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The laminar organization of multiple neuronal types in the cerebral cortex is required for normal cognitive function. Zusätzlich bieten wir Ihnen DAB1 Kits (20) und DAB1 Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 139 products:
Human Polyclonal DAB1 Primary Antibody für IHC - ABIN362934
Strasser, Fasching, Hauser, Mayer, Bock, Hiesberger, Herz, Weeber, Sweatt, Pramatarova, Howell, Schneider, Nimpf: Receptor clustering is involved in Reelin signaling. in Molecular and cellular biology 2004
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Human Polyclonal DAB1 Primary Antibody für IHC (p) - ABIN197508
Chen, Ochalski, Tran, Sahir, Schubert, Pramatarova, Howell: Interaction between Dab1 and CrkII is promoted by Reelin signaling. in Journal of cell science 2004
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Human Polyclonal DAB1 Primary Antibody für ELISA, WB - ABIN1531813
Lambert de Rouvroit, Goffinet: Cloning of human DAB1 and mapping to chromosome 1p31-p32. in Genomics 1998
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Human Polyclonal DAB1 Primary Antibody für IF, ELISA - ABIN1532843
Gregory, Barlow, McLay, Kaul, Swarbreck, Dunham, Scott, Howe, Woodfine, Spencer, Jones, Gillson, Searle, Zhou, Kokocinski, McDonald, Evans, Phillips, Atkinson, Cooper, Jones, Hall, Andrews, Lloyd et al.: The DNA sequence and biological annotation of human chromosome 1. ... in Nature 2006
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Human Polyclonal DAB1 Primary Antibody für IHC (p), WB - ABIN652935
Ranganathan, Liu, Migliorini, Von Arnim, Peltan, Mikhailenko, Hyman, Strickland: Serine and threonine phosphorylation of the low density lipoprotein receptor-related protein by protein kinase Calpha regulates endocytosis and association with adaptor molecules. in The Journal of biological chemistry 2004
Mouse (Murine) Polyclonal DAB1 Primary Antibody für WB - ABIN264969
Magdoff-Fairchild, Rosen, Chiu: Triclinic crystals associated with fibers of deoxygenated sickle hemoglobin. in The EMBO journal 1984
Alternatively spliced dab1 isoforms were identified.
these results establish an unstable repeat insertion in DAB1 as a cause of cerebellar degeneration.
Study shows that early neural cells transiently express Reelin (zeige RELN Antikörper) at the time they leave the presumptive olfactory/vomeronasal epithelium and that Dab 1 is present in the migratory cell mass and in the presumptive ensheathing cells in the absence of reelin (zeige RELN Antikörper).
this study is the first to provide genetic evidence for DAB1 as a candidate AD liability/protection gene, although the strength of the contribution of DAB1 may differ among populations
results of this study demonstrated the presence of reelin (zeige RELN Antikörper), its receptors VLDLR (zeige VLDLR Antikörper) and ApoER2 (zeige LRP8 Antikörper) as well as Dab1 in the ENS and might indicate a novel role of the reelin (zeige RELN Antikörper) system in regulating neuronal plasticity and pre-synaptic functions in the ENS.
The finding of this study suggested that variations in DAB1 involved in the Reelin signaling pathway might contribute to genetic susceptibility to autism with Chinese Han decent.
Variation in genes encoding proteins at the gateway of Reelin (zeige RELN Antikörper) signaling: ligands RELN (zeige RELN Antikörper) and APOE (zeige APOE Antikörper), their common receptors APOER2 (zeige LRP8 Antikörper) and VLDLR (zeige VLDLR Antikörper), and adaptor DAB1, was examined.
show that Reelin (zeige RELN Antikörper)-stimulated Notch-1 (zeige NOTCH1 Antikörper) activation is dependent on Reelin (zeige RELN Antikörper) signaling
alternative splicing of Dab1 is conserved in avian and mammalian species, with Dab1-L driving SFK phosphorylation in both species
we report for the first time, that DAB1 is significantly up-regulated in human frontal cortex brain samples of Alzheimer disease patients
Dab1, an essential component of the reelin (zeige RELN Antikörper) pathway, is required for glia-independent somal translocation in the neocortex.
This study showed that spontaneous Dab1 mutations causing cerebellar pathology are impaired in motor functions during the neonatal period.
an aberrant fragment of Dab1 protein (p64 (zeige IL2RG Antikörper)/60) is present in the brain of yotari mouse
When pregnant mice are exposed to EMF, Dab1 expression increases in the EMF-treated fetal cerebral cortex extracts as compared to controls and SHAM group.
It findings link reelin (zeige RELN Antikörper) with Dab1 and suggest that Dab1 functions downstream of reelin (zeige RELN Antikörper) action on the homeostasis of the crypt-villus unit.
The results of this study indicate a causal relation between the downregulation of Dab1 protein levels during development and the structural and behavioral deficits associated with psychiatric diseases in the adult.
The expression of Dab-1 was analyzed in the developing mouse autopod. Transcripts of Dab-1 were abundant in the autopodial tendon primordia, but transcripts were also present in the undifferentiated mesoderm located around the digits.
results suggest a major role of VEGF in the regulation of reelin signaling, and Dab1 as a key molecule in the cross talk between reelin and VEGF signaling pathways.
Dab1 is specifically knocked out in immature neurons while it remains active in glial cells.
Dab1 mediated the association of CIN85 (zeige SH3KBP1 Antikörper) with ApoER2 (zeige LRP8 Antikörper) or VLDLR (zeige VLDLR Antikörper) in neurons.
Dab1 is a critical regulator of synaptic function and hippocampal-dependent associative and spatial learning.
Alternative spliced forms of disabled homolog 1 (DAB1) have been isolated from brain and liver.
The laminar organization of multiple neuronal types in the cerebral cortex is required for normal cognitive function. In mice, the disabled-1 gene plays a central role in brain development, directing the migration of cortical neurons past previously formed neurons to reach their proper layer. This gene is similar to disabled-1, and the protein encoded by this gene is thought to be a signal transducer that interacts with protein kinase pathways to regulate neuronal positioning in the developing brain. Alternatively spliced transcript variants of this gene have been reported, but their full length nature has not been determined.
disabled homolog 1
, disabled homolog 1 (Drosophila)
, DAB1 variant protein-like
, disabled homolog 1-like