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The protein encoded by HAVCR1 is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. Zusätzlich bieten wir Ihnen HAVCR1 Antikörper (160) und HAVCR1 Kits (84) und viele weitere Produktgruppen zu diesem Protein an.
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Human HAVCR1 Protein expressed in Human Cells - ABIN2003797
Gandhi, Yi, Ha, Shi, Ismail, Nathoo, Bonventre, Zhang, Gunaratnam: Accelerated receptor shedding inhibits kidney injury molecule-1 (KIM-1)-mediated efferocytosis. in American journal of physiology. Renal physiology 2014
Data suggest that T-cell immunoglobulin domain and mucin domain containing protein 1 (TIM1) to be under positive natural selection in primates.
Cisplatin enhances kidney injury molecule-1 (Kim-1) gene expression in kidney S3 cells.
By preventing ERK1/2 phosphorylation following renal injury, STAT3 (zeige STAT3 Proteine) phosphorylation is decreased, leading to less phosphorylated STAT3 (zeige STAT3 Proteine) within the nucleus, and subsequently less KIM-1 mRNA increases post injury
Study used a previously described highly mobile membrane mimetic membrane in combination with a conventional lipid bilayer model to generate a membrane-bound configuration of Tim1 in silico, identified two possible states for a membrane-bound form of Tim1.
Our data reveal a previously unknown role for Galpha12 in regulating efferocytosis and that renal tubular epithelial cells require KIM-1 to mediate this process.
Urinary L-FABP (zeige FABP1 Proteine), NGAL (zeige LCN2 Proteine), Kim-1 and albumin (zeige ALB Proteine) levels increased during the acute phase of kidney injury and were significantly correlated with the degree of tubulointerstitial fibrosis during the chronic phase. These markers could detect higher risk of progression to CKD.
Blockade of Tim-1 changes Th1 (zeige HAND1 Proteine)/Th2 balance and reduces circulating regulatory T cells to enhance atherosclerosis in LDL receptor (zeige LDLR Proteine) knockout mice.
Our results suggest that KIM-1 is an endogenous protective mechanism against renal ischemia-reperfusion injury
data suggest that TIM-1 signaling plays a direct role in Breg maintenance and induction both under physiological conditions (in response to ACs (zeige Acsl1 Proteine)) and in response to therapy through TIM-1 ligation
Deletion of the mucin (zeige SLC13A2 Proteine) domain impaired KIM-1-mediated phagocytic function, resulting in increased proinflammatory cytokine production, decreased antiinflammatory growth factor secretion by proximal epithelial cells, and an increase in tissue macrophages.
Tim-1 is critical for maintaining self-tolerance by regulating IL-10 (zeige IL10 Proteine) production in Bregs
Report detection of drug-induced acute kidney injury in humans by combining the sensitivity of urinary KIM-1 along with urinary miR (zeige MLXIP Proteine)-21, -200c, and -423.
measurement of urinary and renal KIM-1 level may be helpful to evaluate severity of renal pathological damage and prognosis in adult Henoch-Schonlein purpura patients with nephritis
Urine KIM-1 level in acute kidney injury patients was significantly higher than that in the healthy controls.
PCNSL is characterized by frequent Tim-1 (zeige ARHGEF5 Proteine) expression, and its soluble form in CSF (zeige CSF2 Proteine) may become a useful biomarker for PCNSL.
TIM-1 (zeige ARHGEF5 Proteine) is a unique marker for the identification of a human IL-10 (zeige IL10 Proteine)(+) Breg subpopulation which is partially superimposed with transitional B cells
in the current meta-analysis, based on ten prospective studies involving 29366 participants, we evaluated the role of urinary tubular injury markers (NGAL, KIM-1 and NAG) in predicting clinical outcomes including CKD stage 3, end stage renal disease and mortality.
Urinary KIM-1 levels in the acute kidney injury (AKI) preterm infant group were not significantly higher than in no-AKI group on day of life 1, 3 and 7.
We conclude that KIM-1 might serve as a sensitive biomarker to screen children for kidney damage induced by environmental toxic agents, such as chromium and arsenic
These results suggest that a 6-amino acid deletion polymorphism in the mucin (zeige SLC13A2 Proteine) domain of TIM-1 (zeige ARHGEF5 Proteine) protects from HIV-1 infection with a recessive effect.
The present study shows that serum and urine levels of NGAL (zeige LCN2 Proteine) and KIM-1 are higher in patients with acute kidney injury than in those without acute kidney injury, and that serum NGAL (zeige LCN2 Proteine) and the presence of chronic kidney disease are significant predictors of acute kidney injury in scrub typhus.
The protein encoded by this gene is a membrane receptor for both human hepatitis A virus (HHAV) and TIMD4. The encoded protein may be involved in the moderation of asthma and allergic diseases. The reference genome represents an allele that retains a MTTVP amino acid segment that confers protection against atopy in HHAV seropositive individuals. Three transcript variants encoding the same protein have been found for this gene.
hepatitis A virus cellular receptor 1
, T-cell immunoglobulin and mucin domain containing 1
, hepatitis A virus cellular receptor 1 homolog
, kidney injury molecule 1
, t cell immunoglobulin and mucin domain-containing protein 1
, t cell membrane protein 1
, T cell immunoglobin domain and mucin domain protein 1
, T-cell membrane protein 1
, rho guanine nucleotide exchange factor 5