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The protein encoded by S100A8 is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. Zusätzlich bieten wir Ihnen S100A8 Kits (44) und S100A8 Proteine (42) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 212 products:
Human Monoclonal S100A8 Primary Antibody für EIA, IHC (fro) - ABIN111890
Brandtzaeg, Jones, Flavell, Fagerhol: Mac 387 antibody and detection of formalin resistant myelomonocytic L1 antigen. in Journal of clinical pathology 1989
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Human Monoclonal S100A8 Primary Antibody für EIA, IHC (fro) - ABIN111891
Odink, Cerletti, Brüggen, Clerc, Tarcsay, Zwadlo, Gerhards, Schlegel, Sorg: Two calcium-binding proteins in infiltrate macrophages of rheumatoid arthritis. in Nature 1987
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Human Monoclonal S100A8 Primary Antibody für DB, EIA - ABIN111892
Delabie, de Wolf-Peeters, van den Oord, Desmet: Differential expression of the calcium-binding proteins MRP8 and MRP14 in granulomatous conditions: an immunohistochemical study. in Clinical and experimental immunology 1990
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Human Monoclonal S100A8 Primary Antibody für EIA, FACS - ABIN111893
Hogg, Allen, Edgeworth: Monoclonal antibody 5.5 reacts with p8,14, a myeloid molecule associated with some vascular endothelium. in European journal of immunology 1989
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Human Monoclonal S100A8 Primary Antibody für ELISA, WB - ABIN393635
Schenten, Bréchard, Plançon, Melchior, Frippiat, Tschirhart: iPLA2, a novel determinant in Ca2+- and phosphorylation-dependent S100A8/A9 regulated NOX2 activity. in Biochimica et biophysica acta 2010
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Human Monoclonal S100A8 Primary Antibody für FACS, IF - ABIN954652
Klein, Pino, Johnson, Davies, Nagpal, Thacher, Krasinski, Chandraratna: Identification and functional separation of retinoic acid receptor neutral antagonists and inverse agonists. in The Journal of biological chemistry 1996
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Human Monoclonal S100A8 Primary Antibody für FACS, IF - ABIN954653
Nagpal, Thacher, Patel, Friant, Malhotra, Shafer, Krasinski, Asano, Teng, Duvic, Chandraratna: Negative regulation of two hyperproliferative keratinocyte differentiation markers by a retinoic acid receptor-specific retinoid: insight into the mechanism of retinoid action in psoriasis. in Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1997
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Human Monoclonal S100A8 Primary Antibody für FACS, IF - ABIN954654
Thacher, Nagpal, Klein, Arefieg, Krasinski, DiSepio, Agarwal, Johnson, Eckert, Chandraratna: Cell type and gene-specific activity of the retinoid inverse agonist AGN 193109: divergent effects from agonist at retinoic acid receptor gamma in human keratinocytes. in Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1999
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Human Monoclonal S100A8 Primary Antibody für EIA, WB - ABIN1108923
Amano, Kanda, Shibayama, Kamei, Matsuda: Model generation interface for simulation of left ventricular motion. in Conference proceedings : ... Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Conference 2007
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S100A4 (zeige S100A4 Antikörper), A8, and A9 proteins represent promising marker genes to evaluate the risk potential of suspicious oral lesions in molecular pathology.
Low S100A8/A9 levels may contribute to poor wound healing in colonized chronic wounds with striking difference between diabetic foot ulcers and venous leg ulcers.
Suggest that, in individuals with lichen planus, S100A8 may exert distinct immunomodulatory and cytotoxicity functions.
Serum calprotectin was significantly higher in rheumatoid arthritis patients than controls, and was correlated with disease activity. It was higher in seropositive than in seronegative patients.
S100A8, S100A9 (zeige S100A9 Antikörper) and S100A8/A9 protein levels were higher in chronic rhinosinusitis (CRS (zeige CARS Antikörper)) with nasal polyps (NP) patients, showing increased deposition on extracellular matrix (ECM (zeige MMRN1 Antikörper)) structures in contrast to CRS (zeige CARS Antikörper) without NP and controls.
MRP8/14 is a good marker of disease activity in rheumatoid arthritis, and higher levels predict response to methotrexate.
