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Dihydropyrimidinase catalyzes the conversion of 5,6-dihydrouracil to 3-ureidopropionate in pyrimidine metabolism. Zusätzlich bieten wir Ihnen DPYS Kits (8) und DPYS Proteine (7) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 107 products:
Cow (Bovine) Polyclonal DPYS Primary Antibody für WB - ABIN2776832
Thomas, Chen, Lin, Tomlinson, Lam, Liu, Yeung, Chan, Wong: Middle cerebral artery stenosis increased the risk of vascular disease mortality among type 2 diabetic patients. in Cerebrovascular diseases (Basel, Switzerland) 2008
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Human Monoclonal DPYS Primary Antibody für WB - ABIN2779197
Kunicka, Prochazka, Krus, Bendova, Protivova, Susova, Hlavac, Liska, Novak, Schneiderova, Pitule, Bruha, Vycital, Vodicka, Soucek: Molecular profile of 5-fluorouracil pathway genes in colorectal carcinoma. in BMC cancer 2016
Cow (Bovine) Polyclonal DPYS Primary Antibody für WB - ABIN2776833
Thomas, Ezzeldin, Guarcello, Mattison, Fridley, Diasio: Genetic regulation of beta-ureidopropionase and its possible implication in altered uracil catabolism. in Pharmacogenetics and genomics 2008
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Two unrelated pediatric DPYS deficiency cases are being described as compound heterozygotes for a novel intronic mutation c.1443+5G>A in intron 8 and a previously described missense mutation c.1001A>G (p.Q334R) in exon 6.
Stepwise Cox regression modelling suggested that the methylation of genes HSPB1, CCND2 and DPYS contributed objective prognostic information to Gleason score and PSA with respect to prostate cancer-related death.
The p.S379R and p.L7V mutations were likely to cause structural destabilization and protein misfolding. Four mutations were identified in multiple unrelated DHP (zeige DPYD Antikörper) patients, indicating that DHP (zeige DPYD Antikörper) deficiency may be more common than anticipated.
Results indicate that missense and nonsense variants in DPYS are infrequent, however, the development of serious primarily gastrointestinal toxicity could be influenced by non-coding DPYS sequence variants c.-1T>C and IVS1-58T>C.
clinical, biochemical & genetic findings of two newly identified patients with a complete DHP (zeige DPYD Antikörper) deficiency; both patients were compound heterozygous for the missense mutation 1078T>C (W360R) in exon 6 and a novel missense mutation 1235G>T (R412M) in exon 7
data presented in this study offers evidence for the possible genetic regulation of the DPYS gene and its possible influence on uracil catabolic pathway
Dihydropyrimidinase catalyzes the conversion of 5,6-dihydrouracil to 3-ureidopropionate in pyrimidine metabolism. Dihydropyrimidinase is expressed at a high level in liver and kidney as a major 2.5-kb transcript and a minor 3.8-kb transcript. Defects in the DPYS gene are linked to dihydropyrimidinuria.
, dihydropyrimidine amidohydrolase