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EBF3 encodes a member of the early B-cell factor (EBF) family of DNA binding transcription factors. Zusätzlich bieten wir Ihnen EBF3 Antikörper (40) und EBF3 Proteine (4) und viele weitere Produktgruppen zu diesem Protein an.
Our findings highlight the critical role of Ebf3 in multipolar-to-bipolar transition via positive feedback regulation of NeuroD1 in the developing neocortex.
Ebf3 is a new regulator of terminal muscle differentiation in the diaphragm, and Ebf (zeige EBF1 ELISA Kits) factors cooperate with MyoD (zeige MYOD1 ELISA Kits) in the induction of muscle-specific (zeige EIF3K ELISA Kits) genes
The regulation of Ebf3 expression by miR218 controls the terminal differentiation of dopaminergic neurons.
Ebf2 (zeige EBF2 ELISA Kits) and Ebf3, singly or together, control the migration of Cajal-Retzius cells arising in the cortical hem. These findings provide evidence that Ebfs directly regulate Cajal-Retzius cell development
In 11 affected individuals from 11 unrelated families, we identified de novo variants in EBF3 (zeige MAPRE3 ELISA Kits) as potentially causative for the neurodevelopmental phenotype. The variants include one nonsense, two frameshift deletions, one splice, and three missense variants. There are three de novo missense variants, (p.(Lys64Thr), p.(His157Gln), and p.(Arg209Gln), which are all in the COE1 (zeige EBF1 ELISA Kits) DNA-binding domain.
EBF3 (zeige MAPRE3 ELISA Kits), a transcription factor previously unknown to be associated with human disease, is important for brain and other organ development and warrants further investigation
findings indicate that mutations in EBF3 cause a genetic neurodevelopmental syndrome and suggest that loss of EBF3 function might mediate a subset of neurologic phenotypes shared by ARX-related disorders, including intellectual disability, abnormal genitalia, and structural CNS malformations
findings demonstrate that variants disrupting EBF3 (zeige MAPRE3 ELISA Kits)-mediated transcriptional regulation cause intellectual disability and developmental delay and are present in approximately 0.1% of individuals with unexplained neurodevelopmental disorders
Early B-cell factor 3 (EBF3) is a novel tumor suppressor gene with promoter hypermethylation in pediatric acute myeloid leukemia (zeige BCL11A ELISA Kits)
EBF3 (zeige MAPRE3 ELISA Kits) tumor suppressor is epigenetically silenced and that it serves as an independent prognostic marker in gastric carcinoma.
Results verify IRX1 (zeige IRX3 ELISA Kits), EBF3 (zeige MAPRE3 ELISA Kits), SLC5A8 (zeige SLC5A8 ELISA Kits), SEPT9 (zeige SEPT9 ELISA Kits), and FUSSEL18 as valid methylation markers in two separate sets of HNSCC specimens; also preliminarily show a trend between HPV16 positivity and target gene hypermethylation of IRX1 (zeige IRX3 ELISA Kits), EBF3 (zeige MAPRE3 ELISA Kits), SLC5A8 (zeige SLC5A8 ELISA Kits), and SEPT9 (zeige SEPT9 ELISA Kits).
Findings suggested that the transfection of EBF3 (zeige MAPRE3 ELISA Kits) gene into HepG2 induced the cell proliferation from G1 phase to G2 phase by increasing the number of cells.
Expression of EBF3 resulted in cell cycle arrest and apoptosis. EBF3 regulates a transcriptional program underlying a putative tumor suppression pathway.
Frequent methylation of EBF3 (zeige MAPRE3 ELISA Kits) gene is associated with head and neck squamous cell carcinoma
Data revealed that expression of DNA binding inhibitor 3, early B cell factor 2, Ebf3, Iroquois related homeobox 1, Kruppel-like factor 7 , mab-21-like 1 , fatty acid binding protein 7 and stathmin-like 4,were enriched in the diencephalon of zebrafish.
This gene encodes a member of the early B-cell factor (EBF) family of DNA binding transcription factors. EBF proteins are involved in B-cell differentiation, bone development and neurogenesis, and may also function as tumor suppressors. The encoded protein inhibits cell survival through the regulation of genes involved in cell cycle arrest and apoptosis, and aberrant methylation or deletion of this gene may play a role in multiple malignancies including glioblastoma multiforme and gastric carcinoma.
, Olf-1/EBF-like 2
, early B-cell factor 3
, transcription factor COE3
, olf-1/EBF-like 2
, LOW QUALITY PROTEIN: transcription factor COE3