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anti-Human Lamin B1 Antikörper:
anti-Mouse (Murine) Lamin B1 Antikörper:
anti-Rat (Rattus) Lamin B1 Antikörper:
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Cow (Bovine) Monoclonal Lamin B1 Primary Antibody für DB, ELISA - ABIN108542
Weaver, Carson, Walker, Chaly, Lach, Raymond, Brown, Sikorska: Degradation of nuclear matrix and DNA cleavage in apoptotic thymocytes. in Journal of cell science 1996
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Human Monoclonal Lamin B1 Primary Antibody für ICC, FACS - ABIN335391
Broers, Bronnenberg, Kuijpers, Schutte, Hutchison, Ramaekers: Partial cleavage of A-type lamins concurs with their total disintegration from the nuclear lamina during apoptosis. in European journal of cell biology 2003
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Human Polyclonal Lamin B1 Primary Antibody für WB - ABIN4229152
Kanemaru, Tuthill, Takeuchi, Sidell, Wada: Retinoic acid induced downregulation of MYCN is not mediated through changes in Sp1/Sp3. in Pediatric blood & cancer 2008
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Human Polyclonal Lamin B1 Primary Antibody für IHC (p), WB - ABIN3042505
Zhu, Liu, Li, Liu, Wang, Sun, Xiong, Jiang, Zheng, Hu: Protein tyrosine phosphatase receptor U (PTPRU) is required for glioma growth and motility. in Carcinogenesis 2014
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Human Polyclonal Lamin B1 Primary Antibody für ICC, IHC (fro) - ABIN3043687
Liu, Ma, Wu, Li, Zhang, Han, Wang, Reindl, Wu, Ma: Arginine deiminase augments the chemosensitivity of argininosuccinate synthetase-deficient pancreatic cancer cells to gemcitabine via inhibition of NF-?B signaling. in BMC cancer 2014
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Human Polyclonal Lamin B1 Primary Antibody für WB - ABIN2668811
Yang, Fan, Camp, van Buren, Liu, Somcio, Gray, Cheng, Hoff, Ellis: Chronic oxaliplatin resistance induces epithelial-to-mesenchymal transition in colorectal cancer cell lines. in Clinical cancer research : an official journal of the American Association for Cancer Research 2006
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Human Polyclonal Lamin B1 Primary Antibody für IHC (p), IHC - ABIN446989
Fernández Larrosa, Ruíz Grecco, Mengual Gómez, Alvarado, Panelo, Rubio, Alonso, Gómez, Costas: RAC3 more than a nuclear receptor coactivator: a key inhibitor of senescence that is downregulated in aging. in Cell death & disease 2015
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Human Polyclonal Lamin B1 Primary Antibody für WB - ABIN537491
Koenig, Mueller, Hasel, Adler, Menke: Collagen type I induces disruption of E-cadherin-mediated cell-cell contacts and promotes proliferation of pancreatic carcinoma cells. in Cancer research 2006
Cow (Bovine) Polyclonal Lamin B1 Primary Antibody für WB - ABIN2782509
Lee, Seng, Sekine, Hinton, Fu, Avraham, Avraham: Vascular endothelial growth factor mediates intracrine survival in human breast carcinoma cells through internally expressed VEGFR1/FLT1. in PLoS medicine 2007
Cow (Bovine) Monoclonal Lamin B1 Primary Antibody für ELISA, FACS - ABIN4330073
Mai, Wang, Qiu, Tang, Lin, Luo, Yuan, Wang, Chen, Wang, Chen: Tachycardia pacing induces myocardial neovascularization and mobilizes circulating endothelial progenitor cells partly via SDF-1 pathway in canines. in Heart and vessels 2014
Lamin B counteracts the kinesin Eg5 (zeige KIF11 Antikörper) to restrain spindle pole separation during spindle assembly.
These results suggest that the nuclear lamins and progerin have marginal roles in the activation of the antioxidant Nrf2 (zeige GABPA Antikörper) response to arsenic and cadmium.
