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The protein encoded by TRIM33 is thought to be a transcriptional corepressor. Zusätzlich bieten wir Ihnen TRIM33 Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
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Human Monoclonal TRIM33 Primary Antibody für ICC, IF - ABIN261638
Tirard, Hsiao, Nikolov, Urlaub, Melchior, Brose: In vivo localization and identification of SUMOylated proteins in the brain of His6-HA-SUMO1 knock-in mice. in Proceedings of the National Academy of Sciences of the United States of America 2012
Human Monoclonal TRIM33 Primary Antibody für IF, IHC (p) - ABIN565640
Ding, Jin, Wang, Chen, Wu, Ai, Chen, Zhang, Liang, Laurence, Zhang, Datta, Zhang, Chen: Reduced expression of transcriptional intermediary factor 1 gamma promotes metastasis and indicates poor prognosis of hepatocellular carcinoma. in Hepatology (Baltimore, Md.) 2014
suggest that SnoN (zeige SKIL Antikörper) suppresses TGF-betainduced epithelial-mesenchymal transition and invasion of bladder cancer cells in a TIF1gammadependent manner
The adenovirus E4-ORF3 protein functions as a SUMO E3 ligase for TIF-1gamma sumoylation and poly-SUMO chain elongation.
our findings reveal a new mechanism by which SOX2 (zeige SOX2 Antikörper)-mediated transcription repression of TIF1gamma promotes TGF-beta (zeige TGFB1 Antikörper)-induced epithelial-mesenchymal transition in non-small-cell lung cancer
our work indicates that TIF1gamma exerts its tumor-suppressive functions in part by promoting chromosomal stability.
Results show that TRIM33 is significantly downregulated in clear renal cell carcinoma (zeige MOK Antikörper) tissues which seems to correlate with pathologic stages and grades.
Tumour suppressor TRIM33 targets nuclear beta-catenin (zeige CTNNB1 Antikörper) degradation
Study suggests TRIM33 and NRAS (zeige NRAS Antikörper)-CSDE1 (zeige CDSE1 Antikörper) as candidate genes for autism, and may provide a novel insight into the etiology of autism
The dermatomyositis autoantigen TIF1gamma is markedly up-regulated during muscle regeneration in human and mouse muscle cells.
Data indicate that tripartite motif containing 33 protein TIF1gamma promotes sumoylation of SKI-like proto-oncogene (zeige RAB1A Antikörper) protein SnoN1 and regulates epithelial-mesenchymal transition (EMT (zeige ITK Antikörper)).
The ubiquitination of DHX33 (zeige DHX33 Antikörper) by TRIM33 is lysine 63 specific and is required for the formation of the DHX33 (zeige DHX33 Antikörper)-NLRP3 (zeige NLRP3 Antikörper) inflammasome complex.
Ectodermin is a key switch in the control of TGF-beta (zeige TGFB1 Antikörper) gene responses during early embryonic development and cell proliferation.
TRIM33 is necessary for osteoblast proliferation by regulating cell cycle and for differentiation via bone morphogenetic protein signaling pathway.
these results indicate that Trim33 deficiency leads to the expansion of a subset of myeloid cells characterizing the myelodysplastic/myeloproliferative neoplasm.
Trim33 altered expression of a small group of genes in the testis and the gene with the most significant increase was transcribed from an upstream RLTR10B.
TRIM33 deficiency results in high expression of Ifnb1 (zeige IFNB1 Antikörper) at late stages of macrophages activation.
The findings reveal an essential role for TRIM33 in preventing apoptosis in B lymphoblastic leukemia by interfering with enhancer-mediated Bim (zeige BCL2L11 Antikörper) activation.
This study demonistrated that Smad4 (zeige SMAD4 Antikörper) and Trim33 regulate neural stem cells in the developing cortex in mice.
Epithelium-specific Tak1 (zeige NR2C2 Antikörper):Smad4 (zeige SMAD4 Antikörper)- and Trim33:Smad4 (zeige SMAD4 Antikörper)-double mutants display reduced expression of Mmp13 (zeige MMP13 Antikörper) in palatal medial edge epithelial cells.
These data provide a new paradigm for Tif1gamma in regulating the balance between lymphoid- and myeloid-derived hematopoietic stem cells (HSC (zeige FUT1 Antikörper)) through TGF-beta (zeige TGFB1 Antikörper) signaling, leading to HSC (zeige FUT1 Antikörper) aging.
The authors show that tif1gamma modulates the erythroid versus myeloid fate outcomes from HSCs by differentially controlling the levels of gata1 (zeige GATA1 Antikörper) and pu.1.
TIF1gamma couples the blood-specific transcriptional complex with Pol II elongation machinery to promote the transcription elongation of erythroid genes by counteracting Pol II pausing.
The protein encoded by this gene is thought to be a transcriptional corepressor. However, molecules that interact with this protein have not yet been identified. The protein is a member of the tripartite motif family. This motif includes three zinc-binding domains, a RING, a B-box type 1 and a B-box type 2, and a coiled-coil region. Three alternatively spliced transcript variants for this gene have been described, however, the full-length nature of one variant has not been determined.
tripartite motif-containing 33
, tripartite motif-containing 33 protein
, E3 ubiquitin-protein ligase TRIM33
, e3 ubiquitin-protein ligase TRIM33-like
, tripartite motif containing 33
, RET-fused gene 7 protein
, ectodermin homolog
, protein Rfg7
, transcriptional intermediary factor 1 gamma
, transcription intermediary factor 1-gamma
, tripartite motif-containing protein 33
, tripartite motif protein 33