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The glycoprotein encoded by VWF functions as both an antihemophilic factor carrier and a platelet-vessel wall mediator in the blood coagulation system. Zusätzlich bieten wir Ihnen VWF Antikörper (518) und VWF Proteine (21) und viele weitere Produktgruppen zu diesem Protein an.
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Rat (Rattus) VWF ELISA Kit für Sandwich ELISA - ABIN367459
Baraka, Guemei, Gawad: Role of modulation of vascular endothelial growth factor and tumor necrosis factor-alpha in gastric ulcer healing in diabetic rats. in Biochemical pharmacology 2010
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Human VWF ELISA Kit für Sandwich ELISA - ABIN2703573
Ma, Su, Zhang, Ling, Yin, Bai, Ruan: The co-influence of VWD type 2B/2M mutations in the A1 domain and platelet GPIbα on the rate of cleavage to VWF by ADAMTS13. in Thrombosis research 2015
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Rat (Rattus) VWF ELISA Kit für Sandwich ELISA - ABIN416122
Imaoka, Sayama, Suzuki, Jindo, Sanbuissho: Effect of Hypertension on the Occurrence of Micro-hemorrhage in the Pancreatic Islet of Dahl Salt-sensitive Rats. in Journal of toxicologic pathology 2012
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Human VWF ELISA Kit für Sandwich ELISA - ABIN612793
Zimmerman, Ruggeri: von Willebrand disease. in Human pathology 1987
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Human VWF ELISA Kit für Sandwich ELISA - ABIN365425
Tong, Wan, Zhang, Duan, Tang, Chen, Tang, Su: Vascular endothelial cell injury partly induced by mesenteric lymph in heat stroke. in Inflammation 2014
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Human VWF ELISA Kit für Sandwich ELISA - ABIN2685886
Morton, Griffin, Pepper, Barnes: The interaction between collagens and factor VIII/von Willebrand factor: investigation of the structural requirements for interaction. in Thrombosis research 1984
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Rabbit VWF ELISA Kit für Competition ELISA - ABIN415967
Du, Wu, Qing, Wang, Liang, Yu, Tang: Systemic and flap inflammatory response associates with thrombosis in flap venous crisis. in Inflammation 2015
The >30 nm macroglycopeptide separating the two domains of GPIbalpha (zeige GP1BA ELISA Kits) transmits force on the VWF-GPIbalpha (zeige GP1BA ELISA Kits) bond (whose lifetime is prolonged by leucine-rich repeat domain unfolding) to the juxtamembrane mechanosensitive domain to enhance its unfolding, resulting in unfolding cooperativity at an optimal force.
von Willebrand factor processing and function has been summarized using various biophysical techniques. (Review)
Soluble VWF was statistically elevated in major depressive disorder.
Rare genetic variants in the ADAMTS13 (zeige ADAMTS13 ELISA Kits) on Willebrand factor-binding domain contribute to pediatric stroke.
results indicate that VWF multimer accumulation on endothelium, as occurs in congenital thrombotic thrombocytopenic purpura, promotes the activation of the complement alternative pathway that proceeds until C5 cleavage with the formation of C5a and the terminal C5b-9 complex
Findings also identify VWF as a new complement regulator on vascular endothelial cells and suggest that VWF has a protective effect on endothelial cells and complement-mediated injury.
LVAD recipients achieved a new hemostatic equilibrium characterized by infrequent major hemorrhagic and thrombotic events, despite a mildly impaired vWF function and a markedly enhanced thrombin (zeige F2 ELISA Kits) formation.
These results demonstrate that cancer cells of non-endothelial origin can acquire de novo expression of VWF, which can enhance processes, including endothelial and platelet adhesion and extravasation, that contribute to cancer metastasis.
level of vWF-ag in the chronic phase correlated with the amounts of VAT and PAT, but not with subcutaneous adipose tissue
both in acute and chronic cerebrovascular disease patients, ADAMTS13 (zeige ADAMTS13 ELISA Kits) levels were significantly decreased, with the lowest ADAMTS13 (zeige ADAMTS13 ELISA Kits) levels found in acute stroke patients. This difference was even more distinct when the ratio of VWF:ADAMTS13 was considered. These results demonstrate the potentially important involvement of the VWF/ADAMTS13 (zeige ADAMTS13 ELISA Kits) axis in ischemic stroke.
alterations in glycosylation of vWF and other adhesion proteins associated with the targeting of the alpha1,3-Gal (zeige GAL ELISA Kits)-epitope in mutant swine may have salutatory effects on the primate platelet activation observed in these xenografts.
Hemodynamic activation of vWF and increased plasma ADAMTS-13 (zeige ADAMTS13 ELISA Kits) may have contributed to reduced high-molecular-weight vWF multimers and impairment of the vWF-platelet aggregation pathway during mechanical circulatory support.
both the gpIb-VWF interaction and the integrin alpha(2 (zeige ITGA2 ELISA Kits))beta(1)-collagen interaction contribute to platelet adhesion under high shear stress; integrin alpha(II (zeige GSTA3 ELISA Kits))beta(1) makes a greater contribution to adhesion to type I collagen because less VWF is bound
BLOC-2 (zeige HPS6 ELISA Kits) subunit HPS6 (zeige HPS6 ELISA Kits) deficiency affects the tubulation and secretion of von Willebrand factor from mouse endothelial cells
A novel single-domain antibody against von Willebrand factor A1 domain that interferes with VWF-platelet interactions in vivo. By using this sdAb, show that the A1 domain is pertinent to the participation of VWF in the inflammatory response.
These experiments delineate an unexpected pathway in which microbiota-triggered TLR2 (zeige TLR2 ELISA Kits) signaling alters the synthesis of proadhesive VWF by the liver endothelium and favors platelet integrin-dependent thrombus growth.
ADAMTS13 (zeige ADAMTS13 ELISA Kits) controls vascular remodeling by modifying VWF reactivity during stroke recovery.
these novel findings support the hypothesis that conformation of the VWF A domains plays a critical role in modulating macrophage-mediated clearance of VWF in vivo.
results revealed localized vascular expression of FVIII (zeige F8 ELISA Kits) and von Willebrand factor and identified lymphatic endothelial cell as a major cellular source of FVIII (zeige F8 ELISA Kits) in extrahepatic tissues.
Endothelial cell derivedVWF is the major determinant that mediates VWF-dependent ischemic stroke by promoting postischemic thrombo-inflammation.
VWF deficiency reduces the progression of liver fibrosis, suggesting a mechanistic role of elevated plasma VWF levels in cirrhosis
von Willebrand factor exerts beneficial effects in a mouse sepsis model via recruitment of neutrophils to inflammatory sites.
Staphylococcus lugdunensis binds directly to von Willebrand factor, which proved to be vital for withstanding shear forces and for its adhesion to the vessel wall and cardiac valves.
The glycoprotein encoded by this gene functions as both an antihemophilic factor carrier and a platelet-vessel wall mediator in the blood coagulation system. It is crucial to the hemostasis process. Mutations in this gene or deficiencies in this protein result in von Willebrand's disease. An unprocessed pseudogene has been found on chromosome 22.
von Willebrand factor
, coagulation factor VIII VWF