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anti-Human SOX9 Antikörper:
anti-Rat (Rattus) SOX9 Antikörper:
anti-Mouse (Murine) SOX9 Antikörper:
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Human Polyclonal SOX9 Primary Antibody für ICC, IF - ABIN4355424
Vestentoft, Jelnes, Hopkinson, Vainer, Møllgård, Quistorff, Bisgaard: Three-dimensional reconstructions of intrahepatic bile duct tubulogenesis in human liver. in BMC developmental biology 2011
Show all 10 Pubmed References
Human Polyclonal SOX9 Primary Antibody für IF (p), IHC (p) - ABIN754963
Papaioannou, Mirzamohammadi, Lisse, Nishimori, Wein, Kobayashi: MicroRNA-140 provides robustness to the regulation of hypertrophic chondrocyte differentiation by the PTHrP-HDAC4 pathway. in Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 2014
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Human Monoclonal SOX9 Primary Antibody für IF, IHC (p) - ABIN520382
Hodgin, Borczuk, Nasr, Markowitz, Nair, Martini, Eichinger, Vining, Berthier, Kretzler, DAgati: A molecular profile of focal segmental glomerulosclerosis from formalin-fixed, paraffin-embedded tissue. in The American journal of pathology 2010
Show all 4 Pubmed References
Human Monoclonal SOX9 Primary Antibody für ICC, ELISA - ABIN969568
Dupasquier, Abdel-Samad, Glazer, Bastide, Jay, Joubert, Cavaillès, Blache, Quittau-Prévostel: A new mechanism of SOX9 action to regulate PKCalpha expression in the intestine epithelium. in Journal of cell science 2009
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Zebrafish Polyclonal SOX9 Primary Antibody für ELISA - ABIN408740
Yan, Willoughby, Liu, Crump, Wilson, Miller, Singer, Kimmel, Westerfield, Postlethwait: A pair of Sox: distinct and overlapping functions of zebrafish sox9 co-orthologs in craniofacial and pectoral fin development. in Development (Cambridge, England) 2005
Human Polyclonal SOX9 Primary Antibody für IF, IHC (p) - ABIN272082
Zhang, Ding, Nie, Li-Ling, Zhang, Zhang, Chen, Li, Ding: Association of hsa-miR‑145 overexpression in human testicular cells with male infertility. in Molecular medicine reports 2015
Cow (Bovine) Polyclonal SOX9 Primary Antibody für FACS, WB - ABIN2780338
Vidal, Ortonne, Schedl: SOX9 expression is a general marker of basal cell carcinoma and adnexal-related neoplasms. in Journal of cutaneous pathology 2008
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Human Monoclonal SOX9 Primary Antibody für ELISA, WB - ABIN562960
Wainwright, Svingen, Ng, Wicking, Koopman: Primary cilia function regulates the length of the embryonic trunk axis and urogenital field in mice. in Developmental biology 2014
Amphibian Polyclonal SOX9 Primary Antibody für DB, IHC - ABIN4355422
Sharma, Knowell, Chinaranagari, Komaragiri, Nagappan, Patel, Havrda, Chaudhary: Id4 deficiency attenuates prostate development and promotes PIN-like lesions by regulating androgen receptor activity and expression of NKX3.1 and PTEN. in Molecular cancer 2014
Cat (Feline) Polyclonal SOX9 Primary Antibody für IHC (p), SimWes - ABIN4355427
Antunes, Tsaryk, Gonçalves, Pereira, Landes, Brochhausen, Ghanaati, Barbosa, Kirkpatrick: Poly(γ-Glutamic Acid) as an Exogenous Promoter of Chondrogenic Differentiation of Human Mesenchymal Stem/Stromal Cells. in Tissue engineering. Part A 2015
Data show that the gene encoding the transcription factor SOX9 was identified by a global transcriptomic approach as an HDAC9 (zeige HDAC9 Antikörper) target gene.
SOX9 is a proliferation and stem cell factor (zeige KITLG Antikörper) in hepatocellular carcinoma and possess widespread prognostic significance in different cancer types
Sox9 and Ngn3 (zeige NEUROG3 Antikörper), key transcription factors associated with pancreatic development.
Expression of bone morphogenetic protein (BMP) 4, an upstream stimulator of SOX9, was upregulated by CG.
