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anti-Human SLC39A3 Antikörper:
anti-Mouse (Murine) SLC39A3 Antikörper:
anti-Rat (Rattus) SLC39A3 Antikörper:
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Human Polyclonal SLC39A3 Primary Antibody für WB - ABIN949250
Kanninen, Grubman, Caragounis, Duncan, Parker, Lidgerwood, Volitakis, Ganio, Crouch, White: Altered biometal homeostasis is associated with CLN6 mRNA loss in mouse neuronal ceroid lipofuscinosis. in Biology open 2013
Silence of p62 promotes apoptosis induced by mitochondrial depolarization.
Rare variants in SQSTM1 (zeige SQSTM1 Antikörper) are risk for sporadic inclusion body myositis.
The WDR81 coordinates p62 and LC3C to facilitate autophagic removal of Ub proteins, and provide important insights into CAMRQ2 syndrome, a WDR81-related developmental disorder.
SQSTM1-Nup214, although mostly cytoplasmic, also forms nuclear bodies and inhibits nuclear protein but not poly(A)(+) RNA export.
autophagy upregulation by Sequestosome 1 (p62 (zeige SQSTM1 Antikörper)/SQSM1) knockdown ameliorated this cell injury and relieved insulin (zeige INS Antikörper) resistance
This work suggests that UVA-induced p62 (zeige GTF2H1 Antikörper) acts through COX-2 (zeige COX2 Antikörper) to promote skin tumor growth and progression.
we provide evidence that p62 (zeige GTF2H1 Antikörper) is implicated in the activation of NF-kappaB (zeige NFKB1 Antikörper) signaling that is partly dependent on RIP1 (zeige UQCRFS1 Antikörper)
In this study, we first observed that p62-positive intranuclear aggregates also appeared in controls and were not specific for the Neuronal Intranuclear Hyaline Inclusion Disease cases.
Data suggest that phosphorylation of SQSTM1 (zeige SQSTM1 Antikörper) by AMP-activated protein kinase (AMPK (zeige PRKAA2 Antikörper)) and translocation of SQSTM1 (zeige SQSTM1 Antikörper) to mitochondria drives autophagic cell death in adult hippocampal neural stem cells; activation of AMPK (zeige PRKAA1 Antikörper) promotes autophagic cell death; inhibition of AMPK (zeige PRKAA1 Antikörper) (by enzyme inhibitor or RNA interference) suppresses autophagic cell death. (SQSTM1 (zeige SQSTM1 Antikörper) = sequestosome 1 (zeige SQSTM1 Antikörper))
Authors found that p62/SQSTM1 (zeige SQSTM1 Antikörper), a protein upregulated in liver parenchymal cells but downregulated in HCC (zeige FAM126A Antikörper)-associated HSCs, negatively controls hepatic stellate cells activation.
ZnT3 (zeige Slc30a3 Antikörper) KO mice showed abnormality in trace fear conditioning is involved in associative fear memory and extinction, but not in innate fear.
Znt-3-deficient mice lacking synaptic Zn also show less hippocampal cell damage following kainic acid injection. Zip transporters may provide selective therapeutic targets to protect these neurons from early Zn-induced neurodegeneration following injury.
The expression of zinc transporter 3 (ZnT3 (zeige Slc30a3 Antikörper)) starts early in development, lasts throughout embryonic life, and is detected in proliferative and differentiating areas of the brain in distinct chelatable vesicular zinc pools.
ZnT3 (zeige Slc30a3 Antikörper)-immunoreactive ependymal cells possess secretory activity directed towards the central canal.
ZIP1 (zeige SLC39A1 Antikörper), ZIP2 (zeige Slc39a2 Antikörper) and ZIP3 may play cell-specific roles in zinc homeostasis rather than primary roles in the acquisition of dietary zinc
ZIP1 (zeige SLC39A1 Antikörper) and ZIP3 zinc uptake transporter activity is controlled by zinc-stimulated endocytosis
Reduced expression of Zip3 ultimately resulted in cell death, indicating that mammary epithelial cells have a unique requirement for Zip3-mediated Zn(2+) import.
Knockout mice lacking Zip3 were generated and characterized.
Mutations in the ZIP1 (zeige SLC39A1 Antikörper) and ZIP3 zinc transporter genes are silent when dietary intake of zinc is normal, but can dramatically compromise the success of pregnancy when dietary intake of zinc is limiting.
temporal and spatial patterns of expression of the mouse ZIP1 (zeige SLC39A1 Antikörper), 3, 4, and 5 genes in the developing intestine and the effects of maternal dietary zinc deficiency on these patterns of expression were examined
This gene encodes a multifunctional protein that binds ubiquitin and regulates activation of the nuclear factor kappa-B (NF-kB) signaling pathway. The protein functions as a scaffolding/adaptor protein in concert with TNF receptor-associated factor 6 to mediate activation of NF-kB in response to upstream signals. Alternatively spliced transcript variants encoding either the same or different isoforms have been identified for this gene. Mutations in this gene result in sporadic and familial Paget disease of bone.
EBI3-associated protein of 60 kDa
, EBI3-associated protein p60
, oxidative stress induced like
, phosphotyrosine independent ligand for the Lck SH2 domain p62
, phosphotyrosine-independent ligand for the Lck SH2 domain of 62 kDa
, ubiquitin-binding protein p62
, solute carrier family 39 member 3
, zinc transporter ZIP3
, zrt- and Irt-like protein 3