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anti-Human TGFB1 Antikörper:
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anti-Mouse (Murine) TGFB1 Antikörper:
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Various Species Monoclonal TGFB1 Primary Antibody für CyTOF, ELISA (Capture) - ABIN4900861
Phillips: Rapid analysis of inflammatory cytokines in cerebrospinal fluid using chip-based immunoaffinity electrophoresis. in Electrophoresis 2004
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Various Species Monoclonal TGFB1 Primary Antibody für CyTOF, ELISA (Capture) - ABIN4900860
Rafei, Wu, Annabi, Lejeune, François, Galipeau: A GMCSF and IL-15 fusokine leads to paradoxical immunosuppression in vivo via asymmetrical JAK/STAT signaling through the IL-15 receptor complex. in Blood 2007
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Human Polyclonal TGFB1 Primary Antibody für IF (p), IHC (p) - ABIN724685
Kou, Hu, Yao, Wang, Shen, Kang, Hong: Transforming growth factor-?1 promotes Treg commitment in nasal polyposis after intranasal steroid treatment. in Inflammation research : official journal of the European Histamine Research Society ... [et al.] 2013
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Human Polyclonal TGFB1 Primary Antibody für WB - ABIN223608
Tamaki, Hatano, Taura, Tada, Kodama, Nitta, Iwaisako, Seo, Nakajima, Ikai, Uemoto: CHOP deficiency attenuates cholestasis-induced liver fibrosis by reduction of hepatocyte injury. in American journal of physiology. Gastrointestinal and liver physiology 2008
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Human Monoclonal TGFB1 Primary Antibody für FACS - ABIN4898892
Yamauchi, Ueki, Konno, Ito, Takeda, Nakamura, Nishikawa, Moritoki, Omokawa, Saga, Hirokawa: The effect of hepatocyte growth factor on secretory functions in human eosinophils. in Cytokine 2016
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Human Monoclonal TGFB1 Primary Antibody für CyTOF, FACS - ABIN4900169
Boswell, Sharif, Alisa, Pereira, Williams, Behboudi et al.: Induction of latency-associated peptide (transforming growth factor-β(1)) expression on CD4+ T cells reduces Toll-like receptor 4 ligand-induced tumour necrosis factor-α production in a transforming ... in Immunology 2011
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Human Monoclonal TGFB1 Primary Antibody für FACS - ABIN4897280
Azukizawa, Döhler, Kanazawa, Nayak, Lipp, Malissen, Autenrieth, Katayama, Riemann, Weih, Berberich-Siebelt, Lutz: Steady state migratory RelB+ langerin+ dermal dendritic cells mediate peripheral induction of antigen-specific CD4+ CD25+ Foxp3+ regulatory T cells. in European journal of immunology 2011
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Human Monoclonal TGFB1 Primary Antibody für ELISA, FACS - ABIN4359062
Makboul, Makboul, Abdelhafez, Hassan, Youssif: Evaluation of the effect of fractional CO2 laser on histopathological picture and TGF-β1 expression in hypertrophic scar. in Journal of cosmetic dermatology 2014
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Human Monoclonal TGFB1 Primary Antibody für EIA, Func - ABIN114558
Crawford, Stellmach, Murphy-Ullrich, Ribeiro, Lawler, Hynes, Boivin, Bouck: Thrombospondin-1 is a major activator of TGF-beta1 in vivo. in Cell 1998
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Human Polyclonal TGFB1 Primary Antibody für ELISA, ICC - ABIN4359055
Barsby, Guest: Transforming growth factor beta3 promotes tendon differentiation of equine embryo-derived stem cells. in Tissue engineering. Part A 2013
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TGF-beta1 regulated pAKT (zeige AKT1 Antikörper) and IFNgamma expressions were associated with epithelial cell survival in rhesus macaque colon explants and suggest a potential role of mucosal TGF-beta1 in regulating intestinal homeostasis and EC integrity.
