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results define molecular determinants that control the production of Lin(Ub)n-Bcl10, an important signaling intermediate in TCR and oncogenic CARD11 (zeige CARD11 ELISA Kits) signaling.
Psoriasis mutations disrupt CARD14 (zeige CARD14 ELISA Kits) autoinhibition promoting BCL10-MALT1 (zeige MALT1 ELISA Kits)-dependent NF-kappaB (zeige NFKB1 ELISA Kits) activation
BCL10 promoted DNA double-strand breaks repair, enhancing cell survival after DNA damage.
Data show that caspase recruitment domain-containing protein 11 (zeige CARD11 ELISA Kits)/B-cell CLL/lymphoma 10/mucosa-associated lymphoid tissue lymphoma translocation gene 1 (zeige MALT1 ELISA Kits) signaling drives lymphoproliferation (zeige FAS ELISA Kits) through NF-kappa B (zeige NFKB1 ELISA Kits) and c-Jun N-terminal kinase activation.
BCL10 is not essential for actin polymerization after fibroblast FcgammaR stimulation.
characterization of zebrafish (Danio rerio) Bcl10
we identified BCL10 as a bona fide target of BCR (zeige BCR ELISA Kits)-induced linear ubiquitylation and demonstrated an important role of the linear ubiquitin ligase HOIP (zeige RNF31 ELISA Kits) in BCR (zeige BCR ELISA Kits)-induced phosphorylation
The results of this study indicate that inherited BCL10 deficiency should be considered in patients with combined immunodeficiency with B cell, T cell, and fibroblast defects.
B-cell lymphoma/leukemia 10 promotes oral cancer progression through STAT1 (zeige STAT1 ELISA Kits)/ATF4 (zeige ATF4 ELISA Kits)/S100P (zeige S100P ELISA Kits) signaling pathway.
BCL10 induces cleavage of MALT1 (zeige MALT1 ELISA Kits) at R149 in 293T cells.
Data show that BCL10 was located in cytoplasm in Pig Kidney Epithelial (PK15) cells.
Data found that, BCL10 expressed in high level in swine spleen, and with a modest level in thymus, brain and lymph node; low level mRNA was expressed in liver and not detectable level in kidney.
Inherited BCL10 deficiency impairs hematopoietic and nonhematopoietic immunity.
cell receptor-dependent activation of mTOR signaling in T cells is mediated by Carma1 and MALT1, but not Bcl10.
Trehalose 6,6'-dimycolate-induced Mincle (zeige CLEC4E ELISA Kits) expression is dependent on Dectin-3-mediated NF-kappaB (zeige NFKB1 ELISA Kits) activation through the CARD9 (zeige CARD9 ELISA Kits)-BCL10-MALT1 (zeige MALT1 ELISA Kits) complex.
Bcl10 mediates angiotensin II-induced cardiac damage and electrical remodeling.
TCR-stimulated activation of NF-kappaB requires the assembly of cytosolic p62-Bcl10-Malt1-IKK signalosomes, which may ensure highly regulated activation of NF-kappaB in response to TCR engagement.
Bcl10 is an essential regulator for A20 (zeige TNFAIP3 ELISA Kits) gene expression.
Data indicate that caspase recruitment domain-containing protein 11 (CARD11 (zeige CARD11 ELISA Kits)) is involved in the pathogenesis of collagen-induced arthritis (CIA (zeige NCOA5 ELISA Kits)) by formation of the CARD11 (zeige CARD11 ELISA Kits)/Bcl10 complex and enhancement of the Th17 cell response.
Findings demonstrate a Bcl10 requirement for maximum development of carrageenan-induced inflammation and lack of complete suppression by IL-10 (zeige IL10 ELISA Kits) of carrageenan-induced inflammation.
Bcl10 links saturated fat overnutrition with hepatocellular NF-kB activation and insulin (zeige INS ELISA Kits) resistance.
identify Bcl10 as an early coordinator of NF-kappaB-mediated immune response with endosomal trafficking and signaling to F-actin remodeling
This gene was identified by its translocation in a case of mucosa-associated lymphoid tissue (MALT) lymphoma. The protein encoded by this gene contains a caspase recruitment domain (CARD), and has been shown to induce apoptosis and to activate NF-kappaB. This protein is reported to interact with other CARD domain containing proteins including CARD9, 10, 11 and 14, which are thought to function as upstream regulators in NF-kappaB signaling. This protein is found to form a complex with MALT1, a protein encoded by another gene known to be translocated in MALT lymphoma. MALT1 and this protein are thought to synergize in the activation of NF-kappaB, and the deregulation of either of them may contribute to the same pathogenetic process that leads to the malignancy.
B-cell lymphoma/leukemia 10
, CARD containing molecule enhancing NF-kB
, CARD-containing apoptotic signaling protein
, CARD-containing molecule enhancing NF-kappa-B
, CARD-containing proapoptotic protein
, CED-3/ICH-1 prodomain homologous E10-like regulator
, caspase-recruiting domain-containing protein
, cellular homolog of vCARMEN
, mammalian CARD-containing adapter molecule E10
, B-cell leukemia/lymphoma 10
, B-cell lymophoma 10 protein
, B-cell CLL/lymphoma 10
, B cell lymphoma/leukemia 10
, CARD-like apoptotic protein