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anti-Human EPH Receptor A4 Antikörper:
anti-Rat (Rattus) EPH Receptor A4 Antikörper:
anti-Mouse (Murine) EPH Receptor A4 Antikörper:
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Chicken Monoclonal EPH Receptor A4 Primary Antibody für IF, IP - ABIN967994
Becker, Gilardi-Hebenstreit, Seitanidou, Wilkinson, Charnay: Characterisation of the Sek-1 receptor tyrosine kinase. in FEBS letters 1995
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Human Monoclonal EPH Receptor A4 Primary Antibody für ELISA, WB - ABIN1724712
Walkenhorst, Dütting, Handwerker, Huai, Tanaka, Drescher: The EphA4 receptor tyrosine kinase is necessary for the guidance of nasal retinal ganglion cell axons in vitro. in Molecular and cellular neurosciences 2001
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Human Polyclonal EPH Receptor A4 Primary Antibody für IHC (p) - ABIN2473463
Theil, Frain, Gilardi-Hebenstreit, Flenniken, Charnay, Wilkinson: Segmental expression of the EphA4 (Sek-1) receptor tyrosine kinase in the hindbrain is under direct transcriptional control of Krox-20. in Development (Cambridge, England) 1998
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These results demonstrate a novel role for SORLA (zeige SORL1 Antikörper) as a physiological and pathological EphA4 modulator.
The expression of both EphA4-FL and EphA4-N was significantly higher in the nervous tissue of SOD1 (zeige SOD1 Antikörper)(G93A) compared to wild-type mice suggesting that both forms are modulated during the disease process.
the PI3K (zeige PIK3CA Antikörper)/AKT (zeige AKT1 Antikörper), Wnt (zeige WNT2 Antikörper)/beta-catenin (zeige CTNNB1 Antikörper) signaling pathways as well as ERK1/2 (zeige MAPK1/3 Antikörper) downstream of EPHA4 receptor activation, play an important role in the regulation of events related with the EMT (zeige ITK Antikörper) development, which may be associated with the therapeutic failure in rectal cancer after radiotherapy.
Molecular interactions of EphA4, growth hormone receptor (zeige GHR Antikörper), Jak2 (zeige JAK2 Antikörper), and STAT5B (zeige STAT5B Antikörper) have been described.
Findings demonstrated that mutant alpha2-chimaerin and EphA4 have different genetic interactions in distinct motor neuron pools: abducens neurons use bidirectional ephrin signaling via mutant alpha2-chimaerin to direct growth, while cervical spinal neurons use only ephrin forward signaling
Reduced EphA4 expression is associated with EBV-associated B lymphoma.
No difference was found in the expression of EPHA4 in morphologically normal glands, HGPIN, or prostatic cancer.
we supposed that EphA4 interacted with CDK5 (zeige CDK5 Antikörper) and promoted its expression which in turn enhanced p-AKT (zeige AKT1 Antikörper) expression and promoted cell adhesion-mediated drug resistance in multiple myeloma.
EphA4 was reduced in breast carcinoma, which is associated with high grade, advanced TNM (zeige ODZ1 Antikörper) stage, lymph node metastasis, and poor outcome of patients
Host EphA4 expression regulates cancer development mainly via EphA4-mediated IGF1 (zeige IGF1 Antikörper) synthesis signal.
findings suggest a role for EphA4 in shaping cortical oscillations during sleep that is independent from sleep need
The present study demonstrates that EphA4 and EphA7 (zeige EPHA7 Antikörper) receptors, despite their similar structure, have distinct in vivo effects on corticothalamic system projections into the ventrobasal complex /medial division of the posterior nuclear group.
These results suggest that EphA4, a novel and promising target for treatment, exacerbates EBI (zeige TBL1X Antikörper) through an Ephexin-1 (zeige NGEF Antikörper)/ROCK2 (zeige ROCK2 Antikörper) pathway after subarachnoid hemorrhage.
Our studies show that forward signaling through the EphA4 tyrosine kinase receptor (zeige KDR Antikörper), mediated by ephrins expressed by subpopulations of neuroblasts and astrocytes, is required for compact, directional organization of neuroblasts and astrocytes within the pathway and efficient transit of neuroblasts through the anterior forebrain to the olfactory bulb.
Findings suggest a putative novel mechanism for desipramine to modulate long-term potentiation through the regulation of the ephrinA3/EphA4 signaling pathway
Delineation of the specific mutation in EphA4 in this strain is important for further functional studies, such as protein-protein interactions, immunostaining and gene compensatory studies, investigating the mechanism underlying the effects of altered function of Eph (zeige EPHA1 Antikörper) family of receptor tyrosine kinases on phenotype.
the results of the present study demonstrated that angiogenesis occurs, and that the molecules EphA4 and ephrin-A5 (zeige EFNA5 Antikörper) are expressed in the hippocampal CA1 (zeige CA1 Antikörper) and CA3 (zeige CA3 Antikörper) areas throughout epileptogenesis. PECAM-1 (zeige PECAM1 Antikörper) may detect epileptic microvessel patterns in the hippocampi of mice and EphA4 may contribute to the microvessel plasticity via the ephrin-A5 (zeige EFNA5 Antikörper) signaling pathway.
expression of Eph (zeige EPHA1 Antikörper) A1, A2, A4, and A7 was strongly detected in endometrial epithelial cells during early pregnancy.
The EphA4 gene is significantly associated with litter size in pigs.
EphA4-dependent Brachyury (zeige TBX1 Antikörper) expression is required for dorsal mesoderm involution in the Xenopus gastrula.
show that EphA4 and its putative ligand, ephrin-A1 (zeige EFNA1 Antikörper) are expressed in a complementary manner in the involuting mesodermal and non-involuting ectodermal layers of early gastrulae, respectively
Downregulation of EphA4a compromises actomyosin cables and cells with different rhombomeric identity intermingle, and the phenotype is rescued enhancing myosin II activity.
EphA4 (zeige EPHA3 Antikörper) is required for cell adhesion and rhombomere-boundary formation in the zebrafish.
Data show that EfnB2a (zeige EFNB2 Antikörper) is required in developing hindbrain for normal cell affinity and that EphA4 (zeige EPHA3 Antikörper) and EfnB2a (zeige EFNB2 Antikörper) regulate cell affinity independently within their respective rhombomeres.
This gene belongs to the ephrin receptor subfamily of the protein-tyrosine kinase family. EPH and EPH-related receptors have been implicated in mediating developmental events, particularly in the nervous system. Receptors in the EPH subfamily typically have a single kinase domain and an extracellular region containing a Cys-rich domain and 2 fibronectin type III repeats. The ephrin receptors are divided into 2 groups based on the similarity of their extracellular domain sequences and their affinities for binding ephrin-A and ephrin-B ligands.
, EPH-like kinase 8
, TYRO1 protein tyrosine kinase
, ephrin type-A receptor 4
, receptor protein-tyrosine kinase HEK8
, tyrosine-protein kinase TYRO1
, tyrosine-protein kinase receptor SEK
, ephrin receptor epsilon
, EPH receptor A4
, ephrin receptor EphA4
, ephrin receptor EphA4-like
, ephrin type-A receptor 4-like
, tyrosine-protein kinase receptor MPK-3
, tyrosine-protein kinase receptor SEK-1
, ephrin type-A receptor 4-B
, receptor tyrosine kinase
, tyrosine-protein kinase receptor PAG
, eph receptor A3
, eph-like kinase 2
, eph-like receptor tyrosine kinase 1
, ephrin type-A receptor 4a
, tyrosine-protein kinase receptor ZEK2