Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
Alle Spezies anzeigen
Weitere Synonyme anzeigen
Wählen Sie die Spezies und Applikation aus
anti-Mouse (Murine) Antikörper:
anti-Rat (Rattus) Antikörper:
Sie gelangen zu unserer vorgefilterten Suche.
Rat (Rattus) Polyclonal AGER Primary Antibody für FACS, IF (p) - ABIN725900
Huang, Yao, Zhang, Dong, Yu, Sheng: The effect of high-mobility group box 1 protein on activity of regulatory T cells after thermal injury in rats. in Shock (Augusta, Ga.) 2009
Show all 5 Pubmed References
Human Polyclonal AGER Primary Antibody für IHC (fro), IHC (p) - ABIN3044329
Qin, Niu, Wang, Xu, Qiao, Gu: Heparanase induced by advanced glycation end products (AGEs) promotes macrophage migration involving RAGE and PI3K/AKT pathway. in Cardiovascular diabetology 2013
Show all 4 Pubmed References
Human Polyclonal AGER Primary Antibody für IHC (p), WB - ABIN3043529
Wang, Zhang, Liu, Cui, Yang, Li, Du: Tanshinone II A down-regulates HMGB1, RAGE, TLR4, NF-kappaB expression, ameliorates BBB permeability and endothelial cell function, and protects rat brains against focal ischemia. in Brain research 2010
Show all 4 Pubmed References
Rat (Rattus) Polyclonal AGER Primary Antibody für FACS, IF (p) - ABIN725907
Zhu, Yao, Zhang, Dong, Yu, Sheng: Anti-RAGE antibody ameliorates severe thermal injury in rats through regulating cellular immune function. in Acta pharmacologica Sinica 2014
Show all 2 Pubmed References
Human Polyclonal AGER Primary Antibody für IF (p), IHC (p) - ABIN1386239
Durning, Preston-Hurlburt, Clark, Xu, Herold: The Receptor for Advanced Glycation Endproducts Drives T Cell Survival and Inflammation in Type 1 Diabetes Mellitus. in Journal of immunology (Baltimore, Md. : 1950) 2016
Show all 2 Pubmed References
Mouse (Murine) Polyclonal AGER Primary Antibody für IHC (p), WB - ABIN4349236
Yue, Zhou, Zhu, Rao, Busuttil, Kupiec-Weglinski, Lu, Zhai: Hyperglycemia and liver ischemia reperfusion injury: a role for the advanced glycation endproduct and its receptor pathway. in American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons 2015
Cow (Bovine) Polyclonal AGER Primary Antibody für WB - ABIN2786877
Luo, Saiardi, Nagata, Ye, Yu, Jung, Luo, Jain, Sawa, Snyder: GRAB: a physiologic guanine nucleotide exchange factor for Rab3A, which interacts with inositol hexakisphosphate kinase. in Neuron 2001
Show all 2 Pubmed References
Human Polyclonal AGER Primary Antibody für IHC (p), WB - ABIN1397268
Guan, Sheu, Wu, Chiang, Liu: ATP synthase subunit-β down-regulation aggravates diabetic nephropathy. in Scientific reports 2015
Human Polyclonal AGER Primary Antibody für ELISA, WB - ABIN1573995
Milutinovic, Alcorn, Englert, Crum, Oury: The receptor for advanced glycation end products is a central mediator of asthma pathogenesis. in The American journal of pathology 2012
Human Polyclonal AGER Primary Antibody für ICC, IF - ABIN4349231
Zhu, Ito, Nakahara, Nagae, Fuyuno, Nakao, Akahoshi, Nakagawa, Tu, Uchi, Furue: Upregulation of S100P, receptor for advanced glycation end products and ezrin in malignant melanoma. in The Journal of dermatology 2013
results imply that the interaction of matrix AGEs with RAGE plays a role in the TGFbeta2-mediated EMT (zeige ITK Antikörper) of lens epithelial cells and suggest that the blockade of RAGE could be a strategy to prevent PCO and other age-associated fibrosis.
novel findings suggest that HMGB1 (zeige HMGB1 Antikörper) triggered EPC (zeige TCF21 Antikörper) apoptosis in a manner of RAGE-mediated activation of the PERK (zeige EIF2AK3 Antikörper)/eIF2alpha (zeige EIF2A Antikörper) pathway.
