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Human ENOS ELISA Kit für Sandwich ELISA - ABIN414413
Ge, Mo, Januzzi, Jin, Yang, Han, Wood, Levine: B-type natriuretic peptide, vascular endothelial growth factor, endothelin-1, and nitric oxide synthase in chronic mountain sickness. in American journal of physiology. Heart and circulatory physiology 2011
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Rat (Rattus) ENOS ELISA Kit für Sandwich ELISA - ABIN416101
Suo, Liu, Ferreri, Zhang, Liu, Mu, Han: Impact of matrine on inflammation related factors in rat intestinal microvascular endothelial cells. in Journal of ethnopharmacology 2009
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Human ENOS ELISA Kit für Sandwich ELISA - ABIN365093
Yuan, Jin, Wei, Liu, Xue, Wang et al.: Propranolol given orally for proliferating infantile haemangiomas: analysis of efficacy and serological changes in vascular endothelial growth factor and endothelial nitric oxide synthase in 35 ... in The British journal of oral & maxillofacial surgery 2013
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Rat (Rattus) ENOS ELISA Kit für Competition ELISA - ABIN579853
Dolkart, E, S, S, P, Aa: Temporal determination of lung NO system and COX-2 upregulation following ischemia-reperfusion injury. in Experimental lung research 2014
Mouse (Murine) ENOS ELISA Kit für Sandwich ELISA - ABIN366370
Cheng, Jiang, Hu, Lv: Potential mechanism for endothelial progenitor cell therapy in acute myocardial infarction: Activation of VEGF- PI3K/Akte-NOS pathway. in Annals of clinical and laboratory science 2013
Rat (Rattus) ENOS ELISA Kit für Competition ELISA - ABIN772134
Zhang, Zou, Lin, Bian, Meng, Liu: Effect of Excessive Potassium Iodide on Rat Aorta Endothelial Cells. in Biological trace element research 2015
Rat (Rattus) ENOS ELISA Kit für Sandwich ELISA - ABIN367489
Salgado, Pereiro, López-Doval, Lafuente: Initial study on the possible mechanisms involved in the effects of high doses of perfluorooctane sulfonate (PFOS) on prolactin secretion. in Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association 2015
The NOS3 variant Glu298Asp was not associated with Coronary Artery Disease and the tested lipid traits but had a strong association with blood pressure.
NOSTRIN (zeige NOSTRIN ELISA Kits) inhibited production of nitric oxide (NO) by suppressing the activation of endothelial nitric oxide synthase (eNOS).
Protein engineering to develop a redox insensitive endothelial nitric oxide synthase.
Our results suggest that haplotype HT1 in the eNOS gene may be capable of identifying a subset of hepatocellular carcinoma patients who are resistant to sorafenib.
Studied effect of Aliskiren on interleukin-6 (zeige IL6 ELISA Kits), endothelial nitric oxide synthase and caveolin-1 (zeige CAV1 ELISA Kits) in human aortic endothelial cells. Findings suggest that aliskiren reverses the effects of IL-6 (zeige IL6 ELISA Kits) on eNOS and caveolin-1 (zeige CAV1 ELISA Kits) thru increase in eNOS phosphorylation and nitric oxide production, decrease caveolin-1 (zeige CAV1 ELISA Kits) phosphorylation, and decrease the interaction between eNOS and caveolin-1 (zeige CAV1 ELISA Kits).
Studied genetic polymorphisms of human nitric oxide synthase (zeige NOS ELISA Kits) 3(eNOS) and it's association with Coronary Heart Disease (CHD (zeige CHDH ELISA Kits)); results show eNOS -786T/C, intron 4b/4a and 894G/T genotypes and haplotypes (G-C-B and T-C-B) are significantly associated with the development of CHD (zeige CHDH ELISA Kits) in combination with potential risk factors.
treatment with Mito-TEMPO, a mitochondrial-specific superoxide scavenger, recovered mitochondrial fission-fusion imbalance and blunted mitochondrial superoxide production, and reduced the IDH2 (zeige IDH2 ELISA Kits) knockdown-induced decrease in MnSOD (zeige SOD2 ELISA Kits) expression, eNOS phosphorylation and NO production in endothelial cells
The phosphorylated eNOS and Akt (zeige AKT1 ELISA Kits) levels and VEGF (zeige VEGFA ELISA Kits) levels in ischemic muscle were lower in SMP30 (zeige RGN ELISA Kits) KO and old mice than in young mice. Deficiency of SMP30 (zeige RGN ELISA Kits) exacerbates oxidative stress related to NADPH oxidase (zeige NOX1 ELISA Kits) activity enhancement and impairs eNOS activity, which leads to rarefaction of angiogenesis induced by ischemia
Aspirin rescued TNF-alpha (zeige TNF ELISA Kits)-mediated eNOS downregulation coupled with endothelial dysfunction by inhibiting NF-kappaB (zeige NFKB1 ELISA Kits)-dependent transcriptional miR (zeige MLXIP ELISA Kits)-155 biogenesis. Thus, the redox-sensitive NF-kappaB (zeige NFKB1 ELISA Kits)/miR (zeige MLXIP ELISA Kits)-155/eNOS axis may be crucial in the pathogenesis of vascular disorders including preeclampsia
Our findings emphasize that manipulating mitochondrial Ca(2 (zeige CA2 ELISA Kits)+) uptake may represent a novel strategy to control eNOS-mediated NO(*) production
The increase of oxidative/nitrosative stress induced by NOS-3 overexpression induced DNA damage, p53 (zeige TP53 ELISA Kits), CD95 (zeige FAS ELISA Kits)/CD95L (zeige FASL ELISA Kits) expression and cell death in hepatocellular carcinoma cells. The effectiveness of the gene therapy has been demonstrated in vitro and in vivo.
eNOS deficiency is associated with an upregulation of XOR (zeige XDH ELISA Kits) facilitating the nitrate-nitrite-NO pathway and decreasing the generation of ROS (zeige ROS1 ELISA Kits). This interplay between XOR (zeige XDH ELISA Kits) and eNOS is proposed to play a significant role in NO homeostasis and blood pressure regulation.
