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TIMP3 belongs to the TIMP gene family. Zusätzlich bieten wir Ihnen TIMP3 Antikörper (202) und TIMP3 Proteine (17) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 106 products:
Human TIMP3 ELISA Kit für Sandwich ELISA - ABIN2685673
Tsarouhas, Soufla, Apostolakis, Zaravinos, Panagiotou, Khoury, Hassoulas, Tsatsakis, Spandidos: Transcriptional regulation of TIMPs in ascending aorta aneurysms. in Thrombosis research 2010
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Human TIMP3 ELISA Kit für Sandwich ELISA - ABIN414926
Li, Liang, Tao, Zhou, Li, Chen, Chen: Acidic pH conditions mimicking degenerative intervertebral discs impair the survival and biological behavior of human adipose-derived mesenchymal stem cells. in Experimental biology and medicine (Maywood, N.J.) 2012
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Rat (Rattus) TIMP3 ELISA Kit für Sandwich ELISA - ABIN416332
Kna?, Niczyporuk, Zalewska, Car: The unwounded skin remodeling in animal models of diabetes types 1 and 2. in Physiological research / Academia Scientiarum Bohemoslovaca 2013
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Mouse (Murine) TIMP3 ELISA Kit für Sandwich ELISA - ABIN415606
Shukla, Kumar Shakya, Dhole, Misra: Upregulated expression of matrix metalloproteinases and tissue inhibitors of matrix metalloproteinases in BALB/c mouse brain challenged with Japanese encephalitis virus. in Neuroimmunomodulation 2012
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These results implicate TIMP3 as a modulator of cell surface GHR (zeige GHR ELISA Kits) abundance and the ability of GH to promote cellular signaling.
native glycosaminoglycans interact with TIMP-3.
The expression level of LIPC (zeige LIPC ELISA Kits), SLC16A8 (zeige MCT3 ELISA Kits), and TIMP-3 was significantly associated with age-related macular degeneration pathology.
Levels of miR (zeige MLXIP ELISA Kits)-221/222 are associated negatively with estrogen receptor (zeige ESR1 ELISA Kits) in in situ tumors and positively with tissue inhibitor of metalloproteinase 3 TIMP3 messenger RNA expression levels in pure invasive breast cancers.
Electrostatic potential calculations suggested a competition between negatively charged GAGs and highly negatively charged complement-like domains of LRP-1 (zeige LRP1 ELISA Kits) for the binding to a positively charged area of TIMP-3 as an underlying mechanism.
TIMP3 overexpression after myocardial infarction improves myocardial structural remodeling and function by promoting angiogenesis and inhibiting early proteolysis.
Single Nucleotide Variants of Candidate Genes in Aggrecan (zeige ACAN ELISA Kits) Metabolic Pathway Are Associated with Lumbar Disc Degeneration and Modic Changes
Our data suggest that miR (zeige MLXIP ELISA Kits)-206 may function as an inflammatory regulator and drive the expression of MMP9 (zeige MMP9 ELISA Kits) in M.tb-infected THP-1 (zeige GLI2 ELISA Kits) cells by targeting TIMP3, indicating that miR (zeige MLXIP ELISA Kits)-206 is a potential therapeutic target for patients with TB.
Plasma TIMP3 is a biomarker for predicting the tumor stage in patients with oral squamous cell carcinoma .
TIMP-3 K26A/K45A retained higher affinity for sulfated (zeige SULF1 ELISA Kits) glycosaminoglycans than K42A/K110A and exhibited increased affinity for ADAMTS-5 (zeige ADAMTS5 ELISA Kits) in the presence of heparin.
Circulating smoke components, including acrolein, contribute to vascular diseases through enhanced MMP-1 (zeige MMP1 ELISA Kits) and decreased TIMP-3 secretion.
TIMP-3 is downregulated in a distinct subpopulation of atherosclerotic foam cells which have increased MMP-14 (zeige MMP14 ELISA Kits).
Reactive oxygen species mediate TGF-beta1 (zeige TGFB1 ELISA Kits)-induced TIMP-3 gene expression
TIMP3 has a role in the pericyte-induced stabilization of newly formed vascular networks that are predisposed to undergo regression and reveal specific molecular targets of the inhibitors regulating these events.
