T cells; found overexpressed HO-1 contributes to sepsis-induced immunosuppression by inducing a Th1 (zeige HAND1 ELISA Kits) to Th2 cytokine shift and enhancing Treg function.
Data indicate that chlorogenic acid (CGA (zeige CGA ELISA Kits)) protected osteoblast MC3T3-E1 cells against oxidative damage via PI3K/Akt (zeige AKT1 ELISA Kits)-mediated activation of Nrf2 (zeige NFE2L2 ELISA Kits)/HO-1 pathway, which may be an effective drug in treatment of osteoporosis.
our data suggest that the B. pseudomallei-mediated induction of HO-1 and the release of its metabolite carbon dixode impair bacterial clearance in macrophages and during murine melioidosis.
HO-1 activation mediates early brain damage after ICH (zeige ACE ELISA Kits) (intracerebral hemorrhage) but promotes neurologic function recovery in the later stage of ICH (zeige ACE ELISA Kits)
Kidneys from circulation-restricted fetuses showed reduced heme oxygenase-1 mRNA.
Ligustrazine injection possesses notable protective effects on ischemia/reperfusion injury in rabbits by increasing the expression of HO-1 in lung.
Results add new evidence for the importance of HO-1 in the genesis and development of atherosclerosis and provide several possible mechanisms underlying the anti-atherosclerosis effects of HO-1
the effect of HO-1 on the progression and stabilization of vulnerable plaques and the possible mechanism
Heme-L-lysinate could attenuate atherosclerotic progression through upregulating HO-1 and HSP70 (zeige HSP70 ELISA Kits) expression and increasing CO production.
These results suggest that HO-1 is important in limiting in-stent stenosis and can be regarded as a new therapeutic target.
Statins showed anti-atherosclerotic effects mediated by HO-1/eNOS (zeige NOS3 ELISA Kits), restoring the [NO]/[ONOO(-)] imbalance and reducing lipid peroxidation.
HO-1/CO system was activated and may be one of the protective signal pathway during pulmonary ischemia-reperfusion injury in rabbits.
HO-1 contributes to vascular repair by increasing circulating endothelial progenitor cells (EPCs) derived from the bone marrow.
Heme oxygenase, an essential enzyme in heme catabolism, cleaves heme to form biliverdin, which is subsequently converted to bilirubin by biliverdin reductase, and carbon monoxide, a putative neurotransmitter. Heme oxygenase activity is induced by its substrate heme and by various nonheme substances. Heme oxygenase occurs as 2 isozymes, an inducible heme oxygenase-1 and a constitutive heme oxygenase-2. HMOX1 and HMOX2 belong to the heme oxygenase family.
heme oxygenase (decycling) 1
, heme oxygenase 1
, heme oxygenase
, Heme oxygenase
, heat shock protein, 32-kD
, heat shock protein 32
, heme oxygenase (decyclizing) 1
, P32 protein
, heme oxygenase-1