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anti-Human TEAD4 Antikörper:
anti-Mouse (Murine) TEAD4 Antikörper:
anti-Rat (Rattus) TEAD4 Antikörper:
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Human Monoclonal TEAD4 Primary Antibody für IP, RNAi - ABIN563135
Benhaddou, Keime, Ye, Morlon, Michel, Jost, Mengus, Davidson: Transcription factor TEAD4 regulates expression of myogenin and the unfolded protein response genes during C2C12 cell differentiation. in Cell death and differentiation 2012
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Cow (Bovine) Polyclonal TEAD4 Primary Antibody für IF, WB - ABIN2777935
Appukuttan, McFarland, Davies, Atchaneeyasakul, Zhang, Babra, Pan, Rosenbaum, Acott, Powers, Stout: Identification of novel alternatively spliced isoforms of RTEF-1 within human ocular vascular endothelial cells and murine retina. in Investigative ophthalmology & visual science 2007
Show all 2 Pubmed References
Cow (Bovine) Polyclonal TEAD4 Primary Antibody für IHC, WB - ABIN2780491
Chen, Baty, Maeda, Brooks, Baker, Ueyama, Gursoy, Saba, Salama, London, Stewart: Transcription enhancer factor-1-related factor-transgenic mice develop cardiac conduction defects associated with altered connexin phosphorylation. in Circulation 2004
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the transcriptional regulators in the Hippo pathway, Tead4 and Yap1 (zeige YAP1 Antikörper), are required for general vertebrate epimorphic regeneration as well as for organ size control in appendage regeneration
Osmotic stress promotes TEAD4 cytoplasmic translocation via p38 MAPK (zeige MAPK14 Antikörper) in a Hippo-independent manner. Stress-induced TEAD inhibition predominates YAP (zeige YAP1 Antikörper)-activating signals and selectively suppresses YAP (zeige YAP1 Antikörper)-driven cancer cell growth.
The transcription factor TEAD4 regulates a pro-metastasis transcription program in a YAP (zeige YAP1 Antikörper)-independent manner in CRC (zeige CALR Antikörper), thus providing a novel mechanism of TEAD4 transcriptional regulation and its oncogenic role in CRC (zeige CALR Antikörper), independently of the Hippo pathway.
our work provides a structural basis for understanding the regulatory mechanism of TEAD4-mediated gene transcription
Our results suggest that TEAD4 plays a role in the pathophysiology of atypical teratoid/rhabdoid tumor, which represents a new insight into the biology of this aggressive tumor
It was found that the TEAD4-YAP (zeige YAP1 Antikörper) complex in the nuclei may be related closely to transcriptions of G1 arrest-related genes.
Collectively, these results indicate that human papillomavirus 16 E6 induces upregulation of APOBEC3B (zeige APOBEC3B Antikörper) through increased levels of TEADs, highlighting the importance of the TEAD-APOBEC3B (zeige APOBEC3B Antikörper) axis in carcinogenesis.
Tead4 cooperates with AP1 (zeige FOSB Antikörper) transcription factors to coordinate target gene transcription.
TEAD4 and KLF5 (zeige KLF5 Antikörper), in collaboration, promoted triple negative breast cancer cell proliferation and tumor growth in part by inhibiting p27 (zeige PAK2 Antikörper) gene transcription
potential anti-oxidation gene and can prevent H2O2-induced endothelial cell oxidative damage by activating Klotho (zeige KL Antikörper)
TEAD4 overexpression induced p16 (zeige CDKN2A Antikörper) in HAoSMCs homozygous for the nonrisk coronary disease allele, but not for the risk allele.
AP-1 (zeige JUN Antikörper)- and TEAD4-associated cis (zeige CISH Antikörper)-regulatory elements form hubs for multiple signalling-responsive transcription factors and define the cistrome that regulates vascular and hematopoietic development by extrinsic signals.
Data show that TEAD family of transcription factors Tead1 (zeige TEAD1 Antikörper) and Tead4-regulated gene expression in differentiating primary myoblasts.
Dual-luciferase reporter gene analysis showed that RTEF-1 is a direct target of mir-125a-5p, which regulates angiogenesis by repressing RTEF-1 expression and modulating eNOS and VEGF expression.
TEAD4 establishes the energy homeostasis essential for blastocoel formation.
These results show that RTEF-1-stimulated IGFBP-1 (zeige IGFBPI Antikörper) expression may be central to the mechanism by which RTEF-1 attenuates blood glucose levels.
Vgll1 (zeige VGLL1 Antikörper) interacts with TEAD4 in a manner similar to the transcription coactivators, as well as oncogenes YAP (zeige YAP1 Antikörper) and TAZ (zeige TAZ Antikörper), despite having a varied primary sequence. Vgll1 (zeige VGLL1 Antikörper) has the potential to promote cancer progression.
endothelial-specific RTEF-1 overexpressing mice had enhanced angiogenic sprouting and vascular structure remodeling, resulting in the formation of a denser and more highly interconnected superficial capillary plexus
Data suggest that altered subcellular localization of TEAD4 in blastomeres dictates first mammalian cell fate specification.
TEAD factors directly induce Myogenin (zeige MYOG Antikörper), CDKN1A (zeige CDKN1A Antikörper) and Caveolin 3 (zeige CAV3 Antikörper) expression to promote myoblast differentiation.
Gata3 (zeige GATA3 Antikörper) and Cdx2 (zeige CDX2 Antikörper) can act in parallel pathways downstream of Tead4 to induce the expression of common and independent targets in the trophoblast lineage, whereas Oct4 (zeige POU5F1 Antikörper) is required for continued repression of trophoblast fate in the embryonic lineage
This gene product is a member of the transcriptional enhancer factor (TEF) family of transcription factors, which contain the TEA/ATTS DNA-binding domain. It is preferentially expressed in the skeletal muscle, and binds to the M-CAT regulatory element found in promoters of muscle-specific genes to direct their gene expression. Alternatively spliced transcripts encoding distinct isoforms, some of which are translated through the use of a non-AUG (UUG) initiation codon, have been described for this gene.
TEA domain family member 4
, M-CAT binding factor
, M-CAT-binding factor
, transcriptional enhancer factor TEF-3
, related transcription enhancer factor 1B
, transcription factor 13-like 1
, transcription factor RTEF-1
, transcriptional enhancer factor 1-related
, transcriptional enhancer factor 3
, transcriptional enhancer factor 1-related protein
, ETF-related factor 2
, ETF-related factor-2
, FGF-regulated 19
, TEF-1-related factor 1
, TEF-1-related factor FR-19