On normal salt diet, renal CuZnSOD (zeige SOD1 ELISA Kits) and ECSOD (zeige SOD3 ELISA Kits) proteins were similar but renal MnSOD (zeige SOD2 ELISA Kits) was lower in hGRK4g486V than Non-T mice and remained low on high salt diet. hGRK4gammawild-type mice were normotensive and hGRK4g142V mice were hypertensive but both were salt-resistant and in normal redox balance. Chronic tempol treatment partially prevented the salt-sensitivity of hGRK4g486V mice.
We conclude that pp60(Src (zeige SRC ELISA Kits)) can directly inhibit DDAH (zeige DDAH2 ELISA Kits) II and this is involved in the increased ADMA levels that enhance eNOS uncoupling during the development of acute lung injury (ALI).
these data reveal a novel NOS (zeige NOS ELISA Kits)-independent role for BH4 in the regulation of mitochondrial redox signalling and bioenergetic metabolism
we show that TRPV4 (zeige TRPV4 ELISA Kits) is activated both by damage associated molecular pattern HMGB1 (zeige HMGB1 ELISA Kits) and collagen in diseased Kupffer cells that in turn activate the endothelial NOS (NOS3) to release nitric oxide (NO). The diffusible NO acts in a paracrine fashion in neighboring hepatocytes to deactivate the redox toxicity induced by CYP2E1 (zeige CYP2E1 ELISA Kits)
Omentin (zeige ITLN1 ELISA Kits) protects against lipopysaccharides-induced acute respiratory distress syndrome through suppressing pulmonary inflammation and promoting endothelial barrier via an Akt (zeige AKT1 ELISA Kits)/NOS3-dependent mechanism.
Endothelial cell Gch1 (zeige GCH1 ELISA Kits) and BH4-dependent eNOS regulation play pivotal roles in maintaining vascular homeostasis in resistance arteries.
(-)-Epicatechin counteracts the negative effects that high glucose or simulated type 2 diabetes has on endothelial nitric oxide synthase function.
These results suggest that S-glutathionylation of eNOS within the microvascular endothelial cells inhibited NO production and enhanced TLR4 (zeige TLR4 ELISA Kits) activity, which were implicated in the pathogenesis of necrotizing enterocolitis.
S1179D substitution in eNOS increased the rate of flavin reduction, altered the conformational equilibrium of the reductase domain, and increased the rate of its conformational transitions.
These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (zeige CTNNB1 ELISA Kits) and link eNOS-derived NO to the modulation by VEGF (zeige VEGFA ELISA Kits) of Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits)-induced endothelial cell proliferation.
Endothelial cell autophagy maintains shear stress-induced nitric oxide generation via glycolysis-dependent ATP/P2Y1 receptor (zeige P2RY1 ELISA Kits) signaling to endothelial nitric oxide synthase.
TNFalpha (zeige TNF ELISA Kits) reduces eNOS activity in bovine endothelial cells through serine 116 phosphorylation and Pin1 (zeige PIN1 ELISA Kits) binding.
Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (zeige AKT1 ELISA Kits)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.
Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.
eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation
In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.
A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.
Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (zeige CAV1 ELISA Kits))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).
NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.
Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.
Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (zeige EGFR ELISA Kits) pathway in porcine aortic endothelial cells.
Data suggest that pig sperm contain bNOS (zeige NOS1 ELISA Kits), iNOS (zeige NOS2 ELISA Kits), and eNOS; up-regulation of NOS (zeige NOS ELISA Kits) by leptin (zeige LEP ELISA Kits) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.
Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (zeige NOS1 ELISA Kits) expression in endomyocardium of normal swine.
Data suggest that angiotensin II regulates nNOS (zeige NOS1 ELISA Kits) and eNOS expression and NOS (zeige NOS ELISA Kits) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.
Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.
Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.
Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.
Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.
The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.
Kidneys from circulation-restricted fetuses showed endothelial nitric oxide synthase phosphorylation inhibition.
Antidiabetic drug pioglitazone protects the heart via activation of endothelial nitric oxide synthase (eNOS/Nos3) pathway in a rabbit model of myocardial infarction.
VEGFR2 (zeige KDR ELISA Kits) activation was not affected by Slit2 (zeige SLIT2 ELISA Kits), but eNOS phosphorylation was diminished
Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (zeige NOS2 ELISA Kits) along the radial arteries in the developing placenta.
Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.
eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.
Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.
Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.
, NOS type III
, constitutive NOS
, endothelial NOS
, nitric oxide synthase, endothelial
, endothelial nitric oxide synthase
, nitric oxide synthase 3 (endothelial cell)
, nitric oxide synthase, endothelial-like
, endothelial nitric oxide synthase 3
, endothelial nitric oxide synthase NOS3