These findings suggest that FBLN5 (zeige FBLN5 Antikörper) may interfere with choroidal neovascularization by downregulating VEGF (zeige VEGFA Antikörper), CXCR4 (zeige CXCR4 Antikörper), and TGFB1 expression and inhibiting choroidl endothelial cell proliferation.
SIV infection of rhesus macaques results in the emergence of IL-17 (zeige IL17A Antikörper)-expressing cells during the acute phase. This subpopulation appears at day 14 postinfection concomitantly with an increase in TGF-beta and IL-18 (zeige IL18 Antikörper) expression.
SIV-infected macaques exhibiting progression to AIDS displayed greater expression of TGF-beta and indoleamine 2,3 dioxygenase in CD8 (zeige CD8A Antikörper)+ T cells from mesentric lymph nodes.
transforming growth factor beta (TGFbeta) is required for hematopoietic progenitor cell specification. The requirement for TGFbeta is two fold and sequential: autocrine via Tgfbeta1a and Tgfbeta1b produced in the endothelial cells themselves, followed by a paracrine input of Tgfbeta3 from the notochord, suggesting that the former programs the hemogenic endothelium and the latter drives endothelial-to-hematopoietic tran...
The fine tuning of TGF-beta signaling derives from positive and negative control by Ldb2a (zeige LDB2 Antikörper).
TGFbeta1a regulates zebrafish posterior lateral line formation via Smad5 (zeige SMAD5 Antikörper) mediated pathway.
PCSK7 (zeige PCSK7 Antikörper) is essential for zebrafish development and regulates the expression and proteolytic cleavage of TGFbeta1a.
TGFbeta signaling orchestrates the beneficial interplay between scar-based repair and cardiomyocyte-based regeneration to achieve complete heart regeneration.
TGFbeta1 plays a role in zebrafish keratocyte migration.
data presented show that Zili (zeige PIWIL2 Antikörper) suppresses TGF-beta signaling by physically associating with Smad4 (zeige SMAD4 Antikörper) and preventing the formation of Smad2 (zeige SMAD2 Antikörper)/3/4 and Smad1 (zeige SMAD1 Antikörper)/5/9/4 complexes
TGF-beta1 acts at multiple sites, including LH receptor (zeige LHCGR Antikörper), 20beta-HSD (zeige HAL Antikörper) and membrane progestin receptor-beta (zeige PAQR8 Antikörper), to inhibit zebrafish oocyte maturation
These data suggest Pez (zeige PTPN14 Antikörper) plays a crucial role in organogenesis by inducing TGFbeta and epithelial-mesenchymal transition.
Data show that Rock2 (zeige ROCK2 Antikörper) acts as a negative regulator of the TGFbeta signaling pathway.
These data indicate that TGF-beta signaling is crucial for the function of the transition zone, which in turn may affect the regulation of cilia length.
R-Smads are the key components of TGFbeta beta signals in germ layer induction. SCP3 (zeige SYCP3 Antikörper) serves as a vegetally enriched, intrinsic factor (zeige GIF Antikörper) to ensure a prepared status of Smads for their activation.
the present in vitro system, which permits not only the cell contraction-mediated cell sorting but also the TGF-b-directed mesodermal induction such as cartilage formation, may fairly reflect the embryogenesis in vivo.
Loss of XTgfbi impaired blastopore formation and dorsal tissue morphogenesis.
TGF-beta signaling has a role in nuclear localization of transcription factor Smad4 (zeige SMAD4 Antikörper)
sortilin (zeige SORT1 Antikörper) negatively regulates TGF-beta signaling by diverting trafficking of precursor proteins to the lysosome during transit through the biosynthetic pathway
CRT (zeige SLC6A8 Antikörper) regulates TGF-beta1-induced-EMT (zeige ITK Antikörper) through modulating Smad (zeige SMAD1 Antikörper) signaling
The results suggest that selective RNA decay via TGF-beta and BMP4 (zeige BMP4 Antikörper) signaling is critical for specifying the developmental fate of specific human embryonic cell lineages.
