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The protein encoded by FUT1 is a Golgi stack membrane protein that is involved in the creation of a precursor of the H antigen, which is required for the final step in the soluble A and B antigen synthesis pathway. Zusätzlich bieten wir Ihnen FUT1 Antikörper (36) und FUT1 Proteine (16) und viele weitere Produktgruppen zu diesem Protein an.
Fut1 plays an important role in regulating angiogenesis and ICAM-1 (zeige ICAM1 ELISA Kits) expression in inflammatory arthritis.
We found that Fut1 mRNA and Fut4 (zeige FUT4 ELISA Kits) mRNA were upregulated, while Fut2 (zeige FUT2 ELISA Kits) mRNA and Fut9 (zeige FUT9 ELISA Kits) mRNA were downregulated by androgen in the caput epididymis.
fucosyltransferase (Fut)-deficient (Fut1, Fut2 (zeige FUT2 ELISA Kits) and Fut4 (zeige FUT4 ELISA Kits)) mice which lack LeX (zeige FUT4 ELISA Kits) or LeY antigen are still fertile.
These results indicate that Fut2 (zeige FUT2 ELISA Kits)-mediated fucosylated epithelial cells (F-ECs) share M cell-related fucosylated molecules but maintain distinctive EC characteristics, Fut1 is, therefore, a reliable marker for M cells.
FUT1 is essential for fucosylation of glycolipid GA1 in the pancreas.
In null mutant mice lacking FUT1, the absence of blood group (zeige DARC ELISA Kits) H carbohydrate resulted in the delayed maturation of the glomerular layer of the main olfactory bulb.
Vasculogenesis was the most clearly affected by FUT1 expression, suggesting that tumor angiomorphogenesis may depend on E-selectin (zeige SELE ELISA Kits)-mediated interaction between HCC (zeige FAM126A ELISA Kits) and endothelial cells, the inhibition of which remarkably retards tumor growth.
downregulation of FUT1, which leads to the perinuclear localization of LAMP-1 (zeige LAMP1 ELISA Kits) and 2, is correlated with increased rate of autophagic flux by decreasing mTOR (zeige FRAP1 ELISA Kits) signaling and increasing autolysosome formation.
The H blood group (zeige DARC ELISA Kits) system is defined by a terminal fucose residue found on red blood cells and in secretions formed by the action of alpha-1,2-fucosyltransferases 1 (alpha2FucT1) and 2 (alpha2FucT2), respectively. Mutant alleles of the corresponding FUT1 and FUT2 (zeige FUT2 ELISA Kits) genes result in either a H- phenotype (Bombay phenotype) or a weak H phenotype (para-Bombay). Review.
Two novel FUT1 mutations have been identified in the proband's FUT1 gene. The insertion mutation in the FUT1 that caused a shift of the open reading frame and formed a termination codon early at Amino Acid Position 334 may be the main reason for H deficiency in this case.
Protein O-fucosyltransferase 1 (zeige POFUT1 ELISA Kits) promotes trophoblast cell proliferation by activating MAPK (zeige MAPK1 ELISA Kits) and PI3K (zeige PIK3CA ELISA Kits)/Akt (zeige AKT1 ELISA Kits) signaling pathways.[poFUT1 (zeige POFUT1 ELISA Kits)]
Data suggest that the identification of genes in response to alpha1,2-fucosyl transferase (FUT1) may provide a theoretical basis for the investigations of the molecular mechanism of ovarian cancer.
alpha(1,2)-fucosyltransferase activity is involved in the development of multidrug resistance of chronic myeloid leukemia (zeige BCL11A ELISA Kits) cells probably through FUT1 regulated the activity of EGFR (zeige EGFR ELISA Kits)/MAPK (zeige MAPK1 ELISA Kits) signaling pathway and the expression of P-gp (zeige ABCB4 ELISA Kits).
data confirm the hypothesis that the h2 allele is linked to Se(357, 716), and the concurrence of unique FUT1 and FUT2 (zeige FUT2 ELISA Kits) mutations is geographically specific.
We show that fut1 in RA synovial fibroblasts is important in angiogenesis, leukocyte-synovial fibroblast adhesion, and synovial fibroblast proliferation
FUT1 mediates c-Jun (zeige JUN ELISA Kits)-induced cell proliferation in ovarian cancer cells.c-Jun (zeige JUN ELISA Kits) transcriptionally modulates FUT1 expression in ovarian cancer cells.
FUT1 expression levels and low FUT2 (zeige FUT2 ELISA Kits) expression levels in the intestines of Sutai pigs affected FUT1 and FUT2 (zeige FUT2 ELISA Kits) enzymes, the synthesis of type 2 H and type 1 H antigens, and E. coli F18 (zeige MAMLD1 ELISA Kits) adhesion. Moreover, low FUT2 (zeige FUT2 ELISA Kits) expression levels conferred resistance to E. coli F18 (zeige MAMLD1 ELISA Kits).
Polymorphism of FUT1 and MUC4 (zeige MUC4 ELISA Kits) associated with resistance to colibacteriosis was determined.
The 5'-flanking region of the FUT1 gene (-1150 to +50 bp) exhibited promoter activity and the -1150-bp to -849-bp region showed negative regulation of the gene.
Studied the beneficial effects of alpha (1,2)-fucosyltransferase (FUT1) M307 (A) on piglet survival on commercial farms. Piglet survival for individuals with the AA genotype was almost two-fold greater than for GG individuals.
The results indicated that Sutai pigs with the AA genotype in M307 of FUT1 gene have relatively strong general disease resistance ability in piglets.
The mutation at M307 in the FUT1 gene determines susceptibility of small intestinal epithelium to E. coli F18 (zeige MAMLD1 ELISA Kits) adhesion in Sutai piglets.
FUT1 mutations might play a role in pig infection by multi-pathogens, and the AA may be a favourable genotype for increasing the resistance to disease.
Beneficial genotype of swine FUT1 gene governing resistance to E. coli F18 (zeige MAMLD1 ELISA Kits) is associated with important economic traits
transgenic male pigs were produced that possess an alpha alpha-1,3-galactosyltransferase (zeige GGTA1 ELISA Kits) knockout allele and express a randomly inserted human alpha 1,2-fucosylosyltransferase transgene
Results showed that the genetic polymorphisms of the FUT1 locus were only detected in the 3 exotic pig breeds and indicated that FUT 1 gene and the type of breeds significantly affected total number born.
Data show that si-RNA induced down-regulation of FUT1 and FUT2 (zeige FUT2 ELISA Kits) reduced expression of fucosylated nucleolin (zeige NCL ELISA Kits) glycoforms and their exposure at the cell surface.
The protein encoded by this gene is a Golgi stack membrane protein that is involved in the creation of a precursor of the H antigen, which is required for the final step in the soluble A and B antigen synthesis pathway. This gene is one of two encoding the galactoside 2-L-fucosyltransferase enzyme. Mutations in this gene are a cause of the H-Bombay blood group.
GDP-L-fucose:beta-D-galactoside 2-alpha-L-fucosyltransferase 1
, H transferase
, alpha(1,2)FT 1
, beta-galactoside alpha-1,2-fucosyltransferase
, galactoside 2-alpha-L-fucosyltransferase 1
, alpha 1,2-fucosyltransferase A
, alpha 12-fucosyltransferase
, alpha12-fucosyltransferase a
, fucosyltransferase 1 (galactoside 2-alpha-L-fucosyltransferase)
, alpha (1,2) fucosyltransferase
, blood group H alpha 2-fucosyltransferase
, alpha(1-2) fucosyltransferase 1
, fucosyltransferase 1