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Human Hepcidin ELISA Kit für Sandwich ELISA - ABIN415120
Cheng, Sun, Jiang, Tang, Jiao: Hepcidin expression in patients with acute leukaemia. in European journal of clinical investigation 2012
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Human Hepcidin ELISA Kit für Competition ELISA - ABIN2761946
Sany, Elsawy, Elshahawy: Hepcidin and regulation of iron homeostasis in maintenance hemodialysis patients. in Saudi journal of kidney diseases and transplantation : an official publication of the Saudi Center for Organ Transplantation, Saudi Arabia 2014
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Human Hepcidin ELISA Kit für Sandwich ELISA - ABIN457010
Jiang, Sun, Tang, Xu, Jiao: Hepcidin expression and iron parameters change in Type 2 diabetic patients. in Diabetes research and clinical practice 2011
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Mouse (Murine) Hepcidin ELISA Kit für Sandwich ELISA - ABIN415686
Shin, Chung, Joe, Pae, Chang, Cho, Ryter, Chung: Pretreatment with CO-releasing molecules suppresses hepcidin expression during inflammation and endoplasmic reticulum stress through inhibition of the STAT3 and CREBH pathways. in Blood 2012
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Rat (Rattus) Hepcidin ELISA Kit für Sandwich ELISA - ABIN416434
Huang, Chuang, Yang, Huang, Wu, Chen: Cholestasis downregulate hepcidin expression through inhibiting IL-6-induced phosphorylation of signal transducer and activator of transcription 3 signaling. in Laboratory investigation; a journal of technical methods and pathology 2009
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Pig (Porcine) Hepcidin ELISA Kit für Competition ELISA - ABIN2761945
Pu, Guo, Liu, Xiong, Wang, Du: Iron Supplementation Attenuates the Inflammatory Status of Anemic Piglets by Regulating Hepcidin. in Biological trace element research 2015
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Mouse (Murine) Hepcidin ELISA Kit für Competition ELISA - ABIN2761947
Vecchi, Montosi, Garuti, Corradini, Sabelli, Canali, Pietrangelo: Gluconeogenic signals regulate iron homeostasis via hepcidin in mice. in Gastroenterology 2014
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Rat (Rattus) Hepcidin ELISA Kit für Sandwich ELISA - ABIN578992
Wang, Du, Duan, Guo, Sun, Liu: Inhibiting heme oxygenase-1 attenuates rat liver fibrosis by removing iron accumulation. in World journal of gastroenterology : WJG 2013
This study shows that hepcidin knockdown in zebrafish using morpholinos leads to iron overload.
The data also show that the antibacterial activity of hepcidin-2 depends upon the disulfide bridges.
data support an alternative mechanism for hepcidin regulation during zebrafish embryonic development, which is independent of hemojuvelin (zeige HFE2 ELISA Kits).
Hepcidin expression is regulated by a transferrin-a (zeige Tf ELISA Kits)-dependent pathway in the zebrafish embryo.
Data (including data from studies using knockout mice) suggest that MT2 (zeige MT2 ELISA Kits)/TMPRSS6 (zeige TMPRSS6 ELISA Kits) suppresses hepcidin expression in hepatocytes independently of HJV (zeige HFE2 ELISA Kits); MT2 (zeige MT2 ELISA Kits)/TMPRSS6 (zeige TMPRSS6 ELISA Kits) cleaves ALK2 (zeige ACRV1 ELISA Kits), ALK3 (zeige BMPR1A ELISA Kits), ACTRIIA (zeige ACVR2A ELISA Kits), BMPR2 (zeige BMPR2 ELISA Kits), HFE (zeige HFE ELISA Kits), and, to a lesser extent, HJV (zeige HFE2 ELISA Kits) and TFR2 (zeige TFR2 ELISA Kits); thus, MT2 (zeige MT2 ELISA Kits)/TMPRSS6 (zeige TMPRSS6 ELISA Kits) suppresses hepcidin expression by cleaving multiple components of the hepcidin induction pathway. (MT2 (zeige MT2 ELISA Kits)/TMPRSS6 (zeige TMPRSS6 ELISA Kits) = matriptase-2 (zeige TMPRSS6 ELISA Kits); HJV (zeige HFE2 ELISA Kits) = hemojuvelin (zeige HFE2 ELISA Kits))
two unrelated hepatocellular carcinoma patients bore the HAMP:c.-153C>T mutation at the heterozygous state, which is associated with increased risk of iron overload and severe hemochromatosis (zeige HFE ELISA Kits)
Hepcidin-25 released from plaque macrophages and other cell surfaces contributed to the plaque instability by inducing endothelial cell death.
Hepcidin plasma levels were increased in patients with early rheumatoid arthritis compared with healthy volunteers.
study followed the dynamics of hepcidin-mediated ferroportin (zeige SLC40A1 ELISA Kits) internalization; also showed that the novel p.D84E mutation, associated with the classical form of ferroportin (zeige SLC40A1 ELISA Kits) disease, is both iron transport defective and hepcidin insensitive
During regulation of hepcidin synthesis, multiple promoter elements in the HAMP gene respond to variable signaling pathways corresponding to different extracellular situations.
