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Natural killer (NK) cells are a distinct lineage of lymphocytes that mediate cytotoxic activity and secrete cytokines upon immune stimulation. Zusätzlich bieten wir Ihnen Killer Cell Lectin-Like Receptor Subfamily D, Member 1 Proteine (17) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 269 products:
Mouse (Murine) Monoclonal KLRD1 Primary Antibody für EIA, FACS - ABIN951437
Toyama-Sorimachi, Taguchi, Yagita, Kitamura, Kawasaki, Koyasu, Karasuyama: Mouse CD94 participates in Qa-1-mediated self recognition by NK cells and delivers inhibitory signals independent of Ly-49. in Journal of immunology (Baltimore, Md. : 1950) 2001
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Mouse (Murine) Monoclonal KLRD1 Primary Antibody für EIA, FACS - ABIN951438
Meyers, Ryu, Monney, Nguyen, Greenfield, Freeman, Kuchroo: Cutting edge: CD94/NKG2 is expressed on Th1 but not Th2 cells and costimulates Th1 effector functions. in Journal of immunology (Baltimore, Md. : 1950) 2002
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Mouse (Murine) Monoclonal KLRD1 Primary Antibody für EIA, FACS - ABIN951439
Vance, Kraft, Altman, Jensen, Raulet: Mouse CD94/NKG2A is a natural killer cell receptor for the nonclassical major histocompatibility complex (MHC) class I molecule Qa-1(b). in The Journal of experimental medicine 1999
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Mouse (Murine) Monoclonal KLRD1 Primary Antibody für FACS - ABIN316814
Vance, Jamieson, Raulet: Recognition of the class Ib molecule Qa-1(b) by putative activating receptors CD94/NKG2C and CD94/NKG2E on mouse natural killer cells. in The Journal of experimental medicine 2000
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Human Monoclonal KLRD1 Primary Antibody für FACS - ABIN4898458
Hughes, Becknell, McClory, Briercheck, Freud, Zhang, Mao, Nuovo, Yu, Caligiuri: Stage 3 immature human natural killer cells found in secondary lymphoid tissue constitutively and selectively express the TH 17 cytokine interleukin-22. in Blood 2009
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Human Monoclonal KLRD1 Primary Antibody für ELISA, WB - ABIN969032
Lonigro, Mirabile, Munda, Barchitta, Bottino, Fazzio, Di Cataldo, Agodi, Schilirò: Association between high expression of natural killer related-genes (NCAM/CD94) and early death during induction in children with acute myeloid leukemia. in Leukemia 2008
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Human Monoclonal KLRD1 Primary Antibody für FACS, ELISA - ABIN969031
Masilamani, Narayanan, Prieto, Borrego, Coligan: Uncommon endocytic and trafficking pathway of the natural killer cell CD94/NKG2A inhibitory receptor. in Traffic (Copenhagen, Denmark) 2008
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Human Polyclonal KLRD1 Primary Antibody für EIA, WB - ABIN453720
Rodríguez, Carretero, Glienke, Bellón, Ramírez, Lehrach, Francis, López-Botet: Structure of the human CD94 C-type lectin gene. in Immunogenetics 1998
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Human Monoclonal KLRD1 Primary Antibody für FACS, IHC (fro) - ABIN319857
Aramburu, Balboa, Ramírez, Silva, Acevedo, Sánchez-Madrid, De Landázuri, López-Botet: A novel functional cell surface dimer (Kp43) expressed by natural killer cells and T cell receptor-gamma/delta+ T lymphocytes. I. Inhibition of the IL-2-dependent proliferation by anti-Kp43 monoclonal antibody. in Journal of immunology (Baltimore, Md. : 1950) 1990
Collectively, these results provide evidence for the first time that CD94/NKG2C (zeige KLRC2 Antikörper) is involved in chronic graft-versus-host disease prevention
CD94 and NKG2A (zeige KLRC1 Antikörper) polymorphisms may contribute to genetic susceptibility to rheumatoid arthritis or affect the response to anti-TNF (zeige TNF Antikörper) therapy in patients of Caucasian origin.
