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CD70 (zeige CD70 ELISA Kits) reverse signaling enhances NK cell function and immunosurveillance in CD27-expressing B-cell malignancies.
CD27-dependent CD8 (zeige CD8A ELISA Kits)(+) T cell priming and differentiation are shaped by the efficiency of NK responses to virus infection.
HSCs appear to maintain expression of CD27 after hematopoietic injury when Kit expression is downregulated.
Mature CD27low NK cells promote allograft survival under costimulatory blockade conditions by regulating alloreactive memory CD8+ T-cell responses in T-bet dependent manner.
identify an IL-17 (zeige IL17A ELISA Kits)-dependent lymphoid/myeloid cross-talk involving CD27(-) gammadelta T cells and small peritoneal macrophages that promotes ovarian tumor cell growth and thus counteracts cancer immunosurveillance
memory CD8 (zeige CD8A ELISA Kits) T cells, generated through priming with CD27 agonists, proliferated more extensively than did 4-1BB (zeige TNFRSF9 ELISA Kits)-generated memory cells, but these cells failed to persist.
the central role played by CD27/70 during secondary CD8 (zeige CD8A ELISA Kits)(+) T-cell activation to a peptide Ag, and identify sCD70 as an immunotherapeutic adjuvant for antitumor immunity.
under conditions of constitutive CD70 (zeige CD70 ELISA Kits) expression reflecting chronic immune activation, the CD27/CD70 (zeige CD70 ELISA Kits) system inhibits OC differentiation and favors DC differentiation
mTECs and DCs form dedicated niches in the thymic medulla, in which CD27-CD70 (zeige CD70 ELISA Kits) co-stimulation rescues developing Treg cells from apoptosis, subsequent to Foxp3 (zeige FOXP3 ELISA Kits) induction by TCR and CD28 (zeige CD28 ELISA Kits) signals.
The CD27 and CD70 (zeige CD70 ELISA Kits) costimulatory pathway inhibits effector function of T helper 17 cells and attenuates associated autoimmunity.
High CD27 expression is associated with B-cell Lymphoma.
activated CD70 (zeige CD70 ELISA Kits)-expressing lymphocytes may trigger CD27 on AML (zeige RUNX1 ELISA Kits) blasts or stem/progenitor cells
the CD70 (zeige CD70 ELISA Kits)-CD27 pathway appears to be a crucial component of EBV-specific T cell immunity and more generally for the immune surveillance of B cells and may be a target for immunotherapy of B cell malignancies.
This study demonstrates increased level of sCD27 in patients with active vitiligo (zeige MITF ELISA Kits).
This study demonstrated that Soluble CD27 in cerebrospinal fluid of patients with Clinically Isolated Syndrome was associated with multiple sclerosis diagnosis and a high relapse rate.
HCV infection increased the frequency of CD4 (zeige CD4 ELISA Kits)+CXCR5 (zeige CXCR5 ELISA Kits)+ T cells and decreased the frequency of CD27+IgG+ B cells. CD4 (zeige CD4 ELISA Kits)+CXCR5 (zeige CXCR5 ELISA Kits)+ T cells activated CD27+IgG+ B cells via the secretion of IL-21 (zeige IL17C ELISA Kits).
Novel mutations in TNFRSF7/CD27 cause EBV-associated lymphoproliferative disease/hemophagocytic lymphohistiocytosis, Hodgkin lymphoma, uveitis, and recurrent infections.
NFKB1 (zeige NFKB1 ELISA Kits), CD27, LAG3 (zeige LAG3 ELISA Kits) and IKZF3 (zeige IKZF3 ELISA Kits) are new susceptibility genes for psoriasis.
results provide the first evidence for differential expression of CD27 among CLL prognostic groups, suggest a role for ZAP-70 (zeige ZAP70 ELISA Kits) dependent signaling in CD27 induction and implicate CD27 in cell-cell interactions with the lymphoid tissue microenvironment
A lower primary CD24 (zeige CD24 ELISA Kits)(hi) CD27(+) CD19 (zeige CD19 ELISA Kits)(+) B cells may be an immunologic aspect of new-onset SLE that may be a useful tool to evaluate lupus activity and monitor the response to therapy.
The protein encoded by this gene is a member of the TNF-receptor superfamily. This receptor is required for generation and long-term maintenance of T cell immunity. It binds to ligand CD70, and plays a key role in regulating B-cell activation and immunoglobulin synthesis. This receptor transduces signals that lead to the activation of NF-kappaB and MAPK8/JNK. Adaptor proteins TRAF2 and TRAF5 have been shown to mediate the signaling process of this receptor. CD27-binding protein (SIVA), a proapoptotic protein, can bind to this receptor and is thought to play an important role in the apoptosis induced by this receptor.
tumor necrosis factor receptor superfamily, member 7
, CD27 antigen
, CD27L receptor
, CD antigen 27
, T-cell activation antigen CD27
, tumor necrosis factor receptor superfamily member 7
, T cell activation antigen S152