Our findings indicate that the HAMP-P -582A>G polymorphism (rs10421768) is associated with susceptibility to extrapulmonary TB, but not pulmonary TB. CD14 (zeige NDUFA2 ELISA Kits)+ monocytes from individuals with the rs10421768 GG genotype secreted significantly less hepcidin in response to M. tuberculosis lipoarabinomannan compared with cells from individuals with either the AA or AG genotypes.
Expression of Hepcidin and Ferroportin (zeige SLC40A1 ELISA Kits) in the Placenta, and Ferritin (zeige FTL ELISA Kits) and Transferrin Receptor 1 (zeige TFR ELISA Kits) Levels in Maternal and Umbilical Cord Blood in Pregnant Women with and without Gestational Diabetes
this study shows that hepcidin is involved in the pathogenesis of sepsis-induced acute kidney injury
The hepcidin plays a role in the pathogenesis and progression of kidney injury via modulation of iron-mediated oxidant injury.
The authors generated mice with cardiomyocyte-specific deletion of hepcidin, or knock-in of hepcidin-resistant ferroportin (zeige SLC40A1 ELISA Kits). They find that while both models maintain normal systemic iron homeostasis, the mice nonetheless develop fatal contractile and metabolic dysfunction as a consequence of cardiomyocyte iron deficiency.
Erythroferrone and matriptase-2 (zeige TMPRSS6 ELISA Kits) independently regulate hepcidin expression.
Acute tacrolimus treatment transiently increases hepcidin in wild-type mice. FKBP12 (zeige FKBP1A ELISA Kits) preferentially targets the BMP receptor (zeige BMPR1A ELISA Kits) ALK2 (zeige ACRV1 ELISA Kits). ALK2 (zeige ACRV1 ELISA Kits) mutants defective in binding FKBP12 (zeige FKBP1A ELISA Kits) increase hepcidin expression in a ligand-independent manner, through BMP-SMAD (zeige SMAD1 ELISA Kits) signaling.
Hamp1 mRNA and plasma hepcidin levels are not good predictors of tissue iron levels, at least in males
The lack of effect of erythropoietin (zeige EPO ELISA Kits) on hepcidin expression in mask mice can not be explained by changes in erythroferrone synthesis, as splenic erythroferrone content increased after erythropoietin (zeige EPO ELISA Kits) administration in both C57BL/6 and mask mice.
these results characterise a new model of rapidly inducible hepcidin disruption, and demonstrate the critical contribution of hepcidin to the hypoferraemia of inflammation
Hepatic gene expression of hepcidin is regulated in beta-thalassemia by ATOH8.
Endogenous hepcidin and its agonist mediate resistance to selected infections by clearing non-transferrin (zeige Tf ELISA Kits)-bound iron.
The data demonstrate that endothelial cells are the predominant source of BMP6 (zeige BMP6 ELISA Kits) in the liver and support a model in which endothelial cells BMP6 (zeige BMP6 ELISA Kits) has paracrine actions on hepatocyte hemojuvelin (zeige HFE2 ELISA Kits) to regulate hepcidin transcription and maintain systemic iron homeostasis.
Smad1 (zeige SMAD1 ELISA Kits) and Smad5 (zeige SMAD5 ELISA Kits) have overlapping functions to govern hepcidin transcription. Moreover, erythropoietin (zeige EPO ELISA Kits) and erythroferrone target Smad1 (zeige SMAD1 ELISA Kits)/5 signaling and require Smad1 (zeige SMAD1 ELISA Kits)/5 to suppress hepcidin expression.
this study demonstrates that urine hepcidin-25 concentrations strongly correlate with hepatic hepcidin mRNA abundance, plasma hepcidin-25 levels, iron transferrin (zeige Tf ELISA Kits) saturation and non-heme liver iron levels.
Data suugest that hepcidin might had antiinflammatory function and is a candidate regulator of the cross-talk between iron regulation and inflammation.
report the full-length cDNA sequences of porcine hepcidin and liver-expressed antimicrobial peptide-2 (LEAP-2 (zeige LEAP2 ELISA Kits))
Hepcidin peptide is up-regulated by iron and bacterial components in the trout liver.
The product encoded by this gene is involved in the maintenance of iron homeostasis, and it is necessary for the regulation of iron storage in macrophages, and for intestinal iron absorption. The preproprotein is post-translationally cleaved into mature peptides of 20, 22 and 25 amino acids, and these active peptides are rich in cysteines, which form intramolecular bonds that stabilize their beta-sheet structures. These peptides exhibit antimicrobial activity. Mutations in this gene cause hemochromatosis type 2B, also known as juvenile hemochromatosis, a disease caused by severe iron overload that results in cardiomyopathy, cirrhosis, and endocrine failure.
hepcidin antimicrobial peptide
, antimicrobial peptide
, iron regulatory peptide
, preprohepcidin 1
, liver-expressed antimicrobial peptide 1
, putative liver tumor regressor
, hepcidin antimicrobial peptide 1
, antimicrobial peptide hepcidin
, putative hepcidin antibacterial peptide