Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
Alle Spezies anzeigen
Weitere Synonyme anzeigen
Wählen Sie die Spezies und Applikation aus
anti-Mouse (Murine) Antikörper:
anti-Rat (Rattus) Antikörper:
Sie gelangen zu unserer vorgefilterten Suche.
Mouse (Murine) Monoclonal IL17F Primary Antibody für ICS, Neut - ABIN2689743
Chang, Dong: IL-17F: regulation, signaling and function in inflammation. in Cytokine 2009
Show all 9 Pubmed References
Human Polyclonal IL17F Primary Antibody für IHC (p), WB - ABIN530198
Oda, Shimazu, Naoi, Morimoto, Shimomura, Shimoda, Kagara, Maruyama, Kim, Noguchi: Intratumoral regulatory T cells as an independent predictive factor for pathological complete response to neoadjuvant paclitaxel followed by 5-FU/epirubicin/cyclophosphamide in breast cancer patients. in Breast cancer research and treatment 2012
Show all 2 Pubmed References
Mouse (Murine) Monoclonal IL17F Primary Antibody für CyTOF, FACS - ABIN4900112
Yamaguchi, Fujio, Shoda, Okamoto, Tsuno, Takahashi, Yamamoto: IL-17B and IL-17C are associated with TNF-alpha production and contribute to the exacerbation of inflammatory arthritis. in Journal of immunology (Baltimore, Md. : 1950) 2007
Show all 2 Pubmed References
Human Monoclonal IL17F Primary Antibody für CyTOF, FACS - ABIN4900181
Zrioual, Ecochard, Tournadre, Lenief, Cazalis, Miossec: Genome-wide comparison between IL-17A- and IL-17F-induced effects in human rheumatoid arthritis synoviocytes. in Journal of immunology (Baltimore, Md. : 1950) 2009
Show all 2 Pubmed References
Human Monoclonal IL17F Primary Antibody für ELISA, IHC - ABIN4324721
Liu, Zhao, Sun, Li, Wang, Jiang, Han, Shen, Corrigan, Sun: Expression of IL-17A, E, and F and their receptors in human prostatic cancer: Comparison with benign prostatic hyperplasia. in The Prostate 2015
Human Monoclonal IL17F Primary Antibody für WB - ABIN1043785
Conti, Shen, Nayyar, Stocum, Sun, Lindemann, Ho, Hai, Yu, Jung, Filler, Masso-Welch, Edgerton, Gaffen: Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis. in The Journal of experimental medicine 2009
our data suggest that IL-20 (zeige IL20 Antikörper) subfamily cytokines, particularly IL-20 (zeige IL20 Antikörper), IL-22 (zeige IL22 Antikörper), and IL-24 (zeige IL24 Antikörper), might provide therapeutic benefit for patients with Diabetic foot ulcers (DFU) .
this study shows that IL-24 (zeige IL24 Antikörper) promotes Pseudomonas aeruginosa keratitis through the suppression of early protective mucosal immunity
Loss of IL-24 (zeige IL24 Antikörper) expression is associated with breast cancer.
our results indicate that Th17 cells contribute to the airway vascular remodeling in asthma by mediating EPC (zeige TCF21 Antikörper) chemotaxis, as well as PMVEC tube formation, via IL-17A (zeige IL17A Antikörper) rather than IL-17F.
Data indicate that IL-17A (zeige IL17A Antikörper) repressed IL-17F secretion in vitro and in vivo.
A psoriasis-like skin inflammation in mice with epidermis-specific inhibition of NF-kappaB (zeige NFKB1 Antikörper) was triggered by TNFR1 (zeige TNFRSF1A Antikörper)-dependent upregulation of interleukin-24 (zeige IL24 Antikörper) & activation of STAT3 (zeige STAT3 Antikörper) signaling in keratinocytes.
The concentrations in plasma of IL-17A (zeige IL17A Antikörper), IL-17F, and the IL-17AF heterodimer all were increased greatly in mice after endotoxemia.
IL-24 (zeige IL24 Antikörper) suppresses the growth of normal vascular smooth muscle cells by inhibiting hydrogen peroxide-induced reactive oxygen species (ROS (zeige ROS1 Antikörper)) production through the regulation of mitochondrial ROS (zeige ROS1 Antikörper) production and expression of antioxidant enzymes
IL-17A (zeige IL17A Antikörper) and IL-17F secreted by CD4 (zeige CD4 Antikörper)+Th17 cells specific to lung self-antigens are critical mediators of autoimmunity leading to the pathogenesis of obliterative airway disease.
our study provides the evidence that functional IL-23R (zeige IL23R Antikörper) rs1884444 G/T and IL-17F rs763780 A/G polymorphisms may be a new genetic susceptibility factor to SLE, especially in the Polish population.
IL23R (zeige IL23R Antikörper) rs10889677 and IL17A (zeige IL17A Antikörper) rs2275913 were not associated with the susceptibility to Necrotizing enterocolitis (NEC). In conclusion, data suggest that a variant of IL17F (rs763780) may contribute to the development of NEC.
Our results confirmed IL17A (zeige IL17A Antikörper) and IL17F as potential candidate genes involved in RA. They play pivoting roles in the susceptibility and in clinical features of RA disease. Responses to RA treatments are differently conditioned by polymorphisms in IL17A (zeige IL17A Antikörper) and IL17F genes.
IL17A (zeige IL17A Antikörper) and IL17F gene polymorphism are not the important factors associated with susceptibility and some clinical parameters of rheumatoid arthritis in a Polish population.
we aimed to investigate the associations between the 7383A/G and 7488A/G polymorphisms of the interleukin (IL)-17F gene with disease activity and clinical outcomes in Turkish patients with ankylosing spondylitis
Using an in-vitro migration assay, B cells were shown to migrate towards both IL-17A (zeige IL17A Antikörper) and IL-17F. These observations indicate a direct chemotactic effect of IL-17 (zeige IL17A Antikörper) cytokines on primary peripheral blood B cells with higher effect being on asthmatic B cells.
IL-17 (zeige IL17A Antikörper)-related cytokines expression was amplified in bronchial/nasal mucosa of neutrophilic asthma prone to exacerbation, suggesting a pathogenic role of IL-17F in frequent exacerbators.
IL-17A (-197G/A) and IL-17F (7488T/C) SNPs were not associated with susceptibility to rheumatoid arthritis or secondary Sjogrens syndrome (sSS, p > 0.05 for both SNPs). In addition, they did not influence RA activity or clinical markers of SS.
It can be stated that the IL17A (zeige IL17A Antikörper) and IL17F polymorphisms are not markers of susceptibility to psoriasis. However, the IL17F polymorphism may affect the response to NB-UVB therapy.
Elevated aAbs against IL-17F correlate with disease activity in patients with early rheumatoid arthritis.
The protein encoded by this gene is a cytokine that shares sequence similarity with IL17. This cytokine is expressed by activated T cells, and has been shown to stimulate the production of several other cytokines, including IL6, IL8, and CSF2/GM_CSF. This cytokine is also found to inhibit the angiogenesis of endothelial cells and induce endothelial cells to produce IL2, TGFB1/TGFB, and monocyte chemoattractant protein-1.
, IL-4-induced secreted protein
, melanoma differentiation-associated gene 7 protein
, th2-specific cytokine FISP
, cytokine-like protein Mob-5
, interleukin 17F
, cytokine ML-1
, mutant IL-17F