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Human SEMA3A ELISA Kit für Sandwich ELISA - ABIN481720
Vadasz, Haj, Halasz, Rosner, Slobodin, Attias, Kessel, Kessler, Neufeld, Toubi: Semaphorin 3A is a marker for disease activity and a potential immunoregulator in systemic lupus erythematosus. in Arthritis research & therapy 2012
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Human SEMA3A ELISA Kit für Sandwich ELISA - ABIN858267
Treps, Edmond, Harford-Wright, Galan-Moya, Schmitt, Azzi, Citerne, Bidère, Ricard, Gavard: Extracellular vesicle-transported Semaphorin3A promotes vascular permeability in glioblastoma. in Oncogene 2015
EV-delivered Sema3A orchestrates loss of barrier integrity in glioblastoma
IL6 (zeige IL6 ELISA Kits) underexpression is associated with Breast Cancer.
We also demonstrate that the effects of RET (zeige RET ELISA Kits) and NRG1 (zeige NRG1 ELISA Kits) are universal across European and Asian ancestries. In contrast, we detected a European-specific association of a low-frequency variant, rs80227144, in SEMA3. Conditional analyses on the lead SNPs revealed a secondary association signal, corresponding to an Asian-specific, low-frequency missense variant encoding RET (zeige RET ELISA Kits) p.Asp489Asn
Semaphorin3A serum concentrations were similar in ankylosing spondylitis patients and controls and were not altered on treatment in anti-TNF (zeige TNF ELISA Kits) agents.
The self-renewal ability abolished by Sema3a knockdown could not be recovered by exogenous addition of recombinant SEMA3A. In addition, the activity of mammalian target of rapamycin (zeige FRAP1 ELISA Kits) complex 1 (mTORC1) and the expression of its substrate p70S6K1 (zeige RPS6KB1 ELISA Kits) were also decreased. These results demonstrate that Sema3a is a potential therapeutic target in eradication of CSCs.
Study shows a significant reduction of the serum Sema3A level and its mRNA expression in peripheral blood mononuclear cells in patients with relapsing-remitting multiple sclerosis compared with normal individuals.
Sema3A expression on T and B regulatory lymphocytes is low in familial Mediterranean fever (zeige MEFV ELISA Kits) patients during attack and in smoldering disease compared to the expression in remission and healthy controls.
Tumor cells decrease Sema3a mRNA expression in osteoblasts in an mTORC1-dependent manner.
Semaphorin-3a, neuropilin-1 and plexin-A1 are axonal guidance molecules that have been recently implicated in regulating bone metabolism.
Sema3A and sema7A (zeige SEMA7A ELISA Kits) expression correlated with the inflammatory activity of the multiple sclerosis (MS) lesions, suggesting their involvement in the immunological process that takes place in MS.
We show that PTP-3, a LAR (zeige PTPRF ELISA Kits) homolog in Caenorhabditis elegans, participates in Sema2A-regulated axon guidance. PTPdelta, a member of vertebrate LAR (zeige PTPRF ELISA Kits) class PTPs (zeige PTS ELISA Kits), is involved in Sema3A-regulated cortical dendritic growth. In Sema3A signaling, PTPdelta activates Fyn (zeige FYN ELISA Kits) and Src (zeige SRC ELISA Kits) kinases by dephosphorylating their C-terminal Tyr (zeige TYR ELISA Kits) residues.
Investigated the effects of BotoxA-induced paralysis on the sprouting capacity of neuromuscular junctions (NMJs) in the K108N-SEMA3A mutant, and observed no change in the differential neuronal plasticity found at NMJs on fast-fatigable or slow muscle fibers due to the presence of the SEMA3A mutant protein.
The aim was to elucidate whether ectopic muscles are a result of misguidance of myoblast precursors due to the loss of Sema3A-Npn-1 (zeige NRP1 ELISA Kits) signaling.
These findings suggest that by interacting with PlexA4 (zeige PLXNA4 ELISA Kits), TrkA (zeige NTRK1 ELISA Kits) plays a crucial role in redirecting local Sema3A signaling to retrograde axonal transport, thereby regulating dendritic GluA2 (zeige GRIA2 ELISA Kits) localization and patterning.
Findings indicate that Rho and ROCK knockout may improve the behavior of mice and prevent MPTP (zeige PTPN2 ELISA Kits)-induced dopaminergic neurons damage by regulating Sema3A, PlexinA and NRP-1 (zeige NRP1 ELISA Kits) in a mouse model of Parkinson's disease.
Sema3A signaling exerts its oncogenic effect by promoting an mTORC1-mediated metabolic shift from oxidative phosphorylation to aerobic glycolysis.
Semaphorin 3A (Sema3A) is a protein secreted during the development of the nervous system that acts as a cue for axon induction .
this paper shows that Nav1.7 (zeige SCN9A ELISA Kits), by coupling with CRMP1 (zeige CRMP1 ELISA Kits), mediates the axonal retrograde signaling of Sema3A in axonal guidance
miR (zeige MLXIP ELISA Kits)-30c and sema3A regulate adult neurogenesis by controlling proliferation and differentiation of stem cells in the subventricular zone.
These findings indicate that endothelial cell-derived Sema3A exerts repelling functions on VEGF-A (zeige VEGFA ELISA Kits)-induced tip cell filopodia and that a lack of this signaling cannot be rescued by paracrine sources of Sema3A.
This gene is a member of the semaphorin family and encodes a protein with an Ig-like C2-type (immunoglobulin-like) domain, a PSI domain and a Sema domain. This secreted protein can function as either a chemorepulsive agent, inhibiting axonal outgrowth, or as a chemoattractive agent, stimulating the growth of apical dendrites. In both cases, the protein is vital for normal neuronal pattern development. Increased expression of this protein is associated with schizophrenia and is seen in a variety of human tumor cell lines. Also, aberrant release of this protein is associated with the progression of Alzheimer's disease.
, sema domain, immunoglobulin domain (Ig), short basic domain, secreted, (semaphorin) 3A
, semaphorin 3A
, collapsin 1
, semaphorin D
, semaphorin III
, semaphorin L
, sema D
, sema III
, Semaphorin 3a
, sema domain, immunoglobulin domain (Ig), short basic domain, sec