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anti-Human SMARCE1 Antikörper:
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Human Polyclonal SMARCE1 Primary Antibody für IHC, IHC (p) - ABIN251031
Link, Balasubramaniam, Sharma, Comstock, Godoy-Tundidor, Powers, Cao, Haelens, Claessens, Revelo, Knudsen: Targeting the BAF57 SWI/SNF subunit in prostate cancer: a novel platform to control androgen receptor activity. in Cancer research 2008
Show all 4 Pubmed References
Human Polyclonal SMARCE1 Primary Antibody für ICC, IF - ABIN4282875
Stadler, Hjelmare, Neumann, Jonasson, Pepperkok, Uhlén, Lundberg: Systematic validation of antibody binding and protein subcellular localization using siRNA and confocal microscopy. in Journal of proteomics 2012
Show all 2 Pubmed References
Human Polyclonal SMARCE1 Primary Antibody für IHC (p), WB - ABIN658277
Watanabe, Ui, Kanno, Ogiwara, Nagase, Kohno, Yasui: SWI/SNF factors required for cellular resistance to DNA damage include ARID1A and ARID1B and show interdependent protein stability. in Cancer research 2014
Human Polyclonal SMARCE1 Primary Antibody für IHC (p), IHC - ABIN249995
Wang, Chi, Xue, Zhou, Kuo, Crabtree: Architectural DNA binding by a high-mobility-group/kinesin-like subunit in mammalian SWI/SNF-related complexes. in Proceedings of the National Academy of Sciences of the United States of America 1998
BAF57, BAF60a (zeige SMARCD1 Antikörper) and SNF5 (zeige SMARCB1 Antikörper) might act as novel pro-senescence factors in both normal and tumor human skin cells
We report here three additional individuals with clinical features consistent with CSS (zeige CMAS Antikörper) and alterations in SMARCE1, one of which is novel. The probands all exhibited dysmorphic facial features, moderate developmental and cognitive delay, poor growth, and hypoplastic digital nails/phalanges, including digits not typically affected in the other genes associated with CSS (zeige CMAS Antikörper).
SMARCE1 mutational hits, including novel SMARCE1 mutations, were found in six of eight tested patients with clear cell meningioma
SMARCE1 plays an essential role in breast cancer metastasis by protecting cells against anoikis through the HIF1A (zeige HIF1A Antikörper)/PTK2 (zeige PTK2 Antikörper) pathway.
An exhaustive analysis of the BAF57 molecular and biochemical properties, cellular functions, loss-of-function phenotypes in living organisms and pathological manifestations in cases of human mutations. [review]
a family with a pediatric CCM patient and an adult CCM patient and several asymptomatic relatives carrying a germline SMARCE1 mutation.
Addition of the EGFR (zeige EGFR Antikörper) inhibitor gefitinib restores the sensitivity of SMARCE1-knockdown cells to MET and ALK (zeige ALK Antikörper) inhibitors in NSCLCs. Our findings link SMARCE1 to EGFR (zeige EGFR Antikörper) oncogenic signaling and suggest targeted treatment options for SMARCE1-deficient tumors.
The results suggested that BAF57 is involved in ovarian cancer cell growth and sensitivity to anticancer agents, and that BAF57 may be a target for ovarian cancer therapy.
BAF (zeige BANF1 Antikörper) complex gene SMARCE1 is mutated in Coffin-Siris syndrome patients.
Genotype-phenotype correlation of Coffin-Siris syndrome caused by mutations in SmarCE1 gene.
The N terminus of Brdt (zeige BRDT Antikörper) is involved in the recognition of Smarce1 as well as in the reorganization of hyperacetylated round spermatid chromatin.
suggest a novel mechanism for transcriptional repression by TSHZ3 (zeige ZNF537 Antikörper) in which TSHZ3 (zeige ZNF537 Antikörper) and BAF57 cooperate to modulate MyoD (zeige MYOD1 Antikörper) activity on the Myog (zeige MYOG Antikörper) promoter to regulate skeletal muscle differentiation.
Data show that neuregulin-1 (zeige NRG1 Antikörper) (NRG) inhibition is followed by a decrease in NSC proliferation and of neuronal or oligodendroglial differentiation, and that Nrg-1 (zeige NRG1 Antikörper) physically interacts with BRM (zeige SMARCA2 Antikörper) and BAF57.
Reciprocal regulation of CD4 (zeige CD4 Antikörper)/CD8 (zeige CD8A Antikörper) expression by SWI (zeige SMARCA1 Antikörper)/SNF (zeige SNRPA Antikörper)-like BAF (zeige BANF1 Antikörper) complexes.
The ER interacts with BAF57, which is stimulated by estrogen and requires both a functional hormone-binding domain & the DNA-binding region of the ER. The p160 (zeige MSH6 Antikörper) family of coactivators interacts with BAF57. Their potentiation of transcription depends on it.
Smarce1 (Baf27)is a critical modulator of the androgen neceptor (AR) that is capable of altering AR activity, coactivator function, and AR-dependent proliferation.
BAF (zeige BANF1 Antikörper) complex uses BAF57 to remodel the endogenous, H1-containing chromatin at the CD4 (zeige CD4 Antikörper) silencer, which facilitates the binding of Runx1 (zeige RUNX1 Antikörper), a key repressor of CD4 (zeige CD4 Antikörper) transcription.
The protein encoded by this gene is part of the large ATP-dependent chromatin remodeling complex SWI/SNF, which is required for transcriptional activation of genes normally repressed by chromatin. The encoded protein, either alone or when in the SWI/SNF complex, can bind to 4-way junction DNA, which is thought to mimic the topology of DNA as it enters or exits the nucleosome. The protein contains a DNA-binding HMG domain, but disruption of this domain does not abolish the DNA-binding or nucleosome-displacement activities of the SWI/SNF complex. Unlike most of the SWI/SNF complex proteins, this protein has no yeast counterpart.
SWI/SNF related, matrix associated, actin dependent regulator of chromatin, subfamily e, member 1
, SWI/SNF-related matrix-associated actin-dependent regulator of chromatin subfamily E member 1
, BRG1-associated factor 57
, SWI/SNF-related matrix-associated actin-dependent regulator of chromatin e1
, chromatin remodeling complex BRG1-associated factor 57
, SWI/SNF-related matrix-associated actin-dependent regulator chromatin subfamily E member 1