TGF-beta1 levels were found significantly higher in the hemodialysis patients than those of the control groups.
Smad7 (zeige SMAD7 Antikörper) expression in necrotizing enterocolitis macrophages interrupts TGF-beta signaling and promotes NF-kappaB (zeige NFKB1 Antikörper)-mediated inflammatory signaling in these cells through increased expression of IKK-beta (zeige IKBKB Antikörper)
P311 (zeige C5orf13 Antikörper) is a novel TGFbeta1/Smad (zeige SMAD1 Antikörper) signaling-mediated regulator of transdifferentiation in epidermal stem cells during cutaneous wound healing.
Paroxysmal atrial fibrillation is associated with a higher TGF-beta1 level. Lower TGF-beta1 level in AF patients could be a cause of recurrent AF after catheter ablation.
This study demonstrated that the Indian subjects TGFB1 rs1800469 were associated with higher and lower morphine use.
UCN (zeige UCN Antikörper) counteracted TGF-beta-mediated cPLA2 (zeige PLA2G4A Antikörper) expression and activation, thereby contributing to TGF-beta-mediated mitoinhibition of VSMCs
High TGFB1 expression is associated with prostate cancer.
Cardiac overexpression of TGF-beta1 led to an increase in fibrosis and myocyte size in the atria, and to progressive P-wave prolongation, increasing atrial fibrillation susceptibility.
Activated TGF-beta signaling rescued miR (zeige MYLIP Antikörper)-143-reduced FSHR (zeige FSHR Antikörper) and intracellular signaling molecules, and miR (zeige MYLIP Antikörper)-143-induced porcine granulosa cell apoptosis.
TGF-beta1-induced epithelial-myofibroblast plasticity is FAK (zeige PTK2 Antikörper)- dependent, whereas TGF-beta1-induced apoptosis is favored when FAK (zeige PTK2 Antikörper) signaling is inhibited.
this study shows that TGF-beta1 protects intestinal integrity and influences Smad (zeige SMAD1 Antikörper) and MAPK (zeige MAPK1 Antikörper) signal pathways in intestinal epithelium cells after TNF-alpha (zeige TNF Antikörper) challenge
this study shows that anemonin may ameliorate LPS (zeige IRF6 Antikörper)-induced intestinal injury and improve restoration by regulating the TGF-b1 and EGFR (zeige EGFR Antikörper) signaling pathways
The results indicated that TGF-beta-1 was associated with the restoration of intestinal morphology and barrier function following weaning stress.
Data (including data from in vitro and in vivo experiments) suggest that day 14 elongated conceptus secretes proteins that up-regulate TGFbeta1 mRNA and TGFbeta1 expression in endometrium; TGFbeta1 may be important during pregnancy maintenance.
The effects of semen, spermatozoa in extender, or extender alone on the expression of TGF-beta1, IL-10 (zeige IL10 Antikörper), and IL-6 (zeige IL6 Antikörper) in ovarian follicles are reported.
Dietary (1,3/1,6)-beta-D-glucan reduced the mRNA expression of transforming growth factor (TGF) beta2 (zeige TGFB2 Antikörper) and tended to reduce the mRNA expression of TGF-beta1 in lung tissue of neonatal piglet.
TGF-beta1, via TGF-beta1 receptor I and p38 MAPK (zeige MAPK14 Antikörper) signaling, reduces CFTR (zeige CFTR Antikörper) expression to impair CFTR (zeige CFTR Antikörper)-mediated anion secretion, which would likely compound the effects associated with mild CFTR (zeige CFTR Antikörper) mutations and ultimately would compromise male fertility.
High yield isolation of BMP-2 (zeige BMP2 Antikörper) from bone and in vivo activity of a combination of BMP-2 (zeige BMP2 Antikörper)/TGF-beta1.
TGF-beta1 modulates the expression of syndecan-4 (zeige SDC4 Antikörper) in cultured vascular endothelial cells in a biphasic manner.
