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Human TMSB4X ELISA Kit für Sandwich ELISA - ABIN511647
Xiao, Qu, Ge, Wang, Wu, Xu, Chen: Depletion of thymosin ?4 promotes the proliferation, migration, and activation of human hepatic stellate cells. in Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2014
Tbeta4 exerts a protective effect on murine colitis, indicating that recombinant adeno-associated virus carrying thymosin beta4 could potentially be developed into a promising agent for the therapy of inflammatory bowel diseases.
Results showed that the TMSB4 pseudogene, lncRNA-MSR (zeige MTRR ELISA Kits), was upregulated in the damaged cartilage and was activated in chondrocytes in response to mechanical stress. Furthermore, lncRNA-MSR (zeige MTRR ELISA Kits) regulated the expression of TMSB4 by competing with miRNA-152 in chondrocytes.
Tbeta4 gene silencing in A549 and H1299 cells inhibited cell proliferation, migration, and invasion in vitro and decreased tumor growth in vivo. Mechanistic investigations revealed a significant decrease in Notch1 (zeige NOTCH1 ELISA Kits) activation in Tbeta4 gene-silenced cells.
Developed an innovative biotechnological method for thymosin beta4 production that employs the peptide acetylation in vivo.
Serum thymosin beta4 concentrations were correlated with the occurrence and severity of obstructive sleep apnea syndrome.
this study demonstrated, for the first time, that Tbeta4 was down-regulated in ROS (zeige ROS1 ELISA Kits)-stimulated PDLCs as well as Tbeta4 activation exhibited anti-inflammatory effects and anti-osteoclastogenesis in vitro.
This article reviews the potential role of thymosin beta 4 in liver fibrosis by describing the effects of exogenous and endogenous thymosin beta 4 , and discusses the possible signaling pathway regulated by TB4X. [review]
The results of the present study suggested that Tb4 may promote HB metastasis via the induction of EMT (zeige ITK ELISA Kits), and that Tb4 may therefore be a target for the development of novel treatments for patients with Hepatoblastoma .
these results suggest that NO could regulate the expression of Tbeta4 by direct or indirect effect of HIF-1alpha (zeige HIF1A ELISA Kits) on Tbeta4 promoter.
the exchange of bound actin between Tbeta4 and profilin (zeige PFN1 ELISA Kits) involves both steric and allosteric components.
Our data suggest that Tbeta4 is required for setting correct sarcomere length and for appropriate splicing of titin (zeige TTN ELISA Kits), not only in the heart but also in skeletal muscle.
Increased Tb4 expression was observed in ethanol treated microglia. Knockdown of Tb4 enhanced ethanol-induced inflammatory mediators tumor necrosis factor-alpha and interleukin-1beta and nitric oxide in BV-2 cells was performed.
Results provide evidence that Tbeta4 regulate the levels of VEGF (zeige VEGFA ELISA Kits) and MMP-2 (zeige MMP2 ELISA Kits) via the Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits)/Lef-1 (zeige LEF1 ELISA Kits) signaling pathway to influence the growth of blood vessels around hair follicles and to activate cell migration.
Tbeta4 appears to regulate P38/ERK (zeige MAPK1 ELISA Kits)/AKT (zeige AKT1 ELISA Kits) signaling via its effect on VEGF (zeige VEGFA ELISA Kits) expression, with a resultant effect on the speed of hair growth, the pattern of HFs and the number of hair shafts
This study provides putative information concerning the signaling pathway through which Tb4 induces RUNX2 (zeige RUNX2 ELISA Kits) expression, which may help to understand the regulation of tooth development and tooth regeneration.
Tbeta4 substantially inhibited bleomycin-induced IL-17 (zeige IL17A ELISA Kits) over-expression in the lung tissue: a decreased amount of IL17 (zeige IL17A ELISA Kits)-producing cells and inhibited IL-17 (zeige IL17A ELISA Kits) expression in lung with Tbeta4 treatment correlate with its anti-inflammatory and anti-fibrotic effects.
These data suggest that Tbeta4 suppresses the TLR proinflammatory pathway by up-regulating miR (zeige MLXIP ELISA Kits)-146a.
Thymosin beta4 may play a role in aging-related hearing loss.
These data show that thymosinbeta4 and beta-actin are over-expressed during nurse cell formation upon T. spiralis infection and may be involved in nurse cell formation along with other actin-binding proteins.
TMSB4 and/or TMSB10 (zeige TMSB10 ELISA Kits) ovarian expression patterns suggest that these two thymosins may be involved in cumulus modifications during maturation.
This gene encodes an actin sequestering protein which plays a role in regulation of actin polymerization. The protein is also involved in cell proliferation, migration, and differentiation. This gene escapes X inactivation and has a homolog on chromosome Y.
, t beta-4
, thymosin beta-4
, thymosin, beta 4, X chromosome
, prothymosin beta 4
, t beta 4
, thymosin beta 4, X-linked
, thymosin beta 4Xen
, thymosin beta 4, X chromosome
, thymosin, beta 4, X-linked
, thymosin, beta 4
, thymosin beta 4 X-linked
, thymosin b4