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Targets myosin phosphatase to the actin cytoskeleton. Zusätzlich bieten wir Ihnen und viele weitere Produktgruppen zu diesem Protein an.
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Mouse (Murine) Polyclonal MPRIP Primary Antibody für IHC, ELISA - ABIN1031778
McQuade, Cho, Chan: Positive and Negative Phosphorylation Regulates RIP1 and RIP3-Induced Programmed Necrosis. in The Biochemical journal 2013
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Human Polyclonal MPRIP Primary Antibody für ICC, IF - ABIN4335376
Bracken, Li, Wright, Lawrence, Pillman, Salmanidis, Anderson, Dredge, Gregory, Tsykin, Neilsen, Thomson, Bert, Leerberg, Yap, Jensen, Khew-Goodall, Goodall: Genome-wide identification of miR-200 targets reveals a regulatory network controlling cell invasion. in The EMBO journal 2014
Mouse (Murine) Polyclonal MPRIP Primary Antibody für IHC (p), WB - ABIN2476368
Mills, Thomas, Lynch, Katcher: Effect of pulse palpation on cardiac arrhythmia in coronary care patients. in Nursing research 1976
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Human Polyclonal MPRIP Primary Antibody für IF (p), IHC (p) - ABIN872987
Seifert, Werba, Tiwari, Giao Ly, Alothman, Alqunaibit, Avanzi, Barilla, Daley, Greco, Torres-Hernandez, Pergamo, Ochi, Zambirinis, Pansari, Rendon, Tippens, Hundeyin, Mani, Hajdu, Engle, Miller: The necrosome promotes pancreatic oncogenesis via CXCL1 and Mincle-induced immune suppression. in Nature 2016
The B-Raf (zeige SNRPE Antikörper)(V600E) inhibitor dabrafenib selectively inhibits RIP3 (zeige RIPK3 Antikörper) and alleviates acetaminophen-induced liver injury.
Our data identify M-RIP as a crucial local regulator of the Rho-Rac (zeige AKT1 Antikörper) balance during chemotaxis and antigen recognition.
MLCP (myosin light chain phosphatase) activation is counteracted by a previously unrecognized association between MRIP and the inducible kinase NUAK2 (zeige NUAK2 Antikörper)
p116Rip is an important regulatory component that controls the RhoA signaling pathway, thus regulating MLCP activity and myosin phosphorylation in cells
M-RIP-dependent targeting of myosin phosphatase to stress fibers has an important role in regulating myosin light chain phosphorylation state and morphology in human vascular smooth muscle cells
A functional analysis of JNK1 (zeige MAPK8 Antikörper) and M-RIP with RNA interference reveals a critical role for this cascade in the invasive behavior of cancer cells.
RIP3-mediated necroptosis is a major mechanism of enhanced inflammation and lung tissue injury in high dose LPS (zeige TLR4 Antikörper)- induced severe ARDS in mice
RIP3 deletion suppresses inflammation response.
Results suggest that RGC-5 cell necroptosis following oxygen glucose deprivation is mediated by a RIP3-induced increase in oxidative stress
Lymphocyte infiltrations in the adipocyte tissue and in skin lesions of ApoE (zeige APOE Antikörper) single-knockout mice were significantly mitigated in ApoE (zeige APOE Antikörper)/RIP3 double-knockout mice
RIP3-dependent necroptosis modulates post-ischaemic adverse remodelling in a mouse model of myocardial infarction
The B-Raf (zeige SNRPE Antikörper)(V600E) inhibitor dabrafenib selectively inhibits RIP3 and alleviates acetaminophen-induced liver injury.
By comparing the phosphorylation sites in wild-type and RIP3-knockdown L929 cells, 174, 167, and 177 distinct phosphorylation sites were revealed to be dependent on RIP3 at the 0.5, 2, and 4 h time points after TNF (zeige TNF Antikörper) treatment, respectively.
p116Rip is an F-actin-binding protein (zeige NEXN Antikörper) with in vitro bundling activity and in vivo capability of disassembling the actomyosin-based cytoskeleton.
p116Rip oligomerizes via its C-terminal coiled-coil domain and, when overexpressed, inhibits RhoA (zeige RHOA Antikörper)-induced SRF activation without affecting RhoA (zeige RHOA Antikörper)-GTP (zeige AK3 Antikörper) levels.
Embryonic fibroblasts from RIP3 knockout mice are resistant to necrosis and RIP3 knockout animals are devoid of inflammation inflicted tissue damage in an acute pancreatitis model.
Targets myosin phosphatase to the actin cytoskeleton. Required for the regulation of the actin cytoskeleton by RhoA and ROCK1. Depletion leads to an increased number of stress fibers in smooth muscle cells through stabilization of actin fibers by phosphorylated myosin. Overexpression of MRIP as well as its F- actin-binding region leads to disassembly of stress fibers in neuronal cells.
Rho interacting protein 3
, myosin phosphatase Rho-interacting protein
, Rho-interacting protein 3 (p116RIP) (RIP3)
, p116 Rho interacting protein
, myosin phosphatase-Rho interacting protein
, rho-interacting protein 3