Xenogeneic implantation of Sox9-overexpressing hUCMSCs embedded in the BMG/fibrin scaffolds promotes the formation of cartilage-like tissue without inducing evident host immune response. Therefore, Sox9-overexpressing hUCMSCs represent a promising cell candidate for cartilage tissue engineering.
KLF15 activates SOX9 expression directly. SOX9 is involved in KLF15 function during chondrogenic differentiation.
Tomo-Seq Identifies SOX9 as a Key Regulator of Cardiac Fibrosis During Ischemic Injury
High SOX9 expression is associated with glioblastoma.
These findings suggest that SOX9 may play an important role in tumor progression of Renal Cell Carcinoma (zeige MOK Antikörper) and Bladder Cancer and it may be used as a biomarker of this malignancy.
Loss of DDRGK1 decreases SOX9 expression and causes a human skeletal dysplasia.
data presented here demonstrate that the 18 bp indel in the porcine SOX9 5'-UTR (zeige UTS2R Antikörper) is of functional importance and may therefore indeed be a causative variation in SOX9 associated traits
HIF-1alpha (zeige HIF1A Antikörper) activates Sox9 expression and enhances Sox9-mediated transcriptional activity.
Hypomethylation in the Sox9 promoter is correlated to increased Sox9 expression in db/db (zeige LEPR Antikörper) IESCs. Although there is increased expression of Sox9 in db/db (zeige LEPR Antikörper) IESCs, the loss of Sox9 transcriptional activation in specific repressors of the Wnt (zeige WNT2 Antikörper) signaling pathway might result in abnormalities in this pathway.
Concluding, this study shows that, in addition to its crucial role in testis development, Sox9, together with Sox8 (zeige SOX8 Antikörper) and coordinately with Dmrt1 (zeige DMRT1 Antikörper), also controls adult testis maintenance.
SOX9 regulation of ETV5 (zeige ETV5 Antikörper) contributes to the control of male fertility
Nuclear factors that bind to genomic regions with "Sertoli Cell Signature" could functionally interact with SOX9; TRIM28 (zeige TRIM28 Antikörper) is a new SOX9 partner in fetal testes.
Down-regulated expression of Sox9 and Dazl (zeige DAZL Antikörper) may play important roles in MBP (zeige MBP Antikörper)-induced testis injury.
Sox9 is positively regulated by mesenchymal Fgf10 (zeige FGF10 Antikörper), a process that requires active Erk (zeige EPHB2 Antikörper) signaling during salivary gland development
TSC1 (zeige TSC1 Antikörper)/TSC2 (zeige TSC2 Antikörper) complex upregulation of OPN (zeige SPP1 Antikörper) expression is mediated by transcription factor SOX9 in an mTOR (zeige FRAP1 Antikörper)-independent manner. Moreover, ablation of OPN (zeige SPP1 Antikörper) by deficient TSC1 (zeige TSC1 Antikörper)/TSC2 (zeige TSC2 Antikörper) complex contributed to inactivation of AKT (zeige AKT1 Antikörper) in TSC (zeige SLC12A3 Antikörper) cells
SOX9 is almost exclusively expressed by astrocytes in the adult brain except for ependymal cells and in the neurogenic regions, where SOX9 is also expressed by neural progenitor cells
hese findings indicate that chronic overexpression of Sox9 in the uterine epithelium can induce the development of endometrial hyperplastic lesions. Thus, SOX9 expression may be a factor in the formation of endometrial cancer.
The conditional ablation of Sox9 in the genital tubercle using Shh (zeige SHH Antikörper)-Cre/+;Sox9(flox/flox) mice revealed no genital tubercle abnormalities, possibly due to compensation by similar Sox (zeige QSOX1 Antikörper) factors.
The protein encoded by this gene recognizes the sequence CCTTGAG along with other members of the HMG-box class DNA-binding proteins. It acts during chondrocyte differentiation and, with steroidogenic factor 1, regulates transcription of the anti-Muellerian hormone (AMH) gene. Deficiencies lead to the skeletal malformation syndrome campomelic dysplasia, frequently with sex reversal.
SRY (sex determining region Y)-box 9
, SRY (sex determining region Y)-box 9 (campomelic dysplasia, autosomal sex-reversal)
, transcription factor SOX-10
, SRY (sex-determining region Y)-box 9 protein
, SRY-related HMG-box, gene 9
, transcription factor SOX-9
, SRY-box containing protein 9
, SRY-box containing gene 9
, transcription factor SOX9