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MAOA is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Zusätzlich bieten wir Ihnen Monoamine Oxidase A Kits (44) und Monoamine Oxidase A Proteine (12) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 91 products:
Human Polyclonal Monoamine Oxidase A Primary Antibody für IHC (p), IHC - ABIN446982
Maeda, Morikawa, Takadate, Suzuki, Minowa, Hanagata, Onogawa, Motoi, Nishimura, Unno: Mass spectrometry-based proteomic analysis of formalin-fixed paraffin-embedded extrahepatic cholangiocarcinoma. in Journal of hepato-biliary-pancreatic sciences 2015
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Human Polyclonal Monoamine Oxidase A Primary Antibody für WB - ABIN517731
Xiao, Li, Jones-Brando, Yolken: Abnormalities of neurotransmitter and neuropeptide systems in human neuroepithelioma cells infected by three Toxoplasma strains. in Journal of neural transmission (Vienna, Austria : 1996) 2013
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Human Polyclonal Monoamine Oxidase A Primary Antibody für ELISA, WB - ABIN250814
Domschke, Sheehan, Lowe, Kirley, Mullins, Osullivan, Freitag, Becker, Conroy, Fitzgerald, Gill, Hawi et al.: Association analysis of the monoamine oxidase A and B genes with attention deficit hyperactivity disorder (ADHD) in an Irish sample: preferential transmission of the MAO-A 941G allele to affected ... in American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics 2005
Human Polyclonal Monoamine Oxidase A Primary Antibody für WB - ABIN4890309
Matsumoto, Watanabe, Taguchi, Kobayashi: Mechanisms underlying increased serotonin-induced contraction in carotid arteries from chronic type 2 diabetic Goto-Kakizaki rats. in Pharmacological research 2014
Social behavior was facilitated by chronic fluoxetine treatment in juvenile rhesus monkeys interacting with a familiar peer. The type of social behavior affected depended on the MAOA genotype of the monkey and its partner.
Results provide novel evidence that blood MAOA expression predicts alcohol consumption and that heavy alcohol use is linked to low MAOA expression in both the blood and NAc (zeige NLRP1 Antikörper) core.
study found the ability to consolidate sleep during the dark cycle was disrupted by prenatal iron deprivation, specifically in monkeys with the low-MAOA genotype
The effects of iron deficiency are dependent on MAOA genotype in terms of both direction and size of the effect.
rhMAOA-LPR (zeige FAS Antikörper) allele modifies the effects of maternal temperament on offspring's personality development.
MAOA and MAOB (zeige MAOB Antikörper) variants may contribute to the etiology of Attention deficit hyperactivity disorder (ADHD) in the Indo (zeige IDO1 Antikörper)-Caucasoid population and could be responsible for higher occurrence of ADHD in the boys.
Importance of MAOA and 5-HTTLPR (zeige SLC6A4 Antikörper) polymorphisms in the treatment of spousal violence and drinking in men in batterer intervention programs.
Polymorphisms of COMT (zeige COMT Antikörper) (c.649G>A), MAO-A (c.1460C>T), NET (c.1287G>A) Genes and the Level of Catecholamines, Serotonin in Patients with Parkinson's Disease
The influence of MAOA failed to reach statistical significance within any of the regression equations in predicting adult criminal behavior.
MAOA provides tumor cell growth advantages in the bone microenvironment by stimulating interleukin-6 (IL6 (zeige IL6 Antikörper)) release from osteoblasts, and triggers skeletal colonization by activating osteoclastogenesis through osteoblast production of RANKL (zeige TNFSF11 Antikörper) and IL6 (zeige IL6 Antikörper).
results highlight an association of ventral striatum MAO-A level with the functional connectivity of striatal regions linked to impulsive behavior in antisocial personality disorder
No significant association was found between severe agitation and MAOA uVNTR or MAOB (zeige MAOB Antikörper) rs1799836 polymorphism, revealing that these individual polymorphisms in MAO (zeige MAO Antikörper) genes are not related to severe agitation in subjects with schizophrenia and conduct disorder
The effects of parenting on self-control and offending are most pronounced for those who carry plasticity alleles for both MAOA and DAT1. Thus, MAOA and DAT1 may be implicated in offending because they increase the negative effects of parenting on self-control. Implications for theory are discussed.
These data suggest a close association between MAO-A-dependent reactive oxygen species generation, actin oxidation, and ventricular dysfunction.
MAOA hypomethylation is a panic disorder risk marker.
cloning and characterization of monoamine oxidase A and B genes in pig
Two pools of ten bovine embryos, comprised of the 4-, 8- to 16-cell, morula, blastocyst, and expanded blastocyst stages, were collected. Total RNA was isolated, and the RT-PCR-RFLP technique was used to observe expression of the MAOA gene.
MAOA was seriously demethylated or showed aberrant methylation patterns in four aborted clones.
This study unravels a new link between MAO (zeige MAO Antikörper)-dependent H2O2 production and lysosomal dysfunction. Altogether, our findings demonstrate that the MAO-A/H2O2 axis has a negative impact on the elimination and recycling of mitochondria through the autophagy-lysosome pathway, which participates in cardiomyocyte death and heart failure
Results demonstrate that C17H11IOSe, a selective inhibitor of cortical MAO-A, elicited an antidepressant-like action in mice by interacting with the serotonergic system
data lead us to conclude that elevation of AP-1 (zeige JUN Antikörper) or NF-kB indirectly decreases MAO-A protein levels which, in turn, diminishes MAO-A ability in the VTA of the mesolimbic dopaminergic pathway
Huntington disease (zeige HTT Antikörper) neural cells exhibit increased Monoamine oxidase-A and Monoamine oxidases-B expression and activity
acute stress induces anxiety-like responses by affecting rapid dendritic remodeling in the pyramidal cells of orbitofrontal cortex and basolateral amygdala; furthermore, our data show that MAO-A and monoamine metabolism are required for these phenomena.
These findings demonstrate that regulation of monoamine levels by Mao (zeige MAO Antikörper) activity in beta cells is pivotal for physiological insulin (zeige INS Antikörper) secretion and that loss of MaoB (zeige MAOB Antikörper) expression may contribute to the beta cell dysfunction in type 2 diabetes.
Results suggest a role for KLF11 (zeige KLF11 Antikörper) in upregulating MAO-A in depressive disorder and chronic social stress, suggesting that inhibition of the pathways regulated by this transcription factor may aid in the therapeutics of neuropsychiatric illnesses
The results of this study suggest that heightened dACC and amygdala activation and their connectivity are neuroaffective mechanisms underlying anger control in participants with the low-functioning allele of the MAOA gene.
These results suggest that early developmental enhancements in 5-HT (zeige DDC Antikörper) levels have long-term effects on the modulation of behavioral flexibility associated with MAO-A deficiency.
Under conditions of chronic hemodynamic stress, enhanced MAO-B (zeige MAOB Antikörper) activity is a major determinant of cardiac structural and functional disarrangement.
This gene is one of two neighboring gene family members that encode mitochondrial enzymes which catalyze the oxidative deamination of amines, such as dopamine, norepinephrine, and serotonin. Mutation of this gene results in Brunner syndrome. This gene has also been associated with a variety of other psychiatric disorders, including antisocial behavior. Alternatively spliced transcript variants encoding multiple isoforms have been observed.
monoamine oxidase A
, amine oxidase [flavin-containing] A-like
, amine oxidase [flavin-containing] A
, monoamine oxidase type A
, putative monoamine oxidase A