Coengagement of inhibitory receptors, either KIR2DL1 (zeige KIR2DL1 Antikörper) or CD94-NKG2A (zeige KLRC1 Antikörper), did not inhibit phosphorylation of Stat5 (zeige STAT5A Antikörper) but inhibited selectively phosphorylation of Akt (zeige AKT1 Antikörper) and S6 ribosomal protein (zeige RPS6 Antikörper).
it is not clear if high expression of CD94 on peripheral blood NK cells is related to abnormal activity of endometrial NK cells.
Data indicate that NKG2 receptor NKG2E (zeige KLRC3 Antikörper) was capable of associating with CD94 and DAP12 (zeige TYROBP Antikörper) but that the complex was retained intracellularly at the endoplasmic reticulum.
Balance between activating NKG2D (zeige KLRK1 Antikörper), DNAM-1 (zeige CD226 Antikörper), NKp44 (zeige NCR2 Antikörper) and NKp46 (zeige NCR1 Antikörper) and inhibitory CD94/NKG2A (zeige KLRC1 Antikörper) receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts.
Data indicate that the expression of KLRD1 (CD94) and NKG2E (KLRC3 (zeige KLRC3 Antikörper)) was reduced in NK-enriched cells in fulminant type 1 diabetes.
NKG2C (zeige KLRC2 Antikörper) zygosity influences CD94/NKG2C (zeige KLRC2 Antikörper) receptor function and the NK-cell compartment redistribution in response to human cytomegalovirus.
Synergistic inhibition of natural killer cells by the nonsignaling molecule CD94.
Studies indicate that HLA-E (zeige HLAE Antikörper) interacts with CD94/NKG2 receptors expressed mainly on the surface of natural killer (NK) cells, thus confining its role to the regulation of NK-cell function.
These findings suggest that the CD94/NKG2A (zeige KLRC1 Antikörper) heterodimers in cattle, in contrast to other species, are binding several different ligands.
Segregation of a spontaneous Klrd1 mutation in DBA (zeige RPS19 Antikörper)/2 mouse substrains
The transmembrane region sequences of CD94 and NKG2 in mouse and rat differ markedly from other mammalian orthologs by the presence of a lysine residue in the transmembrane region.
The complex interplay between various stimuli may account for the variable expression of CD94/NKG2A (zeige KLRC1 Antikörper) during responses to different pathogens in vivo.
expression of CD94 and its associated NKG2A (zeige KLRC1 Antikörper), NKG2C (zeige KLRC2 Antikörper), and NKG2E (zeige KLRC3 Antikörper) subunits is dispensable for NK cell development, education, and many NK cell functions
show that CD94, a molecule preferentially expressed by NK cells, is essential for the resistance of C57BL/6 mice to mousepox, a disease caused by the Orthopoxvirus ectromelia virus
Implications of CD94 deficiency and monoallelic NKG2A (zeige KLRC1 Antikörper) expression for natural killer cell development and repertoire formation.
CD94 and NKG2 were both expressed early in NK cell development, sometimes in the absence of NK1.1, with CD94 invariably being expressed at two different levels. IL-4 differentially inhibited the expression of CD94 and Ly49 receptors.
Inhibitory receptor CD94 is expressed on mature fetal thymic and adult epidermal TCR Vgamma3+ lymphocytes.
The acquisition of individual receptor gene expressions during various stages of differentiation in culture from embryonic stem cells to NK cells follows a predetermined order, with the order of receptor acquisition being first CD94.
There is no evidence that CD94 inhibits either the lytic function of lymphocytic choriomeningitis virus-specific T cells or their capacity to produce effector cytokines upon peptide stimulation.
Natural killer (NK) cells are a distinct lineage of lymphocytes that mediate cytotoxic activity and secrete cytokines upon immune stimulation. Several genes of the C-type lectin superfamily, including members of the NKG2 family, are expressed by NK cells and may be involved in the regulation of NK cell function. KLRD1 (CD94) is an antigen preferentially expressed on NK cells and is classified as a type II membrane protein because it has an external C terminus. Three transcript variants encoding two different isoforms have been found for this gene.
, NK cell receptor
, natural killer cells antigen CD94
, killer cell lectin-like receptor family D member 1
, killer cell lectin-like receptor subfamily D member 1
, Killer cell lectin-like receptor subfamily D member 1
, CD94 antigen (located within the rat natural killer gene complex)