Use your antibodies-online credentials, if available.
Keine Produkte auf Ihrer Vergleichsliste.
Ihr Warenkorb ist leer.
Cytochrome b (-245) is composed of cytochrome b alpha (CYBA) and beta (CYBB) chain. Zusätzlich bieten wir Ihnen CYBB Antikörper (117) und CYBB Proteine (6) und viele weitere Produktgruppen zu diesem Protein an.
Showing 5 out of 11 products:
Human CYBB ELISA Kit für Sandwich ELISA - ABIN418998
Tassone, Perticone, Sciacqua, Mafrici, Settino, Malara, Mollace, Sesti, Perticone: Low dose of acetylsalicylic acid and oxidative stress-mediated endothelial dysfunction in diabetes: a short-term evaluation. in Acta diabetologica 2015
Rat (Rattus) CYBB ELISA Kit für Sandwich ELISA - ABIN433568
Du, Wang, Wang: Role of RhoA/MERK1/ERK1/2/iNOS signaling in ocular ischemic syndrome. in Graefe's archive for clinical and experimental ophthalmology 2016
The nox1 (zeige NOX1 ELISA Kits), nox2/cybb expression in zebrafish during early nervous system development from 12 to 48 hours post fertilization
Data indicate that NADPH oxidase (zeige NOX1 ELISA Kits) NOx2 inhibition attenuates endoplasmic reticulum (ER) stress and apoptosis.
Finally, overexpression of miR34a in EBNA1-expressing SNU719 cells induced typical apoptosis, suggesting that reactivation of miR34a in EBNA1-expressing gastric cancer cells could be a strategy for treatment of EBV-infected gastric cancer cells.
NOX2 expression was significantly higher in the cortex of asphyctic suicide subjects than in non-suicidal asphyxia and controls.
Acute myeloid leukemia (zeige BCL11A ELISA Kits) cells -derived NOX2 drives transfer of mitochondria via the generation of superoxide.
NOX2, NOX4 (zeige NOX4 ELISA Kits), and mitochondrial-derived reactive oxygen species contribute to angiopoietin-1 (zeige ANGPT1 ELISA Kits) signaling and angiogenic responses in endothelial cells.
PFK-2 (zeige PFKFB3 ELISA Kits) seems to be a strategic element that links NADPH oxidase (zeige NOX1 ELISA Kits) activation and glycolysis modulation, and, as such, is proposed as a potential therapeutic target in inflammatory diseases.
data support NOX2 as a critical component of the suppressive machinery of CD8 (zeige CD8A ELISA Kits) Tregs and suggest that repairing NOX2 deficiency in these cells may protect older individuals from tissue-destructive inflammatory disease, such as large-vessel vasculitis.
CD8 (zeige CD8A ELISA Kits)+ T cells in close contact with target T cells release NADPH oxidase 2-containing microvesicles that inhibit TCR activation by elevating ROS (zeige ROS1 ELISA Kits) and thereby reducing phosphorylation of the TCR-associated kinase ZAP70 (zeige ZAP70 ELISA Kits).
Thioredoxin (zeige TXN ELISA Kits) attenuates oxidized low-density lipoprotein induced oxidative stress in human umbilical vein endothelial cells by reducing NOX2-NOX4 (zeige NOX4 ELISA Kits) activity.
Nox4 (zeige NOX4 ELISA Kits)-derived H2O2 in part activates Nox2 to increase mitochondrial ROS (zeige ROS1 ELISA Kits) via pSer36-p66Shc (zeige SHC1 ELISA Kits), thereby enhancing VEGFR2 (zeige KDR ELISA Kits) signaling and angiogenesis in endothelial cells.
Cytochrome b558 (zeige CYBA ELISA Kits) also traffics from a lysosomal pool to phagosomes and this is required to replenish oxidatively damaged NOX2.
Data indicate that the efficiency of NADPH oxidase (zeige NOX1 ELISA Kits) enzymatic activity is higher at endoplasmic reticulum (ER).
