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Kruppel-like factors (KLFs) are a family of broadly expressed zinc finger transcription factors. Zusätzlich bieten wir Ihnen KLF2 Antikörper (111) und KLF2 Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
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KLF2 and KLF4 (zeige KLF4 ELISA Kits) serve as important regulators that promote hemoglobin alpha (zeige HBA1 ELISA Kits) expression in the endothelium.
Upregulated long non-coding RNA AGAP2 (zeige AGAP2 ELISA Kits)-AS1 (zeige PTGDR ELISA Kits) represses LATS2 and KLF2 expression through interacting with EZH2 (zeige EZH2 ELISA Kits) and LSD1 (zeige KDM1A ELISA Kits) in non-small-cell lung cancer cells.
Transcription factor Kruppel-like factor 2 (KLF2) of the zinc finger family of DNA-binding proteins has recently emerged as a major molecular switch that controls endothelial homeostasis.
The findings point to KLF2 as a new gene involved in heritable pulmonary arterial hypertension.
we provide evidence that the tumor-suppressive microRNA miR (zeige MLXIP ELISA Kits)-1825 controls KLF2 expression. Reporter gene analyses revealed that both microRNAs directly targeted the 3'-untranslated region of KLF2 messenger RNA. These data demonstrated that miR (zeige MLXIP ELISA Kits)-1825 expression in serum of human glioma was associated with tumorigenesis and miR (zeige MLXIP ELISA Kits)-1825 may be used as a biomarker for identification of the pathological grade of glioma.
Long noncoding RNA XIST acts as an oncogene (zeige RAB1A ELISA Kits) in non-small cell lung cancer by epigenetically repressing KLF2 expression.
lysine-specific demethylase (zeige MBD2 ELISA Kits)-1 is an important oncogene (zeige RAB1A ELISA Kits) in gastric cancer, and lysine-specific demethylase (zeige MBD2 ELISA Kits)-1-mediated epigenetic repression of KLF2 plays a critical role in gastric cancer development and progression.
KLF2 is shown to be downregulated in pancreatic ductal adenocarcinoma (PDAC) clinical samples and overexpression of KLF2 inhibits the growth, migration, and metastasis of PDAC cancer cells.
KLF2 expression was decreased in gastric cancer and negatively correlated with lymphatic metastasis. KLF2 overexpression inhibited cell proliferation and invasive potential and downregulated the protein expression of PCNA (zeige PCNA ELISA Kits), Bcl-2 (zeige BCL2 ELISA Kits) and MMP-9 (zeige MMP9 ELISA Kits) in GC cells. KLF2 overexpression reduced xenograft tumor growth.
our data highlight an important role for KLF2/c-myc (zeige MYC ELISA Kits) pathway in HCC (zeige FAM126A ELISA Kits) development and progression.
miR (zeige MYLIP ELISA Kits)-92a coregulates KLF4 (zeige KLF4 ELISA Kits) and KLF2 expression in arterial endothelium and contributes to phenotype heterogeneity associated with regional atherosusceptibility and protection in vivo.
KLF2 and KLF4 (zeige KLF4 ELISA Kits) serve as important regulators that promote hemoglobin alpha (zeige HBA-A1 ELISA Kits) expression in the endothelium.
Mouse embryos deficient in Orai1 (zeige TMEM132A ELISA Kits), Klf2, or Klf4 (zeige KLF4 ELISA Kits) showed a significantly reduced lymphatic density and impaired lymphatic development.
this study shows that KLF2 governs the degree of dendritic cells activation and hence the intensity of proatherogenic T cell responses
A deficiency of KLF2 increased the expression of Notch ligand (zeige JAG2 ELISA Kits) Jagged2 (zeige JAG2 ELISA Kits) via hypoxia-inducible factor 1alpha (HIF-1alpha (zeige HIF1A ELISA Kits)), which led to Th2 amplification.
our data demonstrate a protective role of KLF2 against glomerular endothelial cell injury and progression of chronic kidney disease in the model of compensatory renal hypertrophy
Under steady-state conditions, KLF2-deficient NK cells alter their expression of homeostatic homing receptors and subsequently undergo apoptosis due to IL-15 (zeige IL15 ELISA Kits) starvation.
KLF2 knockdown markedly reduced expression of miR (zeige MLXIP ELISA Kits)-155 in macrophages and macrophage activation.
study therefore establishes the pivotal role of Klf2 and Tfcp2l1 in mediating ESC self-renewal promoted by Wnt (zeige WNT2 ELISA Kits)/beta-catenin (zeige CTNNB1 ELISA Kits) signaling.
endothelial-specific loss of Mekk3 (zeige MAP3K3 ELISA Kits), Klf2 or Klf4 (zeige KLF4 ELISA Kits) markedly prevents cerebral cavernous malformation lesion formation, reverses the increase in Rho activity, and rescues lethality
KLF2 and ERG (zeige ERG ELISA Kits) associate in a physical complex and the two proteins synergistically activate transcription of Flk1 (zeige KDR ELISA Kits).
Kruppel-like factors (KLFs) are a family of broadly expressed zinc finger transcription factors. KLF2 regulates T-cell trafficking by promoting expression of the lipid-binding receptor S1P1 (S1PR1\; MIM 601974) and the selectin CD62L (SELL\; MIM 153240) (summary by Weinreich et al., 2009
Kruppel-like factor 2 (lung)
, Krueppel-like factor 2
, Kruppel-like factor LKLF
, lung Kruppel-like zinc finger transcription factor
, lung krueppel-like factor
, lung Kruppel-like factor