Activation of RAGE by AGEs causes up regulation of the transcription factor nuclear factor-kappaB and its target genes. of the RAGE engagement stimulates oxidative stress, evokes inflammatory and fibrotic reactions, which all contribute to the development and progression of devastating cardiovascular disorders.
The lowering of glycative stress via modulation of RAGE-AGE axis or glyoxalase 1 (zeige GLO1 Antikörper) activity is beneficial for tubular homeostasis and the subsequent prevention and treatment of kidney disease, suggesting the possibility of novel therapeutic approaches which target glycative stress.
HMGB1 (zeige HMGB1 Antikörper) attenuates TGF-beta (zeige TGFB1 Antikörper)-induced epithelial-mesenchymal transition of FaDu hypopharyngeal carcinoma cells through regulation of RAGE expression
Correlation among soluble receptors for advanced glycation end-products, soluble vascular adhesion protein-1/semicarbazide-sensitive amine oxidase (zeige AOC3 Antikörper) (sVAP-1 (zeige SNAP47 Antikörper)) and cardiometabolic risk markers in apparently healthy adolescents: a cross-sectional study.
RAGE location was present at the cell membrane of alveolar epithelial cellss in control lungs, while it was almost missing in pulmonary fibrotic tissue.
A capture method based on the VC1 domain reveals new binding properties of the AGER.
These data indicate that RAGE plays a central role in maintaining inflammatory signaling in PDAC that benefits tumor growth.
In conclusion, RAGE is clearly involved in corneal re-epithelialization most probably mediated by signalling via S100 proteins.
Our results suggested that the increased RAGE expression in inflammatory circumstances and interaction with AGEs are risk factors in decreasing of aggrecan (zeige ACAN Antikörper) content in nucleus pulposus.
RAGE is phosphorylated by the ATM (zeige ATM Antikörper) kinase and is recruited to the site of DNA-double-strand break via an early DNA damage response.
receptor for advanced glycation end products (RAGE) was required for stabilization of beta-catenin (zeige CTNNB1 Antikörper) in toluene diisocyanate-induced asthma, identifying protective effects of RAGE blockade in this mouse model
perturbation of Bone marrow mesenchymal stromal cells in diabetes mellitus could be partially explained by chronic RAGE signaling.
RAGE mediates inflammation that contributes to lung damage, in cigarette smoke-induced lung pathology.
Ager deletion enhances ischemic muscle inflammation, angiogenesis, and blood flow recovery in diabetic mice.
HMGB1 (zeige HMGB1 Antikörper) neither exerts influence on cardiac remodeling by binding to RAGE nor induces apoptosis of cardiomyocytes under physiological condition
Our results indicate that lack of RAGE has no significant impact on septic arthritis. However, RAGE-/- mice had significantly higher BMD (zeige BEST1 Antikörper) compared to WT mice, which coincided with lower IL-17A (zeige IL17A Antikörper) in RAGE-/- mice. In sepsis, RAGE deficiency impairs bacterial kidney clearance.
SAA (zeige SAA1 Antikörper) is a potential new uremic toxin involved in uremia-related atherosclerosis through interaction with RAGE.
that selective RAGE regulation reflects a self-protective mechanism to maintain low levels of RAGE ligands
Data show that receptor for advanced glycation end products (RAGE) impairs collateral growth in a diabetic setting and also in a non-diabetic setting, indicting the importance of RAGE and alternate RAGE ligands in the setting of collateral vessel growth.
The advanced glycosylation end product (AGE) receptor encoded by this gene is a member of the immunoglobulin superfamily of cell surface receptors. It is a multiligand receptor, and besides AGE, interacts with other molecules implicated in homeostasis, development, and inflammation, and certain diseases, such as diabetes and Alzheimer's disease. Many alternatively spliced transcript variants encoding different isoforms, as well as non-protein-coding variants, have been described for this gene (PMID:18089847).
advanced glycosylation end product-specific receptor
, RAGE isoform NtRAGE-delta
, RAGE isoform sRAGE-delta
, advanced glycation end product receptor
, advanced glycosylation end product-specific receptor variant 1
, advanced glycosylation end product-specific receptor variant 3
, advanced glycosylation end product-specific receptor variant 4
, advanced glycosylation end product-specific receptor variant 5
, advanced glycosylation end product-specific receptor variant 6
, advanced glycosylation end product-specific receptor variant 7
, advanced glycosylation end product-specific receptor variant 8
, receptor for advanced glycosylation end products
, advanced glycation end-products receptor
, advanced glycosylation end product-specific receptor variant 2