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Human Polyclonal CLDN4 Primary Antibody für EIA, WB - ABIN951550
Papageorgis, Lambert, Ozturk, Gao, Pan, Manne, Alekseyev, Thiagalingam, Abdolmaleky, Lenburg, Thiagalingam: Smad signaling is required to maintain epigenetic silencing during breast cancer progression. in Cancer research 2010
Zeige alle 4 Referenzen für ABIN951550
Human Polyclonal CLDN4 Primary Antibody für EIA, IF - ABIN1449984
Mitic, Unger, Anderson: Expression, solubilization, and biochemical characterization of the tight junction transmembrane protein claudin-4. in Protein science : a publication of the Protein Society 2003
Zeige alle 3 Referenzen für ABIN1449984
Human Polyclonal CLDN4 Primary Antibody für ICC, IF - ABIN407633
Rho, Lampe: High-throughput screening for native autoantigen-autoantibody complexes using antibody microarrays. in Journal of proteome research 2013
Human Polyclonal CLDN4 Primary Antibody für IF (p), IHC (p) - ABIN680743
Wang, Chen, Liang, Yan, Lin, Liu, Luo, Huang, Li, Liu, Tang: Notch inhibition promotes fetal liver stem/progenitor cells differentiation into hepatocytes via the inhibition of HNF-1?. in Cell and tissue research 2014
claudin-4 may represent different mechanisms of lymphatic vessel invasion with both biomarkers is related to poor prognosis
These results suggested that increase of Cldn4-expression may be involved in early molecular events during carcinogenesis of adenocarcinoma, whereas increase of Cldn7 (zeige CLDN7 Antikörper)-expression may be associated with tumor invasion or progression.
that claudin-4 can be helpful at least in making a reliable differential diagnosis of spiradenoma when overlapping morphologic features do not allow to further subclassification in the overwhelming variety of the adnexal tumors.
Results showed overexpression of CLDN4 in endometrial cancer cells. Its intracellular presence coupled with the biphasic effects of E2 on its expression in the cytoskeleton suggest that it may be involved in cell signaling to and from tight junctions.
claudin 1 (zeige CLDN7 Antikörper) and claudin 4 are differentially involved in atopic dermatitis pathogenesis
Results suggested that claudin-4, which was regulated by methylation status, plays an important role in breast cancer growth and malignancy via the control of cell proliferation, migration and apoptosis.
Overexpression of claudin-4 induced formation of vascular channels in breast cancer.
This meta-analysis shows that over-expression of claudin-4 is associated with progress of gastric cancer and poor prognosis of gastric cancer patients.
Data suggest that changes in DNA methylation (zeige HELLS Antikörper) in trophoblasts regulate (1) cell mobility/placentation, (2) expression of claudin-4 (CLDN4) and 4-fucosyltransferase (FUT4 (zeige FUT4 Antikörper)), and (3) matrix metalloproteinase (MMP2 (zeige MMP2 Antikörper) and MMP9 (zeige MMP9 Antikörper)) activity.
Data show that claudin-4 and claudin-7 (zeige CLDN7 Antikörper) were observed in hepatocytes of severely damaged mouse and human livers.
a Grhl2 (zeige GRHL2 Antikörper)/Ovol2 (zeige OVOL2 Antikörper) network controls Cldn4 and Rab25 (zeige RAB25 Antikörper) expression that facilitates lumen expansion and barrier formation in subtypes of renal epithelia
The claudin-4-mediated chloride conductance can be regulated endogenously by a protease-channel-activating protease 1 (cap1 (zeige PRSS8 Antikörper)).
Claudin 4 has little effect on normal lung physiology but may function to protect against acute lung injury in mice.
The results suggest that progressive hydronephrosis in Cldn4(-/-) mice arises from urinary tract obstruction due to urothelial hyperplasia, and that Cld4 plays an important role in maintaining the homeostatic integrity of normal urothelium.
the results indicate that Grhl2 (zeige GRHL2 Antikörper) regulates epithelial morphogenesis through transcriptional up-regulation of Cldn3 (zeige CLDN3 Antikörper) and Cldn4, as well as of Rab25 (zeige RAB25 Antikörper), which increases the Cldn4 protein and its localization at TJs
Mechanism of Clostridium perfringens enterotoxin interaction with claudin-3 (zeige CLDN3 Antikörper)/-4 protein suggests structural modifications of the toxin to target specific claudins
Claudin-3 (zeige CLDN3 Antikörper) in the apical-most regions maintains the impermeable tight junctions during lactation, and claudin-4 contributes
Claudin-4 overexpression is associated with epigenetic derepression in gastric carcinoma.
results suggest that Cld4 is induced by E-protein activity in the later stages of DP cells to increase the efficiency of positive selection, uncovering a hitherto unrecognized function of a Cld (zeige LMF1 Antikörper) family protein
Claudin-4 expression shows age-dependent change in cellular localization on pig jejunal villous epithelial cells
These findings indicate that claudin-4 and -7 may play a role in the gingiva junctional epithelium even in the absence of tight junctions.
This gene encodes an integral membrane protein, which belongs to the claudin family. The protein is a component of tight junction strands and may play a role in internal organ development and function during pre- and postnatal life. This gene is deleted in Williams-Beuren syndrome, a neurodevelopmental disorder affecting multiple systems.
, claudin 4
, Clostridium perfringens enterotoxin receptor 1
, Williams-Beuren syndrome chromosomal region 8 protein
, clostridium perfringens enterotoxin receptor
, tight junction-associated protein
, uncharacterized protein LOC73934