Taken together, Staphylococcus aureus induces TGF-beta1 and bFGF (zeige FGF2 Antikörper) expression through the activation of AP-1 (zeige JUN Antikörper) and NF-kappaB (zeige NFKB1 Antikörper) in bovine mammary gland fibroblasts.
The results identify TGFB1 and ESRRA (zeige ESRRA Antikörper) as likely transcriptional regulators of rumen epithelial development and energy metabolism, respectively, and provide targets for modulation of rumen development and function in the growing calf.
the combined treatment with TGF-beta1 and BMP-7 (zeige BMP7 Antikörper) or treatment first with TGF-beta1 followed by BMP-7 (zeige BMP7 Antikörper) was more effective than other treatment groups in both chondrogenic differentiation and SZP (zeige PRG4 Antikörper) secretion.
Tenascin-X (zeige TNXB Antikörper) promotes activation of latent TGF-beta1 and subsequent epithelial to mesenchymal transition in mammary epithelial cells.
a detailed computational model for TGF-beta signalling that incorporates elements of previous models together with crosstalking between Smad1 (zeige SMAD1 Antikörper)/5/8 and Smad2 (zeige SMAD2 Antikörper)/3 channels through a negative feedback loop dependent on Smad7 (zeige SMAD7 Antikörper).
Endogenous TGF-beta1 became more bioactive following activation of the transgene protein product in chondrocytes.
A novel peptide, P2K, regulating TGF-beta1 signaling had an anabolic effect on bovine intervertebral disc cells and rabbit degenerated discs.
Data show that TGF-beta pathways operate during ovarian fetal development, and fibrillin 3 (zeige FBN3 Antikörper) is highly expressed at a critical stage early in developing human and bovine fetal ovaries.
Role of TGF-beta1 and TNF-alpha (zeige TNF Antikörper) in IL-1beta (zeige IL1B Antikörper) mediated activation of proMMP-9 in pulmonary artery smooth muscle cells: involvement of an aprotinin sensitive protease.
PAR1 (zeige F2R Antikörper) in its inactive unligated state functions as a scaffold for TGFbetaRII to downregulate TGF-beta signaling, and thereby promote embryonic stem cell transition to functional endothelial cells.
HSP22 (zeige HSPB8 Antikörper) functions as a negative regulator in the TGF-beta-stimulated migration of osteoblasts.
Lnc-LFAR1 binds directly to Smad2 (zeige SMAD2 Antikörper)/3 and promotes transcription of TGFbeta, Smad2 (zeige SMAD2 Antikörper), Smad3 (zeige SMAD3 Antikörper), Notch2 (zeige NOTCH2 Antikörper) and Notch3 (zeige NOTCH3 Antikörper) which, in turn, results in TGFbeta and Notch (zeige NOTCH1 Antikörper) pathway activation.
how that limiting CCR2 (zeige CCR2 Antikörper)(+) monocytic myeloid-derived suppressor cells accumulation reduces the pulmonary contents of TGF-beta1, TIMP-1 (zeige TIMP1 Antikörper) and collagen after silica treatment
RORgamma mediates epithelial-mesenchymal transition of hepatocytes during hepatic fibrosis facilitated by TGFbeta1.
these data unravel the previously unrecognized role of TGF-beta1 in promoting Treg viability, coinciding with the pronounced immunomodulatory role of these cells during later phase of oropharyngeal candidiasis infection
findings define a novel physiological function of Shp2 (zeige PTPN11 Antikörper) in TGF-beta1/MMP12 (zeige MMP12 Antikörper)-dependent emphysema, adding insights into potential etiologies for this chronic lung disorder.
TGF-beta and PPARalpha (zeige PPARA Antikörper) signaling pathways are involved in radiation-induced heart fibrosis, metabolic dysregulation, and impaired heart contractility, a pathophysiological condition that is often observed in patients that received high radiation doses in thorax.
Data show that Clostridium difficile Toxin A induces TGF-beta1 signaling pathway activation and suggest that this pathway might play a protective role against the effect of C. difficile-toxin.