TIMP3 mRNA expression level was upregulated by multidirectional articular motion.
only the N-terminal, but not the C-terminal domain of TIMP-3, results in developmental defects.
metamorphic tail and intestine RNA levels of TIMP-2 (zeige TIMP2 ELISA Kits), MT1-MMP (zeige MMP14 ELISA Kits) and Gel-A, but not MT3-MMP (zeige MMP24 ELISA Kits) or TIMP-3, are elevated during periods of cell death and proliferation
In a clinically relevant CADASIL (zeige NOTCH3 ELISA Kits) mouse model, we show that exogenous ADAM17 (zeige ADAM17 ELISA Kits) or HB-EGF (zeige HBEGF ELISA Kits) restores cerebral arterial tone and blood flow responses, and identify upregulated voltage-dependent potassium channel (zeige KCNAB2 ELISA Kits) (KV) number in cerebral arterial myocytes as a heretofore-unrecognized downstream effector of TIMP3-induced deficits.
TIMP3 promotes normal microvascular endothelial cell barrier function, at least partially, through inhibition of metalloproteinase-dependent disruption of adherens junctions, and septic downregulation of TIMP3 may contribute to septic MVEC barrier dysfunction.
data strongly suggest that TIMP3 has direct neuroprotective effects that can mitigate the deleterious effects associated with TBI, an area with few if any therapeutic options.
Elevated levels of TIMP3 and vitronectin (zeige VTN ELISA Kits), acting downstream of Notch3 (zeige NOTCH3 ELISA Kits)(ECD) deposition, play a role in CADASIL (zeige NOTCH3 ELISA Kits), producing divergent influences on early CBF (zeige CEBPZ ELISA Kits) deficits and later white matter lesions.
4-Hydroxyisoleucine improved insulin (zeige INS ELISA Kits) resistant-like state in 3T3-L1 adipocytes by targeting TACE (zeige ADAM17 ELISA Kits)/TIMP3 and the insulin (zeige INS ELISA Kits) signaling pathway.
In a mouse model of prostate cancer, increased tumor growth, proliferation index, increased microvascular density, and invasion was observed in Pten(-/-), Timp3(-/-) prostate tumors compared to Pten(-/-), Timp3(+/+) tumors.
Timp3 status determines p53 (zeige TP53 ELISA Kits), p38 (zeige CRK ELISA Kits) and Notch (zeige NOTCH1 ELISA Kits) coactivation to instruct hepatic cell fate and transformation.
TIMP2 (zeige TIMP2 ELISA Kits) and TIMP3 play fundamental and differential roles in mediating pathological remodelling, independent from their MMP-inhibitory function
Expansion of stem cells counteracts age-related mammary regression in compound Timp1 (zeige TIMP1 ELISA Kits)/Timp3-deficient mice.
lack of TIMP3 increases inflammation and polarizes macrophages towards a more inflammatory phenotype resulting in increased atherosclerosis.
These results suggest the crucial role of TIMP-3 in successful implantation and embryo survival and indicate the endometrial stromal decidualization-like in pigs.
This gene belongs to the TIMP gene family. The proteins encoded by this gene family are inhibitors of the matrix metalloproteinases, a group of peptidases involved in degradation of the extracellular matrix (ECM). Expression of this gene is induced in response to mitogenic stimulation and this netrin domain-containing protein is localized to the ECM. Mutations in this gene have been associated with the autosomal dominant disorder Sorsby's fundus dystrophy.
metalloproteinase inhibitor 3
, TIMP metallopeptidase inhibitor 3 (Sorsby fundus dystrophy, pseudoinflammatory)
, tissue inhibitor metalloproteinase-3
, TIMP metallopeptidase inhibitor 3
, Metalloproteinase inhibitor 3
, MIG-5 protein
, protein MIG-5
, tissue inhibitor of metalloproteinases 3
, tissue inhibitor of metalloproteinase-3
, tissue inhibitor of metalloproteinase 3 (Sorsby fundus dystrophy, pseudoinflammatory)
, tissue inhibitor of metalloproteinase 3
, 21 kDa protein of extracellular matrix
, tissue inhibitor of metalloproteinases-3