Blood expression of matrix metalloproteinases 8 and 9 and of their inducers S100A8 and S100A9 (zeige S100A9 Antikörper) supports diagnosis and prognosis of PDAC-associated diabetes mellitus
Increased MRP8/14 levels suggest the instability of the atherosclerotic plaque. ECP (zeige ECP Antikörper) reflects the severity of coronary arteries stenosis, predicting atherosclerosis burden. They may become the new biomarkers of CAD (zeige CAD Antikörper).
Findings suggest that S100 proteins S100A12 (zeige S100A12 Antikörper) and S100A8/A9 are secreted independently either via distinct mechanisms of secretion or following the activation of different signal transduction pathways.
Data suggest up-regulation of pro-inflammatory molecules TLR9 (zeige TLR9 Antikörper), S100A8, and IL-8 (zeige IL8 Antikörper) plus lower levels of CD11b (zeige ITGAM Antikörper)+ CD33 (zeige CD33 Antikörper)+ myeloid cells may be involved in impairment of wound healing in type 2 diabetes leading to chronic ulcers, such as diabetic foot ulcers.
Study verified porcine calprotectin (S100A8/A9) expression at the protein level in multiple Haemophilus parasuis infected tissues and explored their molecular characterization.
Data suggest that up-regulation of S100A8 and S100A9 (zeige S100A9 Antikörper) is a key component of early endometrial response to uterine involution in the post-partum period and to prevent chronic endometritis/uterine inflammation; up-regulation can be influenced by diet.
S100a8 upregulation triggers NF-kappaB (zeige NFKB1 Antikörper) signal pathway through RAGE (zeige AGER Antikörper) and TLR4 (zeige TLR4 Antikörper), in response to laser-induced dermis wound healing.
Although MRP-8/-14 expression is highly increased in experimental, these proteins do not contribute to the pathogenesis in the effector phase of epidermolysis bullosa acquisita and bullous pemphigoid (zeige DST Antikörper).
TLR4 (zeige TLR4 Antikörper), TLR2 (zeige TLR2 Antikörper) also contributed to Mrp8-induced inflammatory response and tolerance.
S100A8 appears to play a crucial role in the activation of the TLR4 (zeige TLR4 Antikörper)/MD-2 (zeige LY96 Antikörper) pathway and the promotion of a tumor growth-enhancing immune microenvironment.
Rps14 (zeige RPS14 Antikörper) haploinsufficiency in del(5q) myelodysplastic syndrome is linked to activation of the innate immune system and induction of S100A8-S100A9 (zeige S100A9 Antikörper) expression, leading to a p53 (zeige TP53 Antikörper)-dependent erythroid differentiation defect.
S100A8 is primarily produced from CXCR2 (zeige CXCR2 Antikörper)-expressing CD11b (zeige ITGAM Antikörper)(+)Gr-1 (zeige GSR Antikörper)(high) cells, and it upregulates TNF-alpha (zeige TNF Antikörper) production in CD11b (zeige ITGAM Antikörper)(+)F4/80(+) cells through cellular cross-talk, which is an important mechanism in the development of Nonalcoholic fatty liver disease
Alarmins S100A8/S100A9 (zeige S100A9 Antikörper) aggravate osteophyte formation in experimental osteoarthritis and predict osteophyte progression in early human symptomatic osteoarthritis.
This study identified Mrp8/14 and TLR4 (zeige TLR4 Antikörper) as important modulators of the leukocyte recruitment cascade during inflammation via integrin beta2.
Inflammation-induced S100A8 promotes colorectal tumorigenesis by acting upstream to activate the Akt1 (zeige AKT1 Antikörper)-Smad5 (zeige SMAD5 Antikörper)-Id3 (zeige ID3 Antikörper) axis.
Data shows that S100A8/A9 plays a critical role during glomerulonephritis, exerting and amplifying autocrine and paracrine proinflammatory effects on bone marrow-derived macrophages, renal endothelial cells, and mesangial cells.
The protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21. This protein may function in the inhibition of casein kinase and as a cytokine. Altered expression of this protein is associated with the disease cystic fibrosis.
S100 calcium binding protein A8
, S100 calcium-binding protein A8 (calgranulin A)
, calgranulin A
, calprotectin L1L subunit
, cystic fibrosis antigen
, leukocyte L1 complex light chain
, migration inhibitory factor-related protein 8
, protein S100-A8
, urinary stone protein band A
, S100 calcium binding protein A8 (calgranulin A)
, S100 calcium-binding protein A8
, neutrophil cytosolic 7 kDa protein
, chemotactic S100 protein
, chemotactic cytokine CP-10
, pro-inflammatory S100 cytokine