We show that epithelial cells failing to undergo proliferation arrest during TGF-beta (zeige TGFB1 Antikörper)-induced EMT (zeige ITK Antikörper) sustain mitotic abnormalities due to failed cytokinesis, resulting in aneuploidy. This genomic instability is associated with the suppression of multiple nuclear envelope proteins implicated in mitotic regulation and is phenocopied by modulating the expression of LaminB1.
The primary response of cells to various stresses leading to senescence consists of the down-regulation of LBR (zeige LBR Antikörper) and LB1 (zeige CKAP2 Antikörper) to attain reversal of the chromatin architecture.
Data show that lamins A and B are differently processed in staurosporine and beta-Amyloid peptide (zeige APP Antikörper) fragments Abeta42-treated cells.
The tail domain of lamin B1 is more strongly modulated by divalent cations than lamin A (zeige LMNA Antikörper).
An upstream mutation alters LMNB1 gene expression in autosomal dominant adult-onset demyelinating leukodystrophy.
Deregulation of LMNB1 expression induces modified splicing of several genes, likely driven by raver-2 (zeige RAVER2 Antikörper) overexpression, and suggest that an alteration of mRNA processing could be a pathogenic mechanism in adult-onset autosomal dominant leukodystrophy.
Lamin B1 levels are increased in oligodendrocytes, the cell type that produces myelin in the central nervous system.
Nuclear envelope remodelling during human spermiogenesis involves somatic B-type lamins and a spermatid-specific B3 lamin (zeige LMNA Antikörper) isoform.
LMNB1-related autosomal-dominant leukodystrophy is a slowly progressive neurological disease with survival lasting more than two decades after clinical onset.
While the distribution patterns of both lamins closely paralleled the respective stages of mitosis, Nup160 (zeige NUP160 Antikörper) localization in metaphase oocytes corresponded to that in mitotic prometaphase rather than metaphase.
Here we report that in primary mouse cortical neurons, LMNB1 overexpression reduces axonal outgrowth, whereas deficiency of endogenous Lmnb1 results in aberrant dendritic development.
Analysis of lamin B1 expression in different regions of the brain in wild-type animals showed that the level of Lmnb1 transcript decreased significantly in an age-dependent manner.
Lamin-B1 is required for organization of lamin-A/C (zeige LMNA Antikörper) and lamin-B2 (zeige LMNB2 Antikörper) in mouse embryonic stem cells and Lamin-B1 is required for organization of lamin-A/C (zeige LMNA Antikörper) and lamin-B2 (zeige LMNB2 Antikörper) in differentiated cells
farnesylation of lamin B1--but not lamin B2 (zeige LMNB2 Antikörper)--is crucial for brain development and for retaining chromatin within the bounds of the nuclear lamina during neuronal migration.
lamin B1 overexpression mediates oligodendrocyte cell-autonomous neuropathology in adult-onset autosomal-dominant leukodystrophy.
We conclude that lamin B1 is a modifier gene of major effect for Neural tube defects resulting from loss of Grhl3 (zeige GRHL3 Antikörper) function
Lamin B1 is lost from primary human and murine cell strains when they are induced to senesce.
Lamin B1 is essential for brain development and is required for the integrity of the nuclear lamina.
Absence of both Lmnb1 and Lmnb2 (zeige LMNB2 Antikörper) in keratinocytes has no effect on cell proliferation or the development of skin and hair.
The nuclear lamina consists of a two-dimensional matrix of proteins located next to the inner nuclear membrane. The lamin family of proteins make up the matrix and are highly conserved in evolution. During mitosis, the lamina matrix is reversibly disassembled as the lamin proteins are phosphorylated. Lamin proteins are thought to be involved in nuclear stability, chromatin structure and gene expression. Vertebrate lamins consist of two types, A and B. This gene encodes one of the two B type proteins, B1. Alternative splicing results in transcript variants and a duplication of this gene is associated with autosomal dominant adult-onset leukodystrophy (ADLD).