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Mouse (Murine) IL23A ELISA Kit für Sandwich ELISA - ABIN921111
Oppmann, Lesley, Blom, Timans, Xu, Hunte, Vega, Yu, Wang, Singh, Zonin, Vaisberg, Churakova, Liu, Gorman, Wagner, Zurawski, Liu, Abrams, Moore, Rennick, de Waal-Malefyt, Hannum, Bazan, Kastelein: Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12. in Immunity 2001
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This study is the first to demonstrate an effect of HIV on the IL-23 signaling pathway in Th17 cells. We show that in vitro and in vivo HIV infection results in impaired IL-23 signaling which is not reversed by HAART nor is it a result of reduced receptor expression, suggesting that HIV interferes with IL-23-activated signaling pathways
We conclude that variants in IL-23A and IL-23R (zeige IL23R ELISA Kits) genes were associated with the risk of multiple sclerosis or other inflammatory demyelinating diseases.
results confirm the role of IFN-gamma (zeige IFNG ELISA Kits) and support the involvement of IL-23/Th17-related cytokines in BCC pathogenesis and in the inflammatory response during IMQ and MAL-PDT (zeige TWIST1 ELISA Kits) treatments
An increase in CSF (zeige CSF2 ELISA Kits) mean levels of IL-23 was observed in amyotrophic lateral sclerosis patients.
results show that IL-23 accounts for the main aspects of human and murine lupus including the expansion of double negative T cells, decreased IL-2 (zeige IL2 ELISA Kits), and increased IL-17 (zeige IL17A ELISA Kits) production
Serum Levels of IL-17A (zeige IL17A ELISA Kits) and IL-23 Are Associated with Decreased Vitamin D3 and Increased Pain in Osteoarthritis
this study shows that latent cytomegalovirus induces IL-23. IL-23 might be an inflammatory mediator of latent cytomegalovirus infection in patients with rnd-stage renal diseases, and predisposes patients for post-transplant cytomegalovirus reactivation
These finding may provide a molecular mechanism involved in sustained ER stress in AS BdCs stimulates the activation of RUNX2 (zeige RUNX2 ELISA Kits) and C/EBPbeta (zeige CEBPB ELISA Kits) genes, leading to IL-23 production.
IL-23 released by keratinocytes in response to endogenous TLR4 (zeige TLR4 ELISA Kits) ligands causes skin DCs, which selectively express IL-23R (zeige IL23R ELISA Kits), to up-regulate their endogenous IL-23 production and drive an IL-22 (zeige IL22 ELISA Kits) response in naive CD4 (zeige CD4 ELISA Kits)(+) T cells that mediates epidermal thickening.
The link between increased expression of TH17 pathway cytokines and clinical disease severity raises the possibility of a new therapeutic paradigm of targeted IL-17 (zeige IL17A ELISA Kits)/IL-23 intervention for patients with ichthyosis (zeige LBR ELISA Kits)
this study shows that IL-23 but not IL-1 (zeige IL1A ELISA Kits) contributes to the IL-17A (zeige IL17A ELISA Kits) expression induced by subacute O3 exposure
In mice overexpressing IL23, enthesis-resident gamma/delta T cells accumulate in the enthesis, ciliary body, and aortic valve.
we describe here a new IL-12 member IL-39 (IL-23p19/Ebi3) secreted by activated B cells that mediate inflammatory responses in lupus-like mice. Thus, IL-39 might contribute to immunopathogenic mechanisms of SLE.
These data identify the important contributions of IL-23 to the development of elastase-induced pulmonary inflammation and emphysema.
The inflammatory phenotype in barrier-defective skin is shaped by counterregulation between the TSLP (zeige TSLP ELISA Kits)/type 2 and IL-23/type 17 axes.
IL-23 is another important bronchial epithelial cell-driven cytokine which may contribute to the development of house dust mite allergic sensitization and asthma.
this study shows that elevated iserum IL-23 is associated with mortality in lipopolysaccharide/d-galactosamine -induced acute liver injury, and that IL-23 neutralizing antibodies attenuated liver injury by reducing the expression of Th17-related inflammatory cytokines, neutrophil chemoattractants and stabilization factors within the liver tissue
IL-23 is a considerable molecular in tumor progression, which simultaneously facilitates processes of pro-tumor inflammation, such as angiogenesis, immunosuppressive cytokines as well as infiltrations of M2 macrophages and neutrophils, and suppresses antitumor immune responses through reduction of CD4 (zeige CD4 ELISA Kits)(+) T cells and CD8 (zeige CD8A ELISA Kits)(+) T cells
These results provide genetic information about both swine IL-23 the IL-23 receptor alpha (IL-23Ralpha) and IL-12 receptor beta1 (IL-12Rbeta1), which allows for better understanding of interleukin systems involved in pig immunity.
cloning and characterization of interleukin-17 (zeige IL17A ELISA Kits) expressed gene sequence from mRNA obtained from intestinal tissue and interleukin-23 expressed gene sequence from mRNA obtained from peripheral blood mononuclear cells
This study examined effects of in vitro exposure to solutions of hay (zeige GTF2H5 ELISA Kits) dust, lipopolysaccharides, or beta-glucan on cytokine expression in pulmonary mononuclear cells isolated from healthy horses and horses with recurrent airway obstruction.
This gene encodes a subunit of the heterodimeric cytokine interleukin 23 (IL23). IL23 is composed of this protein and the p40 subunit of interleukin 12 (IL12B). The receptor of IL23 is formed by the beta 1 subunit of IL12 (IL12RB1) and an IL23 specific subunit, IL23R. Both IL23 and IL12 can activate the transcription activator STAT4, and stimulate the production of interferon-gamma (IFNG). In contrast to IL12, which acts mainly on naive CD4(+) T cells, IL23 preferentially acts on memory CD4(+) T cells.
IL-23 subunit alpha
, JKA3 induced upon T-cell activation
, interleukin 23 p19 subunit
, interleukin-23 subunit alpha
, interleukin-23 subunit p19
, interleukin-six, G-CSF related factor
, IL-23 p19 subunit
, Interleukin-23 p19 subunit
, interleukin 23 p19