A p47(phox) and Src kinase (zeige CSK ELISA Kits) activation of peroxide production by Nox2 appears to be an important contributor to vascular contractile mechanisms mediated through activation of protein kinase C (zeige PKC ELISA Kits)
Virus infection activates endosomal NOX2 oxidase and restricts TLR7 (zeige TLR7 ELISA Kits) signaling, and that an endosomal NOX2 inhibitor decreases viral pathogenicity.
These data suggest that endothelial SHIP2 (zeige INPPL1 ELISA Kits) is required to maintain normal systemic glucose homeostasis and prevent oxidative stress-induced (zeige SQSTM1 ELISA Kits) endothelial dysfunction.
NOX2 is a negative regulator of maximal glucose stimulation of insulin (zeige INS ELISA Kits) secretion in C57BL/6J mouse islets.
Nox2- and Nox4 (zeige NOX4 ELISA Kits)-derived reactive oxygen species contribute to stem cell pluripotency maintenance and self-renewal.
Redox-dependent regulation of EGFR (zeige EGFR ELISA Kits) activation in airway epithelial cells was found to strongly depend on activation of either DUOX1 (zeige DUOX1 ELISA Kits) or NOX2.
NOX2 signaling in macrophages participates in the pathogenesis of obesity, and reinforce a key role for macrophage inflammation in diet-induced metabolic and neurologic decline
Studies in knockout and knock-in mice showed that the phospholipase A2 (zeige YWHAZ ELISA Kits) activity of peroxiredoxin 6 (zeige PRDX6 ELISA Kits) activates NOX2 in cultured pulmonary endothelial cells and alveolar macrophages, clarifying the metabolism of lysophosphatidyl chold to lysophosphatidic acid.
NOX2 knockout mice had significantly lower blood pressure than littermate controls, with both endothelial cells and myeloid cells having a deficiency in NOX2.
Data show that pan-NOX-inhibitor APX (zeige SHROOM1 ELISA Kits)-115 treatment decreased NADPH oxidase (zeige NOX1 ELISA Kits) (Nox) Nox1 (zeige NOX1 ELISA Kits), Nox2, and Nox4 (zeige NOX4 ELISA Kits) protein expression in the kidney.
sub-vasomotor concentration of ET-1 (zeige EDN1 ELISA Kits) leads to vascular dysfunction by impairing endothelium-dependent NO-mediated dilation via p38 (zeige MAPK14 ELISA Kits) kinase-mediated production of superoxide from NADPH oxidase (zeige NOX1 ELISA Kits) following ETA receptor activation
Reactive oxygen species generated by NADPH oxidase (zeige NOX1 ELISA Kits) contribute to the aberrant pulmonary arterial responses in piglets exposed to 3 days of hypoxia.
Upregulation of PPAR-gamma and NADPH oxidases are involved in restenosis.
Cytochrome b (-245) is composed of cytochrome b alpha (CYBA) and beta (CYBB) chain. It has been proposed as a primary component of the microbicidal oxidase system of phagocytes. CYBB deficiency is one of five described biochemical defects associated with chronic granulomatous disease (CGD). In this disorder, there is decreased activity of phagocyte NADPH oxidase\; neutrophils are able to phagocytize bacteria but cannot kill them in the phagocytic vacuoles. The cause of the killing defect is an inability to increase the cell's respiration and consequent failure to deliver activated oxygen into the phagocytic vacuole.
cytochrome b-245, beta polypeptide
, NADPH oxidase 2
, cytochrome b-245, beta polypeptide (chronic granulomatous disease)
, Cytochrome b-245 heavy chain
, NADPH oxidase heavy chain subunit
, NADPH oxidase 1
, NADPH oxidase flavocytochrome b subunit
, cytochrome b-245 heavy chain
, cytochrome b(558) subunit beta
, cytochrome b558 subunit beta
, heme-binding membrane glycoprotein gp91phox
, neutrophil cytochrome b 91 kDa polypeptide
, p22 phagocyte B-cytochrome
, superoxide-generating NADPH oxidase heavy chain subunit
, endothelial type gp91-phox
, predicted NADPH oxidase-2