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CCL5 is one of several CC cytokine genes clustered on the q-arm of chromosome 17. Zusätzlich bieten wir Ihnen CCL5 Proteine (114) und CCL5 Kits (93) und viele weitere Produktgruppen zu diesem Protein an.
Showing 10 out of 304 products:
Human Monoclonal CCL5 Primary Antibody für FACS, IHC - ABIN4900684
van Bladel, Roest, de Groot, Schutgens: Up-regulation of platelet activation in hemophilia A. in Haematologica 2011
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Human Monoclonal CCL5 Primary Antibody für EIA, Func - ABIN118992
Conway, Sahni, Szkopek: Plasmonic interconnects versus conventional interconnects: a comparison of latency, crosstalk and energy costs. in Optics express 2009
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Human Monoclonal CCL5 Primary Antibody für FACS - ABIN2657999
von Luettichau, Nelson, Pattison, van de Rijn, Huie, Warnke, Wiedermann, Stahl, Sibley, Krensky: RANTES chemokine expression in diseased and normal human tissues. in Cytokine 1996
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Human Polyclonal CCL5 Primary Antibody für IHC, IHC (fro) - ABIN446965
Sakamoto, Tsuruda, Hatakeyama, Imamura, Asada, Kitamura: Impact of age-dependent adventitia inflammation on structural alteration of abdominal aorta in hyperlipidemic mice. in PLoS ONE 2014
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Human Monoclonal CCL5 Primary Antibody für FACS - ABIN4898185
Ferrarini, Delfanti, Gianolini, Rizzi, Alfano, Lazzarin, Biswas: NF-kappa B modulates sensitivity to apoptosis, proinflammatory and migratory potential in short- versus long-term cultured human gamma delta lymphocytes. in Journal of immunology (Baltimore, Md. : 1950) 2008
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Cow (Bovine) Polyclonal CCL5 Primary Antibody für FACS, IF (p) - ABIN674949
Pattappa, Peroglio, Sakai, Mochida, Benneker, Alini, Grad: CCL5/RANTES is a key chemoattractant released by degenerative intervertebral discs in organ culture. in European cells & materials 2014
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Human Monoclonal CCL5 Primary Antibody für FACS - ABIN4898184
Tikhonov, Deetz, Paca, Berg, Lukyanenko, Lim, Pauza: Human Vgamma2Vdelta2 T cells contain cytoplasmic RANTES. in International immunology 2006
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Chicken Polyclonal CCL5 Primary Antibody für WB - ABIN2779989
Palaniappan, Singh, Singh, Singh, Ades, Briles, Hollingshead, Royal, Sampson, Stiles, Taub, Lillard: CCL5 modulates pneumococcal immunity and carriage. in Journal of immunology (Baltimore, Md. : 1950) 2006
Mouse (Murine) Polyclonal CCL5 Primary Antibody für EIA, WB - ABIN116261
Gonzalez-Rey, Chorny, Robledo, Delgado: Cortistatin, a new antiinflammatory peptide with therapeutic effect on lethal endotoxemia. in The Journal of experimental medicine 2006
In perennial allergic rhinitis patients, we found positive correlation between RANTES levels and nasal symptom score and eosinophil counts. In patients with non-allergic rhinitis with eosinophilia syndrome, the concentrations of MCP-1 (zeige CCL2 Antikörper) and RANTES were associated with nasal symptom score and percentage of eosinophils.
Oral lipid loading is linked to reduced circulating pro-inflammatory chemokines CCL2 (zeige CCL2 Antikörper), IP-10 (zeige CXCL10 Antikörper), and chemerin (zeige RARRES2 Antikörper) and to increased RANTES levels, suggesting that dietary lipids affect immune function.
Results indicated that cisplatin-induced CCL5 secretion derived from the cancer-associated fibroblasts may promote cisplatin resistance, which was mediated by regulation of the STAT3 (zeige STAT3 Antikörper) and PI3K (zeige PIK3CA Antikörper)/Akt (zeige AKT1 Antikörper) signal pathways.
Authors demonstrate that the inhibition of the CCL5 expression is the cause of the observed effects resulting from RKIP (zeige PEBP1 Antikörper) expression. Results underscore the significance of RKIP (zeige PEBP1 Antikörper) as important negative regulator of tumor microenvironment.
The N termini of CC chemokines are shown to be involved in receptor binding and oligomerization. We also report an alternative CCL3 (zeige CCL3 Antikörper) oligomer structure that reveals how conformational changes in CCL3 (zeige CCL3 Antikörper) N termini profoundly alter its surface properties and dimer-dimer interactions to affect GAG binding and oligomerization. Such complexity in oligomerization and GAG binding enables intricate, physiologically relevant regulatio
findings demonstrated that CCL5 gene -28C>G polymorphism might be a protective factor for the development of TB
The results support the fact that chemokines play important roles in the pathogenesis of HSP. RANTES/CCL5 gene polymorphisms may be related to disease severity and Henoch-Schonlein purpura (HSP) nephritis.
This is the first study to note that elevated systemic CCL5 and CXCL6 were associated with moderate/severe lumbar disc degeneration. These chemokines may be systemic biomarkers for the diagnosis and monitoring of disc degeneration.
