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TREM1 encodes a receptor belonging to the Ig superfamily that is expressed on myeloid cells. Zusätzlich bieten wir Ihnen TREM1 Antikörper (271) und TREM1 Kits (63) und viele weitere Produktgruppen zu diesem Protein an.
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Human TREM1 Protein expressed in Human Cells - ABIN2002977
Bouchon, Facchetti, Weigand, Colonna: TREM-1 amplifies inflammation and is a crucial mediator of septic shock. in Nature 2001
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Human TREM1 Protein expressed in Wheat germ - ABIN1323572
Li, Birkenheuer, Marr, Levy, Yoder, Nordone: Expression and function of triggering receptor expressed on myeloid cells-1 (TREM-1) on canine neutrophils. in Developmental and comparative immunology 2011
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Human TREM1 Protein expressed in Escherichia coli (E. coli) - ABIN1098432
Kwofie, Rapoport, Fickl, Meyer, Rheeder, Hlope, Anderson, Tintinger et al.: Evaluation of circulating soluble triggering receptor expressed on myeloid cells-1 (sTREM-1) to predict risk profile, response to antimicrobial therapy, and development of complications in patients ... in Annals of hematology 2012
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TREM-1 was found to be significantly higher in tissues biopsies and blood of subjects with obesity. Greater expression and activity of TREM-1 suggest a possible role in the underlying pathophysiology of obesity and associated comorbidities.
Carriage of the TREM1 rs2234237T allele appears to be a risk factor for the development of severe malaria.
TREM-1, HMGB1 (zeige HMGB1 Proteine) and RAGE (zeige AGER Proteine) in the Shoulder Tendon: Dual Mechanisms for Inflammation Based on the Coincidence of Glenohumeral Arthritis
is concluded that both HMGB1 (zeige HMGB1 Proteine) and sTREM-1 are released during the inflammatory response of periodontal tissues and they can promote inflammatory process, which leads to tissue destruction.
The soluble TREM-1 level, core temperature and SAPS (zeige SKAP2 Proteine) II value were the only independent predictors of death after adjustment for potential confounders in sepsis patients admitted to intensive care.
TREM-1 plays a critical role in osteoarthritis development and IL1B (zeige IL1B Proteine) induced chondrocyte injury through regulation of NF-kappaB (zeige NFKB1 Proteine) signaling
this study shows higher levels of TREM-1 in plasma of systemic lupus erythematosus in Egyptian patients
Increased expression of TREM-1 in S compared to AS patients involving TNFa (zeige TNF Proteine), MMP-1 (zeige MMP1 Proteine) and MMP-9 (zeige MMP9 Proteine) suggest a potential role of TREM-1 in plaque destabilization
Plasma levels of sTREM-1 were found to identify patients with septic shock more effectively than procalcitonin and C-reactive protein (zeige CRP Proteine). Moreover, sTREM-1 was identified to be an early predictor for survival in patients with septic shock.
This study provides new insights into the possible mechanism of TREM-1 and HIF-1alpha (zeige HIF1A Proteine) in psoriasis.
This study is the first to show the regulatory role of TREM-1 in RLRs and TLRs expression.
TREM-1 is upregulated in renal inflammation and plays a vital role in driving disease. Thus, TREM-1 blockade emerges as a potential therapeutic avenue for immune-mediated renal diseases such as lupus nephritis
TGF-beta1 (zeige TGFB1 Proteine) increased the expression of TREM-1 in mouse macrophages partly via the transcription factor AP-1 (zeige JUN Proteine).
in macrophages, oxidized low-density lipoprotein enhanced expression of TREM-1, which amplifies the innate immune response of Toll (zeige TLR4 Proteine)-like receptor pathway; activation of TREM-1 contributes to atherogenesis process by enhancing proinflammatory cytokine production and foam cell formation.
TREM-1 and Dectin-1 (zeige CLEC7A Proteine) function concurrently in the corneal innate immune response by regulating inflammatory cytokine expression in fungal keratitis.
a novel positive feedback cycle mediated by TREM-1 and GM-CSF (zeige CSF2 Proteine) was revealed to enhance macrophage polarization under unilateral ureteral obstruction.
TREM-1-mediated innate immune response played an essential role in the activation of neutrophils and Streptococcus suis clearance.
Data indicate that triggering receptor expressed on myeloid cells-1(TREM-1) inhibition improves heart function after myocardial infarction (MI).
TREM-1 is significantly associated with LPS (zeige TLR4 Proteine)-induced left ventricular systolic dysfunction in BALB/c mice
TREM-1 deficiency can attenuate disease severity without affecting pathogen clearance.
Molecular cloning of bovine TREM-1 extends the repertoire of bovine pattern recognition receptors and provides information important for investigating their role in bovine innate immunity.
This gene encodes a receptor belonging to the Ig superfamily that is expressed on myeloid cells. This protein amplifies neutrophil and monocyte-mediated inflammatory responses triggered by bacterial and fungal infections by stimulating release of pro-inflammatory chemokines and cytokines, as well as increased surface expression of cell activation markers. Alternatively spliced transcript variants encoding different isoforms have been noted for this gene.
triggering receptor expressed on myeloid cells 1
, triggering receptor expressed on monocytes 1
, triggering-receptor TREM1
, triggering receptor expressed in monocytes 1
, triggering receptor expressed on myeloid cells-1