Data indicate that cardiac contractility modulation (CCM) therapy exerted protective effects against myocardial fibrosis potentially by inhibiting TGF-beta1/Smad3 (zeige SMAD3 Antikörper) signaling pathway in chronic heart failure .
study suggested that TGF-beta1/Smad3/smad7 is a major pathway which plays an important role in the regulation of the IUA and specific inhibitor of Smad3 (SIS3) may provide a new therapeutic strategy for IUA.
cell therapy promoted TGF-beta1 expression in nucleus pulposus, leading to anti-inflammatory effects via the inhibition of NF-kappaB (zeige NFKB1 Antikörper), and the amelioration of disc degradation.
that prenatal tracheal occlusion increases TGF-beta/Rho kinase (zeige ROCK1 Antikörper) pathway, myofibroblast differentiation, and matrix deposition in neonatal rabbit and human congenital diaphragmatic hernia lungs
observation. Based on the above results, we conclude that TGF-beta1-immobilized PLGA-gelatin scaffold seeded with ASCs considerably enhances the quality of the tissue-engineered cartilage, therefore, advancing the field of cartilage tissue engineering
Smad7 (zeige SMAD7 Antikörper) plays a crucial role in antagonizing epithelial-mesenchymal transition induced by TGFbeta signaling and is a Notch (zeige NOTCH1 Antikörper) signaling target in limbal epithelial stem cells.
the role of IL-10 in inhibited allograft rejection may be associated with CD4+CD25+ regulatory T cells and IL-10, and may be independent of TGF-b
After NOTCH1 (zeige NOTCH1 Antikörper) knockdown and TGFB1 stimulation, rabbit mesenchymal stem cells expressed higher levels of proteoglycan (zeige Vcan Antikörper) and collagen II.
TGF-beta1 gene transcription significantly correlates with the surgical vaginal and dermal wound closure rate.
TGF beta is involved in the pathogenesis of tympanosclerosis.
There was significantly higher expression of TGF-beta1 and MMP-9 (zeige MMP9 Antikörper) in nasal mucosa of experimental allergic rhinitis guinea pigs than in controls.
Within the limitations of the study design, production of COMP (zeige COMP Antikörper) during healing of skin wounds does not appear to be influenced by wound type or anatomic site, nor does it appear to be correlated with TGF-beta1 concentrations.
Peritoneal TGF-beta(1) concentration was higher in horses with severe gastrointestinal diseases, in horses with an altered peritoneal fluid, and in nonsurvivors.
This gene encodes a member of the transforming growth factor beta (TGFB) family of cytokines, which are multifunctional peptides that regulate proliferation, differentiation, adhesion, migration, and other functions in many cell types. Many cells have TGFB receptors, and the protein positively and negatively regulates many other growth factors. The secreted protein is cleaved into a latency-associated peptide (LAP) and a mature TGFB1 peptide, and is found in either a latent form composed of a TGFB1 homodimer, a LAP homodimer, and a latent TGFB1-binding protein, or in an active form composed of a TGFB1 homodimer. The mature peptide may also form heterodimers with other TGFB family members. This gene is frequently upregulated in tumor cells, and mutations in this gene result in Camurati-Engelmann disease.
transforming growth factor beta1
, transforming growth factor, beta 1
, transforming growth factor, beta-induced, 68kDa
, transforming growth factor-beta-induced protein ig-h3-like
, transforming growth factor beta-1-like
, transforming growth factor beta 4
, transforming growth factor beta-1
, TGF-beta 1 protein
, latency-associated peptide
, transforming growth factor, beta 1 (Camurati-Engelmann disease)
, transforming growth factor-beta-1
, TGF-beta 1
, transforming growth factor-beta 1
, transforming growth factor, beta-1
, transforming growth factor beta 1
, regulatory protein
, transforming growth factor-beta
, tgf beta
, tgf-beta 5
, transforming gorwth factor-Beta5
, transforming growth factor-B5
, transforming growth factor-beta 5
, Transforming growth factor beta-1