ERalpha (zeige ESR1 Antikörper) decreased CCL5 and IL-6 (zeige IL6 Antikörper) expression in conditioned media that was collected from Cancer associated fibroblast cell.
neutrophil-borne human neutrophil peptide 1 (HNP1 (zeige DEFA1 Antikörper), alpha-defensin (zeige DEF Antikörper)) and platelet-derived CCL5 form heteromers.
RANTES produced by renal tubular cells act as a key chemokine (zeige CCL1 Antikörper) in acute kidney injury and HIF-1alpha (zeige HIF1A Antikörper) regulated LncRNA-PRINS might be involved in RANTES production.
Mycobacterium chelonae activates the gene expressions of chemokine (C-C motif) ligand 2 (CCL2 (zeige CCL2 Antikörper)) and CCL5 in murine bone marrow-derived macrophages and in vivo mouse model.
Neutralization of circulating RANTES decreased liver neutrophilic infiltration
CCL5 signaling through CCR5 (zeige CCR5 Antikörper) may increase platelet counts during physiological stress.
An intriguing finding was that S1P (zeige S1PR1 Antikörper) induced c-Fos-inhibited CCL5 directly and also indirectly through inhibition of the IFN-beta (zeige IFNB1 Antikörper) amplification loop
platelet-derived CCL5 is an important link between platelet activation and neutrophil recruitment in acute colitis
Nuclear FAK (zeige PTK2 Antikörper) is associated with chromatin and exists in complex with transcription factors and their upstream regulators that control Ccl5 expression. Furthermore, FAK's immuno-modulatory nuclear activities may be specific to cancerous squamous epithelial cells, as normal keratinocytes do not have nuclear FAK (zeige PTK2 Antikörper).
that mast cells, recruited towards CCL2 and CCL5 expressed by epithelium induced to proliferate by Human Papillomavirus 16 E7 protein
PRDX6 (zeige PRDX6 Antikörper) promotes lung tumor development via its mediated and CCL5-associated activation of the JAK2 (zeige JAK2 Antikörper)/STAT3 (zeige STAT3 Antikörper) pathway.
tumours are characterized by expression of inflammatory chemokines (CCL2 (zeige CCL2 Antikörper), CCL5, CCL7 (zeige CCL7 Antikörper), CCL8 (zeige CCL8 Antikörper), CCL12 (zeige Ccl12 Antikörper), CXCL9 (zeige CXCL9 Antikörper), CXCL10 (zeige CXCL10 Antikörper) and CX3CL1 (zeige CX3CL1 Antikörper)), reflected by an enrichment of activated Foxp3 (zeige FOXP3 Antikörper)(-) and Foxp3 (zeige FOXP3 Antikörper)(+) T cells
These results represent an important molecular mechanism whereby H. parasuis induced RANTES in the inflammatory response.
Foot-and-mouth disease virus leader proteinase inhibits dsRNA-induced RANTES transcription.
we cloned the nucleotide sequence of the 5'-flanking region of the porcine RANTES (poRANTES) gene and characterized the regulatory elements that activate transcription
The Toll (zeige TLR4 Antikörper)-like receptor signaling pathway is involved in porcine reproductive and respiratory syndrome virus-induced RANTES activation.
These results suggest that porcine RANTES can play an important role in xenotransplant rejection, through participating in the interaction between porcine endothelial cells and human monocytes.
CCL5 but not CCL2 (zeige CCL2 Antikörper) mainly attract bovine classical monocytes and promote their differentiation into LPS (zeige IRF6 Antikörper)-hypo-responsive macrophages.
decrease in sensitivity of HIV variants to RANTES neutralization during the course of progressive infection, but not during follow-up of long term survivors; data suggest a role for RANTES neutralization sensitivity of HIV-1 in AIDS pathogenesis
In the pathogenesis of allergic rhinitis, substance P (zeige TAC1 Antikörper) induces expression of RANTES in nasal mucosa.
CCL3 (zeige CCL3 Antikörper), CCL4 (zeige CCL4 Antikörper) and CCL5 gene expression was evaluated in response to simian-human immunodeficiency virus (SHIV) infection in a rhesus macaque model.
This gene is one of several CC cytokine genes clustered on the q-arm of chromosome 17. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene functions as a chemoattractant for blood monocytes, memory T helper cells and eosinophils. It causes the release of histamine from basophils and activates eosinophils. This cytokine is one of the major HIV-suppressive factors produced by CD8+ cells. It functions as one of the natural ligands for the chemokine receptor CCR5 and it suppresses in vitro replication of the R5 strains of HIV-1, which use CCR5 as a coreceptor.
chemokine (C-C motif) ligand 5
, small inducible cytokine A5
, C-C motif chemokine 5
, T-cell-specific protein RANTES
, regulated upon activation normal T-cell expressed and secreted
, small-inducible cytokine A5
, T-cell specific protein p288
, beta-chemokine RANTES
, eosinophil chemotactic cytokine
, regulated upon activation, normally T-expressed, and presumably secreted
, small inducible cytokine subfamily A (Cys-Cys), member 5
, chemokine ah294
, small inducible cytokine A5 RANTES
, chemokine CCL5/RANTES
, chemokine ligand 5